LMP1 of Epstein-Barr virus suppresses cellular senescence associated with the inhibition of p16INK4a expression (Q40881096)

24 September 2017

LMP1 of Epstein-Barr virus suppresses cellular senescence associated with the inhibition of p16INK4a expression (English)

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24 September 2017

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Yang X

1

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24 September 2017

He Z

series ordinal

2

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24 September 2017

Xin B

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3

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24 September 2017

Cao L

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4

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24 September 2017

language of work or name

English

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publication date

1 April 2000

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24 September 2017

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24 September 2017

19

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24 September 2017

2002-2013

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24 September 2017

16

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https://scigraph.springernature.com/pub.10.1038/sj.onc.1203515

24 September 2017

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Epstein-Barr virus nuclear protein 2 is a key determinant of lymphocyte transformation

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A biomarker that identifies senescent human cells in culture and in aging skin in vivo

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VIRUS PARTICLES IN CULTURED LYMPHOBLASTS FROM BURKITT'S LYMPHOMA

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Dominant inhibitory mutations in the Mg(2+)-binding site of RasH prevent its activation by GTP

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In vitro establishment is not a sufficient prerequisite for transformation by activated ras oncogenes.

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Latent membrane protein 1 of Epstein-Barr virus interacts with JAK3 and activates STAT proteins

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Latent membrane protein 1 of Epstein-Barr virus mimics a constitutively active receptor molecule.

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Replication of Epstein-Barr virus within the epithelial cells of oral "hairy" leukoplakia, an AIDS-associated lesion

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Genetic analysis of immortalizing functions of Epstein-Barr virus in human B lymphocytes

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Regulation of p16CDKN2 expression and its implications for cell immortalization and senescence

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The Epstein-Barr virus LMP1 amino acid sequence that engages tumor necrosis factor receptor associated factors is critical for primary B lymphocyte growth transformation

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Epstein-Barr virus latent membrane protein 1 is essential for B-lymphocyte growth transformation

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Epstein-Barr virus latent membrane protein-1 triggers AP-1 activity via the c-Jun N-terminal kinase cascade

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Epstein-Barr virus-mediated B-cell proliferation is dependent upon latent membrane protein 1, which simulates an activated CD40 receptor

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Premature senescence involving p53 and p16 is activated in response to constitutive MEK/MAPK mitogenic signaling

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The Epstein-Barr virus transforming protein LMP1 engages signaling proteins for the tumor necrosis factor receptor family

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Murine fibroblasts lacking p21 undergo senescence and are resistant to transformation by oncogenic Ras.

21 January 2018

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21 January 2018

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21 January 2018

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The INK4 family of cell cycle inhibitors in cancer

21 January 2018

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Role of the INK4a locus in tumor suppression and cell mortality.

21 January 2018

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Oncogenic ras provokes premature cell senescence associated with accumulation of p53 and p16INK4a

21 January 2018

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Origins of G1 arrest in senescent human fibroblasts

21 January 2018

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Differential roles for cyclin-dependent kinase inhibitors p21 and p16 in the mechanisms of senescence and differentiation in human fibroblasts

21 January 2018

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An infrequent point mutation of the p53 gene in human nasopharyngeal carcinoma

21 January 2018

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Induction of senescence in human malignant glioma cells by p16INK4A.

21 January 2018

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An EBV membrane protein expressed in immortalized lymphocytes transforms established rodent cells.

21 January 2018

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LMP1 of Epstein-Barr virus induces proliferation of primary mouse embryonic fibroblasts and cooperatively transforms the cells with a p16-insensitive CDK4 oncogene

21 January 2018

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Senescence of human fibroblasts induced by oncogenic Raf.

21 January 2018

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Expression of the p16INK4a tumor suppressor versus other INK4 family members during mouse development and aging.

21 January 2018

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Presence of EB virus nucleic acid homology in a "virus-free" line of Burkitt tumour cells

21 January 2018

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The transforming domain alone of the latent membrane protein of Epstein-Barr virus is toxic to cells when expressed at high levels

21 January 2018

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7 January 2021

The Epstein-Barr virus LMP1 cytoplasmic carboxy terminus is essential for B-lymphocyte transformation; fibroblast cocultivation complements a critical function within the terminal 155 residues.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1203515

7 January 2021

Human T-cell leukemia virus type 1 Tax releases cell cycle arrest induced by p16INK4a

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1203515

7 January 2021

The Epstein-Barr virus nuclear protein encoded by the leader of the EBNA RNAs is important in B-lymphocyte transformation

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1203515

7 January 2021

Inhibitors of cyclin-dependent kinases induce features of replicative senescence in early passage human diploid fibroblasts

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1203515

7 January 2021

All three domains of the Epstein-Barr virus-encoded latent membrane protein LMP-1 are required for transformation of rat-1 fibroblasts.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1203515

7 January 2021

Identifiers

DOI

10.1038/SJ.ONC.1203515

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Dimensions Publication ID

24 September 2017

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1 reference