Role of mitogen-activated protein kinases and c-Jun/AP-1 trans-activating activity in the regulation of protease mRNAs and the malignant phenotype in NIH 3T3 fibroblasts. (Q42823190)

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scientific article published on January 1999

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English	Role of mitogen-activated protein kinases and c-Jun/AP-1 trans-activating activity in the regulation of protease mRNAs and the malignant phenotype in NIH 3T3 fibroblasts.	scientific article published on January 1999

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scholarly article

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8 December 2019

Role of mitogen-activated protein kinases and c-Jun/AP-1 trans-activating activity in the regulation of protease mRNAs and the malignant phenotype in NIH 3T3 fibroblasts (English)

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8 December 2019

J. Silvio Gutkind

4

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8 December 2019

author name string

M Janulis

1

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8 December 2019

S Silberman

2

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8 December 2019

A Ambegaokar

3

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8 December 2019

R M Schultz

5

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8 December 2019

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1 January 1999

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8 December 2019

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13

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Journal of Biological Chemistry

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8 December 2019

274

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8 December 2019

801-813

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8 December 2019

2

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8 December 2019

Signal-transducing protein phosphorylation cascades mediated by Ras/Rho proteins in the mammalian cell: the potential for multiplex signalling

1 reference

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21 January 2018

The Ras signal transduction pathway

1 reference

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21 January 2018

JNK1: a protein kinase stimulated by UV light and Ha-Ras that binds and phosphorylates the c-Jun activation domain

1 reference

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21 January 2018

Oncogenic Ras activates c-Jun via a separate pathway from the activation of extracellular signal-regulated kinases

1 reference

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21 January 2018

The stress-activated protein kinase subfamily of c-Jun kinases

1 reference

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Stimulation of 92-kDa gelatinase B promoter activity by ras is mitogen-activated protein kinase kinase 1-independent and requires multiple transcription factor binding sites including closely spaced PEA3/ets and AP-1 sequences.

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Characterization of downstream Ras signals that induce alternative protease-dependent invasive phenotypes

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Transforming p21ras mutants and c-Ets-2 activate the cyclin D1 promoter through distinguishable regions

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Requirement of an upstream AP-1 motif for the constitutive and phorbol ester-inducible expression of the urokinase-type plasminogen activator receptor gene

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Experimental metastasis in nude mice of NIH 3T3 cells containing various ras genes

1 reference

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21 January 2018

Cellular transformation and malignancy induced by ras require c-jun

1 reference

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21 January 2018

Transformation suppressor activity of a Jun transcription factor lacking its activation domain

1 reference

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21 January 2018

The Bcr-Abl leukemia oncogene activates Jun kinase and requires Jun for transformation

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Autophosphorylation of v-Ha-ras p21 is modulated by amino acid residue 12

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Mammalian and yeast ras gene products: biological function in their heterologous systems

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21 January 2018

Phorbol ester-inducible genes contain a common cis element recognized by a TPA-modulated trans-acting factor

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21 January 2018

Transfer of purified herpes virus thymidine kinase gene to cultured mouse cells

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21 January 2018

Ras-mediated transcription activation: analysis by transient cotransfection assays

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21 January 2018

Biological assays for Ras transformation

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21 January 2018

Transforming G protein-coupled receptors potently activate JNK (SAPK). Evidence for a divergence from the tyrosine kinase signaling pathway.

1 reference

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21 January 2018

The jun proto-oncogene is positively autoregulated by its product, Jun/AP-1

1 reference

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21 January 2018

Use of a reconstituted basement membrane to measure cell invasiveness and select for highly invasive tumor cells

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21 January 2018

Anisomycin-activated protein kinases p45 and p55 but not mitogen-activated protein kinases ERK-1 and -2 are implicated in the induction of c-fos and c-jun

1 reference

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21 January 2018

Deregulated expression of human c-jun transforms primary rat embryo cells in cooperation with an activated c-Ha-ras gene and transforms rat-1a cells as a single gene

1 reference

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21 January 2018

Stepwise transformation of rat embryo fibroblasts: c-Jun, JunB, or JunD can cooperate with Ras for focus formation, but a c-Jun-containing heterodimer is required for immortalization

1 reference

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21 January 2018

Mouse JunD negatively regulates fibroblast growth and antagonizes transformation by ras.

1 reference

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21 January 2018

The transactivating domain of the c-Jun proto-oncoprotein is required for cotransformation of rat embryo cells

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21 January 2018

The multistep nature of cancer

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Cyclins and cancer. II: Cyclin D and CDK inhibitors come of age

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Ras signalling linked to the cell-cycle machinery by the retinoblastoma protein

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Regulation of urokinase-type plasminogen activator expression by an ERK1-dependent signaling pathway in a squamous cell carcinoma cell line

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Role of SAPK/ERK kinase-1 in the stress-activated pathway regulating transcription factor c-Jun

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Expression of matrix metalloproteinase matrilysin (MMP-7) was induced by activated Ki-ras via AP-1 activation in SW1417 colon cancer cells

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