Tumor suppression at the mouse INK4a locus mediated by the alternative reading frame product p19ARF (Q29619663)

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30 April 2017

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ORCID Public Data File 2021

author name string

Kamijo T

1

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Zindy F

2

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Roussel MF

3

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Quelle DE

4

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30 April 2017

Downing JR

5

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Ashmun RA

6

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Grosveld G

7

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Sherr CJ

8

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publication date

28 November 1997

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30 April 2017

language of work or name

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30 April 2017

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volume

91

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issue

5

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page(s)

649-59

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Involvement of the cyclin-dependent kinase inhibitor p16 (INK4a) in replicative senescence of normal human fibroblasts

30 April 2017

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p53 alteration is a common event in the spontaneous immortalization of primary BALB/c murine embryo fibroblasts

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A cell cycle regulator potentially involved in genesis of many tumor types

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p53-deficient mice are extremely susceptible to radiation-induced tumorigenesis

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p53: puzzle and paradigm

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Tumour-derived p16 alleles encoding proteins defective in cell-cycle inhibition

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Mutations of the INK4a locus in squamous cell carcinomas of human skin

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Wild-type p53 is a cell cycle checkpoint determinant following irradiation

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p53, the cellular gatekeeper for growth and division

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Cooperating oncogenes converge to regulate cyclin/cdk complexes

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Retinoblastoma-protein-dependent cell-cycle inhibition by the tumour suppressor p16

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5′ CpG island methylation is associated with transcriptional silencing of the tumour suppressor p16/CDKN2/MTS1 in human cancers

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Deletions of the cyclin-dependent kinase-4 inhibitor gene in multiple human cancers

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Cloning of senescent cell-derived inhibitors of DNA synthesis using an expression screen

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Temperature-sensitive mutants of p16CDKN2 associated with familial melanoma

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Production of high-titer helper-free retroviruses by transient transfection

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Compilation of somatic mutations of the CDKN2 gene in human cancers: non-random distribution of base substitutions

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Alternative reading frames of the INK4a tumor suppressor gene encode two unrelated proteins capable of inducing cell cycle arrest

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Cancer-associated mutations at the INK4a locus cancel cell cycle arrest by p16INK4a but not by the alternative reading frame protein p19ARF.

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Role of the INK4a locus in tumor suppression and cell mortality

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Identifiers

DOI

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Consolidated OpenCitations Corpus – April 2017

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Consolidated OpenCitations Corpus – April 2017

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=9393858

PubMed publication ID

2 references

30 April 2017

Consolidated OpenCitations Corpus – April 2017