Mice Lacking p21CIP1/WAF1 undergo normal development, but are defective in G1 checkpoint control (Q29616465)

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30 April 2017

Mice Lacking p21CIP1/WAF1 undergo normal development, but are defective in G1 checkpoint control (English)

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30 July 2024

author name string

C. Deng

1

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P. Zhang

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J. W. Harper

3

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S. J. Elledge

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P. Leder

5

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publication date

25 August 1995

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language of work or name

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https://doi.org/10.1016/0092-8674(95)90039-x

30 April 2017

work available at URL

online access status

open access

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30 July 2024

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volume

82

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issue

4

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page(s)

675-84

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A single ataxia telangiectasia gene with a product similar to PI-3 kinase

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Accumulation of wild-type p53 protein upon gamma-irradiation induces a G2 arrest-dependent immunoglobulin kappa light chain gene expression.

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WAF1, a potential mediator of p53 tumor suppression

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WAF1/CIP1 is induced in p53-mediated G1 arrest and apoptosis

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Cdk inhibitors: on the threshold of checkpoints and development

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Cell cycle regulation of CDK2 activity by phosphorylation of Thr160 and Tyr15

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Expression of wild-type p53 is not compatible with continued growth of p53-negative tumor cells

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A mammalian cell cycle checkpoint pathway utilizing p53 and GADD45 is defective in ataxia-telangiectasia

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