Microglia-derived TGF-beta as an important regulator of glioblastoma invasion--an inhibition of TGF-beta-dependent effects by shRNA against human TGF-beta type II receptor. (Q52581027)

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scientific article published on 6 August 2007

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English	Microglia-derived TGF-beta as an important regulator of glioblastoma invasion--an inhibition of TGF-beta-dependent effects by shRNA against human TGF-beta type II receptor.	scientific article published on 6 August 2007

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scholarly article

1 reference

Europe PubMed Central

3 May 2018

http://europepmc.org/abstract/MED/17684491

Microglia-derived TGF-beta as an important regulator of glioblastoma invasion--an inhibition of TGF-beta-dependent effects by shRNA against human TGF-beta type II receptor. (English)

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Europe PubMed Central

3 May 2018

http://europepmc.org/abstract/MED/17684491

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microglial inhibitor

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author name string

Wesolowska A

1

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3 May 2018

http://europepmc.org/abstract/MED/17684491

Kwiatkowska A

2

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Europe PubMed Central

3 May 2018

http://europepmc.org/abstract/MED/17684491

Slomnicki L

3

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Europe PubMed Central

3 May 2018

http://europepmc.org/abstract/MED/17684491

Dembinski M

4

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Europe PubMed Central

3 May 2018

http://europepmc.org/abstract/MED/17684491

Master A

5

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Europe PubMed Central

3 May 2018

http://europepmc.org/abstract/MED/17684491

Sliwa M

6

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Europe PubMed Central

3 May 2018

http://europepmc.org/abstract/MED/17684491

Franciszkiewicz K

7

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Europe PubMed Central

3 May 2018

http://europepmc.org/abstract/MED/17684491

Chouaib S

8

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Europe PubMed Central

3 May 2018

http://europepmc.org/abstract/MED/17684491

Kaminska B

9

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3 May 2018

http://europepmc.org/abstract/MED/17684491

language of work or name

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publication date

6 August 2007

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3 May 2018

http://europepmc.org/abstract/MED/17684491

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Europe PubMed Central

3 May 2018

http://europepmc.org/abstract/MED/17684491

27

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3 May 2018

http://europepmc.org/abstract/MED/17684491

918-930

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http://europepmc.org/abstract/MED/17684491

7

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Europe PubMed Central

3 May 2018

http://europepmc.org/abstract/MED/17684491

https://scigraph.springernature.com/pub.10.1038/sj.onc.1210683

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Mads and Smads in TGF beta signalling

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Role of microglia in glioma biology

1 reference

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A system for stable expression of short interfering RNAs in mammalian cells

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Inhibition of Akt kinase signalling and activation of Forkhead are indispensable for upregulation of FasL expression in apoptosis of glioma cells.

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SB-505124 is a selective inhibitor of transforming growth factor-beta type I receptors ALK4, ALK5, and ALK7.

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The tumor suppressor Smad4 is required for transforming growth factor beta-induced epithelial to mesenchymal transition and bone metastasis of breast cancer cells

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Direct binding of Smad3 and Smad4 to critical TGF beta-inducible elements in the promoter of human plasminogen activator inhibitor-type 1 gene

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Smad-dependent and Smad-independent pathways in TGF-beta family signalling

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Smads: transcriptional activators of TGF-beta responses

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Duplexes of 21-nucleotide RNAs mediate RNA interference in cultured mammalian cells

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RNA interference targeting transforming growth factor-beta enhances NKG2D-mediated antiglioma immune response, inhibits glioma cell migration and invasiveness, and abrogates tumorigenicity in vivo.

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TGF-beta signalling from cell membrane to nucleus through SMAD proteins

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Local gene knockdown in the brain using viral-mediated RNA interference.

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Single small-interfering RNA expression vector for silencing multiple transforming growth factor-beta pathway components

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The role of transforming growth factor beta in glioma progression

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In situ detection of transforming growth factor-beta mRNA in experimental rat glioma and reactive glial cells.

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Expression of TGF-beta isoforms, TGF-beta receptors, and SMAD molecules at different stages of human glioma.

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Transforming growth factor beta 1 null mutation in mice causes excessive inflammatory response and early death

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Inhibition of cathepsin B and MMP-9 gene expression in glioblastoma cell line via RNA interference reduces tumor cell invasion, tumor growth and angiogenesis.

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Microglia and macrophages are major sources of locally produced transforming growth factor-beta1 after transient middle cerebral artery occlusion in rats.

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Transforming growth factor-beta controls development, homeostasis, and tolerance of T cells by regulatory T cell-dependent and -independent mechanisms

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Transforming growth factor-beta 1 in the rat brain: increase after injury and inhibition of astrocyte proliferation

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Role of tumor-associated macrophages in tumor progression and invasion

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Cellular mechanisms of fatal early-onset autoimmunity in mice with the T cell-specific targeting of transforming growth factor-beta receptor

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TGF-beta receptor-mediated signalling through Smad2, Smad3 and Smad4

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TGFbeta signaling is necessary for carcinoma cell invasiveness and metastasis

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Adenovirus-delivered antisense RNA and shRNA exhibit different silencing efficiencies for the endogenous transforming growth factor-beta (TGF-beta) type II receptor

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Epidemiology and etiology of gliomas

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Intravenous, non-viral RNAi gene therapy of brain cancer

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Malignant glioma biology: role for TGF-beta in growth, motility, angiogenesis, and immune escape

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Tumour-educated macrophages promote tumour progression and metastasis

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Reduction of tumor cell migration and metastasis by adenoviral gene transfer of plasminogen activator inhibitors

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Molecular mechanisms of glioma invasiveness: the role of proteases

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Targeted tumor therapy with the TGF-beta 2 antisense compound AP 12009.

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Targeted disruption of the mouse transforming growth factor-beta 1 gene results in multifocal inflammatory disease

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The invasion promoting effect of microglia on glioblastoma cells is inhibited by cyclosporin A.

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Smad4/DPC4 and Smad3 mediate transforming growth factor-beta (TGF-beta) signaling through direct binding to a novel TGF-beta-responsive element in the human plasminogen activator inhibitor-1 promoter

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Malignant cells, directors of the malignant process: role of transforming growth factor-beta

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SD-208, a novel transforming growth factor beta receptor I kinase inhibitor, inhibits growth and invasiveness and enhances immunogenicity of murine and human glioma cells in vitro and in vivo

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Microglia function in brain tumors

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Glioma cell invasion: regulation of metalloproteinase activity by TGF-beta.

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Enhanced expression of transforming growth factor-beta and its type-I and type-II receptors in human glioblastoma.

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Targeted disruption of SMAD3 results in impaired mucosal immunity and diminished T cell responsiveness to TGF-beta

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Development of TGF-beta signalling inhibitors for cancer therapy

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TGF-beta receptor-activated p38 MAP kinase mediates Smad-independent TGF-beta responses

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RNAi: double-stranded RNA directs the ATP-dependent cleavage of mRNA at 21 to 23 nucleotide intervals

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Cyclosporine a induces growth arrest or programmed cell death of human glioma cells.

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Microglia stimulate the invasiveness of glioma cells by increasing the activity of metalloprotease-2.

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7 January 2021

based on heuristic

inferred from DOI database lookup

Blockade of TGF-beta inhibits mammary tumor cell viability, migration, and metastases.

1 reference

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7 January 2021

based on heuristic

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Matrix metalloproteinases and tissue inhibitors of metalloproteinases in human gliomas

1 reference

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7 January 2021

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Expression of transforming-growth-factor (TGF)-beta receptors and Smad proteins in glioblastoma cell lines with distinct responses to TGF-beta1

1 reference

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7 January 2021

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inferred from DOI database lookup

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10.1038/SJ.ONC.1210683

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Europe PubMed Central

3 May 2018

http://europepmc.org/abstract/MED/17684491

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Europe PubMed Central

3 May 2018

http://europepmc.org/abstract/MED/17684491

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