BH3-domain mimetic compound BH3I-2' induces rapid damage to the inner mitochondrial membrane prior to the cytochrome c release from mitochondria. (Q40655646)

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scientific article published on April 2003

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English	BH3-domain mimetic compound BH3I-2' induces rapid damage to the inner mitochondrial membrane prior to the cytochrome c release from mitochondria.	scientific article published on April 2003

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2 November 2019

BH3-domain mimetic compound BH3I-2' induces rapid damage to the inner mitochondrial membrane prior to the cytochrome c release from mitochondria (English)

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2 November 2019

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2 November 2019

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2 November 2019

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Wan-Yu Feng

1

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2 November 2019

Yasmeen Patwari

3

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2 November 2019

Samir G Agrawal

4

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2 November 2019

Adrian C Newland

5

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2 November 2019

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1 April 2003

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2 November 2019

British Journal of Haematology

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2 November 2019

121

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2 November 2019

2

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2 November 2019

332-340

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2 November 2019

Life-or-death decisions by the Bcl-2 protein family.

1 reference

https://api.crossref.org/works/10.1046%2FJ.1365-2141.2003.04268.X

21 January 2018

Suppression of apoptosis by overexpression of Bcl-2 or Bcl-xL promotes survival and mutagenesis after oxidative damage.

1 reference

https://api.crossref.org/works/10.1046%2FJ.1365-2141.2003.04268.X

21 January 2018

Identification of small-molecule inhibitors of interaction between the BH3 domain and Bcl-xL

1 reference

https://api.crossref.org/works/10.1046%2FJ.1365-2141.2003.04268.X

21 January 2018

Bid-induced conformational change of Bax is responsible for mitochondrial cytochrome c release during apoptosis

1 reference

https://api.crossref.org/works/10.1046%2FJ.1365-2141.2003.04268.X

21 January 2018

Bcl-2 is an inner mitochondrial membrane protein that blocks programmed cell death

1 reference

https://api.crossref.org/works/10.1046%2FJ.1365-2141.2003.04268.X

21 January 2018

Intracellular distribution of the fluorescent dye nonyl acridine orange responds to the mitochondrial membrane potential: implications for assays of cardiolipin and mitochondrial mass

1 reference

https://api.crossref.org/works/10.1046%2FJ.1365-2141.2003.04268.X

21 January 2018

Trail-induced apoptosis in Type I leukemic cells is not enhanced by overexpression of bax.

1 reference

https://api.crossref.org/works/10.1046%2FJ.1365-2141.2003.04268.X

21 January 2018

Bax translocation is crucial for the sensitivity of leukaemic cells to etoposide-induced apoptosis

1 reference

https://api.crossref.org/works/10.1046%2FJ.1365-2141.2003.04268.X

21 January 2018

Bcl-2-family proteins: the role of the BH3 domain in apoptosis

1 reference

https://api.crossref.org/works/10.1046%2FJ.1365-2141.2003.04268.X

21 January 2018

The pro-apoptotic Bcl-2 family member tBid localizes to mitochondrial contact sites

1 reference

https://api.crossref.org/works/10.1046%2FJ.1365-2141.2003.04268.X

21 January 2018

Identification of novel isoforms of the BH3 domain protein Bim which directly activate Bax to trigger apoptosis

1 reference

https://api.crossref.org/works/10.1046%2FJ.1365-2141.2003.04268.X

21 January 2018

Bim: a novel member of the Bcl-2 family that promotes apoptosis

1 reference

https://api.crossref.org/works/10.1046%2FJ.1365-2141.2003.04268.X

21 January 2018

Proapoptotic BH3-only Bcl-2 family members induce cytochrome c release, but not mitochondrial membrane potential loss, and do not directly modulate voltage-dependent anion channel activity

1 reference

https://api.crossref.org/works/10.1046%2FJ.1365-2141.2003.04268.X

21 January 2018

Prevention of apoptosis by Bcl-2: release of cytochrome c from mitochondria blocked

1 reference

https://api.crossref.org/works/10.1046%2FJ.1365-2141.2003.04268.X

21 January 2018

Sequential reduction of mitochondrial transmembrane potential and generation of reactive oxygen species in early programmed cell death

1 reference

https://api.crossref.org/works/10.1046%2FJ.1365-2141.2003.04268.X

21 January 2018

Bid acts on the permeability transition pore complex to induce apoptosis

1 reference

https://api.crossref.org/works/10.1046%2FJ.1365-2141.2003.04268.X

21 January 2018

BH3 domain of BAD is required for heterodimerization with BCL-XL and pro-apoptotic activity

1 reference

https://api.crossref.org/works/10.1046%2FJ.1365-2141.2003.04268.X

21 January 2018

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