The p38 SAPK pathway is required for Ha-ras induced in vitro invasion of NIH3T3 cells (Q42819230)

7 November 2017

The p38 SAPK pathway is required for Ha-ras induced in vitro invasion of NIH3T3 cells (English)

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7 November 2017

1

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7 November 2017

Konrad Binder

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7 November 2017

Goran Vucelic

3

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7 November 2017

Stephan Herberhold

4

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7 November 2017

Bernhard Hirt

5

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7 November 2017

Hubert Loewenheim

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7 November 2017

Serena Preyer

7

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7 November 2017

Hans Peter Zenner

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7 November 2017

Christian Simon

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7 November 2017

language of work or name

English

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publication date

1 February 2005

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Experimental Cell Research

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7 November 2017

303

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7 November 2017

321-330

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2

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The Ras family of GTPases in cancer cell invasion.

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Signal-transducing protein phosphorylation cascades mediated by Ras/Rho proteins in the mammalian cell: the potential for multiplex signalling

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In the cellular garden of forking paths: how p38 MAPKs signal for downstream assistance

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The p38 pathway provides negative feedback for Ras proliferative signaling

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Transformation of NIH 3T3 cells by microinjection of Ha-ras p21 protein

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Targeting RAS signalling pathways in cancer therapy

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Experimental metastasis in nude mice of NIH 3T3 cells containing various ras genes

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Role of mitogen-activated protein kinases and c-Jun/AP-1 trans-activating activity in the regulation of protease mRNAs and the malignant phenotype in NIH 3T3 fibroblasts.

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Urokinase plasminogen activator/urokinase-specific surface receptor expression and matrix invasion by breast cancer cells requires constitutive p38alpha mitogen-activated protein kinase activity

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Rac1-MKK3-p38-MAPKAPK2 pathway promotes urokinase plasminogen activator mRNA stability in invasive breast cancer cells

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The primary structure of p38 gamma: a new member of p38 group of MAP kinases

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Involvement of the MKK6-p38gamma cascade in gamma-radiation-induced cell cycle arrest

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An inhibitor of p38 mitogen-activated protein kinase prevents insulin-stimulated glucose transport but not glucose transporter translocation in 3T3-L1 adipocytes and L6 myotubes

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Structural basis of inhibitor selectivity in MAP kinases

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A protein kinase involved in the regulation of inflammatory cytokine biosynthesis

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Molecular basis for p38 protein kinase inhibitor specificity

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p38 isoforms have opposite effects on AP-1-dependent transcription through regulation of c-Jun. The determinant roles of the isoforms in the p38 MAPK signal specificity

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Activation mechanisms of the urokinase-type plasminogen activator promoter by hepatocyte growth factor/scatter factor

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UV irradiation induces the murine urokinase-type plasminogen activator gene via the c-Jun N-terminal kinase signaling pathway: requirement of an AP1 enhancer element.

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Mitogen-activated protein (MAP) kinase regulates production of tumor necrosis factor-alpha and release of arachidonic acid in mast cells. Indications of communication between p38 and p42 MAP kinases

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P38 and activating transcription factor-2 involvement in osteoblast osmotic response to elevated extracellular glucose

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Role of distinct mitogen-activated protein kinase pathways and cooperation between Ets-2, ATF-2, and Jun family members in human urokinase-type plasminogen activator gene induction by interleukin-1 and tetradecanoyl phorbol acetate

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Prognostic significance of phosphorylated P38 mitogen-activated protein kinase and HER-2 expression in lymph node-positive breast carcinoma

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High Intensity ras Signaling Induces Premature Senescence by Activating p38 Pathway in Primary Human Fibroblasts

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Loss of oncogenic H-ras-induced cell cycle arrest and p38 mitogen-activated protein kinase activation by disruption of Gadd45a

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Sequential activation of the MEK-extracellular signal-regulated kinase and MKK3/6-p38 mitogen-activated protein kinase pathways mediates oncogenic ras-induced premature senescence

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PD 098059, an inhibitor of ERK1 activation, attenuates the in vivo invasiveness of head and neck squamous cell carcinoma

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PC12 cell neuronal differentiation is associated with prolonged p21ras activity and consequent prolonged ERK activity

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10.1016/J.YEXCR.2004.10.004

7 January 2021

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DOI

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7 November 2017

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