Brain-derived neurotrophic factor protection of cortical neurons from serum withdrawal-induced apoptosis is inhibited by cAMP. (Q44477195)

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4 November 2019

title

Brain-derived neurotrophic factor protection of cortical neurons from serum withdrawal-induced apoptosis is inhibited by cAMP (English)

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4 November 2019

author name string

Steven Poser

1

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4 November 2019

Soren Impey

2

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4 November 2019

Zhengui Xia

3

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4 November 2019

Daniel R Storm

4

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4 November 2019

language of work or name

English

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publication date

1 June 2003

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4 November 2019

published in

Journal of Neuroscience

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4 November 2019

volume

23

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4 November 2019

issue

11

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4 November 2019

page(s)

4420-4427

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Activation of serum- and glucocorticoid-regulated protein kinase by agonists that activate phosphatidylinositide 3-kinase is mediated by 3-phosphoinositide-dependent protein kinase-1 (PDK1) and PDK2

4 November 2019

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Neuroprotection by brain-derived neurotrophic factor is mediated by extracellular signal-regulated kinase and phosphatidylinositol 3-kinase.

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Depolarization and neurotrophins converge on the phosphatidylinositol 3-kinase-Akt pathway to synergistically regulate neuronal survival

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Role of glycogen synthase kinase-3beta in neuronal apoptosis induced by trophic withdrawal

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SRF-dependent gene expression is required for PI3-kinase-regulated cell proliferation

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Spatially and Functionally Distinct Roles of the PI3-K Effector Pathway during NGF Signaling in Sympathetic Neurons

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Cyclic AMP promotes neuronal survival by phosphorylation of glycogen synthase kinase 3beta.

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Activation of phosphatidylinositol-3 kinase (PI-3K) and extracellular regulated kinases (Erk1/2) is involved in muscarinic receptor-mediated DNA synthesis in neural progenitor cells.

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Cyclic AMP inhibits Akt activity by blocking the membrane localization of PDK1.

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Cyclic AMP inhibits extracellular signal-regulated kinase and phosphatidylinositol 3-kinase/Akt pathways by inhibiting Rap1

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Differential signaling of cyclic AMP: opposing effects of exchange protein directly activated by cyclic AMP and cAMP-dependent protein kinase on protein kinase B activation

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DESTRUCTION OF THE SYMPATHETIC GANGLIA IN MAMMALS BY AN ANTISERUM TO A NERVE-GROWTH PROTEIN.

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Impulse activity and the patterning of connections during CNS development

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Rapid enhancement of cAMP accumulation in rat brain particulate fraction after ischaemia.

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12 December 2020

Trophic factors and neuronal survival

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12 December 2020

cAMP analogs promote survival and neurite outgrowth in cultures of rat sympathetic and sensory neurons independently of nerve growth factor

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12 December 2020

cAMP analog antagonists of cAMP action

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The structural organization of layer IV in the somatosensory region (SI) of mouse cerebral cortex. The description of a cortical field composed of discrete cytoarchitectonic units

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Opposing effects of ERK and JNK-p38 MAP kinases on apoptosis

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Cyclic AMP-increasing agents interfere with chemoattractant-induced respiratory burst in neutrophils as a result of the inhibition of phosphatidylinositol 3-kinase rather than receptor-operated Ca2+ influx

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Characterization of the signaling interactions that promote the survival and growth of developing retinal ganglion cells in culture

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Requirement for phosphatidylinositol-3 kinase in the prevention of apoptosis by nerve growth factor

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12 December 2020

Requirement for BDNF in activity-dependent survival of cortical neurons.

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12 December 2020

Two pathways for serum regulation of the c-fos serum response element require specific sequence elements and a minimal domain of serum response factor

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12 December 2020

Programmed cell death and the control of cell survival: lessons from the nervous system

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12 December 2020

Induction of apoptosis in cerebellar granule neurons by low potassium: inhibition of death by insulin-like growth factor I and cAMP.

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12 December 2020

IRS-1 is a common element in insulin and insulin-like growth factor-I signaling to the phosphatidylinositol 3'-kinase

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12 December 2020

Membrane localization of phosphatidylinositol 3-kinase is sufficient to activate multiple signal-transducing kinase pathways

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12 December 2020

Intracellular signaling pathways activated by neurotrophic factors

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12 December 2020

Mechanism of activation of protein kinase B by insulin and IGF-1

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12 December 2020

Pituitary adenylate cyclase-activating polypeptide (PACAP-38) protects cerebellar granule neurons from apoptosis by activating the mitogen-activated protein kinase (MAP kinase) pathway.

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12 December 2020

Insulin-like Growth Factor 1 Inhibits Apoptosis Using the Phosphatidylinositol 3′-Kinase and Mitogen-activated Protein Kinase Pathways

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12 December 2020

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12 December 2020

Inhibition of phosphatidylinositol 3-kinase activity blocks depolarization- and insulin-like growth factor I-mediated survival of cerebellar granule cells.

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12 December 2020

Characterization of a 3-phosphoinositide-dependent protein kinase which phosphorylates and activates protein kinase Balpha

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12 December 2020

cAMP activates MAP kinase and Elk-1 through a B-Raf- and Rap1-dependent pathway

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12 December 2020

Pituitary adenylate cyclase-activating polypeptide is an autocrine inhibitor of mitosis in cultured cortical precursor cells

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12 December 2020

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12 December 2020

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12 December 2020

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12 December 2020

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12 December 2020

Akt phosphorylation of BAD couples survival signals to the cell-intrinsic death machinery

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12 December 2020

Phosphatidylinositol 3-kinase and Akt protein kinase are necessary and sufficient for the survival of nerve growth factor-dependent sympathetic neurons.

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12 December 2020

Neurotrophins rescue cerebellar granule neurons from oxidative stress-mediated apoptotic death: selective involvement of phosphatidylinositol 3-kinase and the mitogen-activated protein kinase pathway.

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12 December 2020

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12 December 2020

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12 December 2020

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12 December 2020

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12 December 2020

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12 December 2020

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10.1523/JNEUROSCI.23-11-04420.2003

Identifiers

DOI

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4 November 2019

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4 November 2019

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