Amyloid-beta induces Smac release via AP-1/Bim activation in cerebral endothelial cells. (Q38361116)

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14 December 2019

title

Amyloid-beta induces Smac release via AP-1/Bim activation in cerebral endothelial cells (English)

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14 December 2019

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K J Yin

1

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14 December 2019

J-M Lee

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14 December 2019

S D Chen

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14 December 2019

J Xu

4

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14 December 2019

C Y Hsu

5

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14 December 2019

language of work or name

English

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publication date

1 November 2002

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14 December 2019

published in

Journal of Neuroscience

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14 December 2019

volume

22

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14 December 2019

page(s)

9764-9770

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14 December 2019

issue

22

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The cell death-promoting gene DP5, which interacts with the BCL2 family, is induced during neuronal apoptosis following exposure to amyloid beta protein

14 December 2019

cites work

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Inhibition of NF-kappaB potentiates amyloid beta-mediated neuronal apoptosis

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Proapoptotic Bcl-2 relative Bim required for certain apoptotic responses, leukocyte homeostasis, and to preclude autoimmunity

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Protein complexes activate distinct caspase cascades in death receptor and stress-induced apoptosis

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Role of perivascular cells and myocytes in vascular amyloidosis

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Oxidative stress induces intracellular accumulation of amyloid beta-protein (Abeta) in human neuroblastoma cells.

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Oxygen-glucose deprivation induces inducible nitric oxide synthase and nitrotyrosine expression in cerebral endothelial cells.

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Smac, a mitochondrial protein that promotes cytochrome c-dependent caspase activation by eliminating IAP inhibition

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Identification of DIABLO, a mammalian protein that promotes apoptosis by binding to and antagonizing IAP proteins

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Expression of the pro-apoptotic Bcl-2 family member Bim is regulated by the forkhead transcription factor FKHR-L1

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Cyclosporin A inhibits chromium(VI)-induced apoptosis and mitochondrial cytochrome c release and restores clonogenic survival in CHO cells.

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BH3-Only proteins-essential initiators of apoptotic cell death

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Amyloid-beta peptides are cytotoxic to oligodendrocytes.

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An anti-apoptotic protein human survivin is a direct inhibitor of caspase-3 and -7

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Induction of BIM, a proapoptotic BH3-only BCL-2 family member, is critical for neuronal apoptosis

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Dominant-negative c-Jun promotes neuronal survival by reducing BIM expression and inhibiting mitochondrial cytochrome c release

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CEP-1347/KT-7515, an inhibitor of SAPK/JNK pathway activation, promotes survival and blocks multiple events associated with Abeta-induced cortical neuron apoptosis

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Spontaneous changes in mitochondrial membrane potential in cultured neurons.

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Endonuclease G is an apoptotic DNase when released from mitochondria

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Amyloid beta peptide-induced cerebral endothelial cell death involves mitochondrial dysfunction and caspase activation

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Effect of the Alzheimer amyloid fragment Abeta(25-35) on Akt/PKB kinase and survival of PC12 cells

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The expanding role of mitochondria in apoptosis

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12 December 2020

Apoptosis-associated release of Smac/DIABLO from mitochondria requires active caspases and is blocked by Bcl-2

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12 December 2020

Rapid kinetics of tBid-induced cytochrome c and Smac/DIABLO release and mitochondrial depolarization

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12 December 2020

Smac is required for cytochrome c-induced apoptosis in prostate cancer LNCaP cells.

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12 December 2020

TRAIL-induced apoptosis requires Bax-dependent mitochondrial release of Smac/DIABLO.

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12 December 2020

Bcl-2 and Bcl-xL inhibit CD95-mediated apoptosis by preventing mitochondrial release of Smac/DIABLO and subsequent inactivation of X-linked inhibitor-of-apoptosis protein

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12 December 2020

Cell death regulation by the mammalian IAP antagonist Diablo/Smac.

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12 December 2020

Inhibition of TRAIL-induced apoptosis by Bcl-2 overexpression.

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12 December 2020

Receptor-linked hydrolysis of phosphoinositides and production of prostacyclin in cerebral endothelial cells

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12 December 2020

Regulation of jun-B messenger RNA and AP-1 activity by light and a circadian clock.

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12 December 2020

Transcriptional control by nuclear receptors

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12 December 2020

Neurotoxicity of a fragment of the amyloid precursor associated with Alzheimer's disease

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Fos and Jun: the AP-1 connection

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12 December 2020

Neuronal origin of a cerebral amyloid: neurofibrillary tangles of Alzheimer's disease contain the same protein as the amyloid of plaque cores and blood vessels

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The amyloid deposits in Alzheimer's disease: their nature and pathogenesis

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Platelet-derived growth factor induces rapid but transient expression of the c-fos gene and protein

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12 December 2020

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Hydrogen peroxide mediates amyloid beta protein toxicity

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12 December 2020

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12 December 2020

beta-Amyloid peptide-derived, oxygen-dependent free radicals inhibit glutamate uptake in cultured astrocytes: implications for Alzheimer's disease.

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12 December 2020

beta-Amyloid-mediated vasoactivity and vascular endothelial damage.

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12 December 2020

Mechanisms of neuronal degeneration in Alzheimer's disease

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12 December 2020

In vitro efficacy of morpholino-modified antisense oligomers directed against tumor necrosis factor-alpha mRNA

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12 December 2020

Amyloid beta-peptide induces cell monolayer albumin permeability, impairs glucose transport, and induces apoptosis in vascular endothelial cells.

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12 December 2020

Oxidative mechanisms in beta-amyloid cytotoxicity

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12 December 2020

Morpholino and phosphorothioate antisense oligomers compared in cell-free and in-cell systems

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12 December 2020

Morpholino antisense oligomers: design, preparation, and properties

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12 December 2020

X-linked IAP is a direct inhibitor of cell-death proteases

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12 December 2020

Superoxide free radical and intracellular calcium mediate A beta(1-42) induced endothelial toxicity.

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12 December 2020

beta-Amyloid induces cerebrovascular endothelial dysfunction in the rat brain.

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12 December 2020

Animal models of cerebral beta-amyloid angiopathy

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12 December 2020

The c-IAP-1 and c-IAP-2 proteins are direct inhibitors of specific caspases

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12 December 2020

Toxic effects of beta-amyloid(25-35) on immortalised rat brain endothelial cell: protection by carnosine, homocarnosine and beta-alanine

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12 December 2020

IAPs block apoptotic events induced by caspase-8 and cytochrome c by direct inhibition of distinct caspases

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12 December 2020

Fragments of amyloid beta induce apoptosis in vascular endothelial cells

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12 December 2020

Involvement of de novo ceramide biosynthesis in tumor necrosis factor-alpha/cycloheximide-induced cerebral endothelial cell death.

1 reference

12 December 2020

Bcl-2-family proteins: the role of the BH3 domain in apoptosis.

1 reference

12 December 2020

Mitochondria and apoptosis

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12 December 2020

Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

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12 December 2020

Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

1 reference

12 December 2020

Caspase cleaved BID targets mitochondria and is required for cytochrome c release, while BCL-XL prevents this release but not tumor necrosis factor-R1/Fas death

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12 December 2020

Suppression of postischemic hippocampal nerve growth factor expression by a c-fos antisense oligodeoxynucleotide.

1 reference

12 December 2020

IAP family proteins--suppressors of apoptosis

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12 December 2020

10.1523/JNEUROSCI.22-22-09764.2002

Identifiers

DOI

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:12427831%20AND%20SRC:MED&resulttype=core&format=json

14 December 2019

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14 December 2019

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