Life-or-death decisions by the Bcl-2 protein family. (Q31879227)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:11165519%20AND%20SRC:MED&resulttype=core&format=json

3 November 2019

title

Life-or-death decisions by the Bcl-2 protein family (English)

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3 November 2019

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1

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3 November 2019

Suzanne Cory

series ordinal

2

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3 November 2019

author name string

Adams JM

series ordinal

1

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4 July 2017

Cory S

series ordinal

2

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4 July 2017

publication date

1 January 2001

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3 November 2019

number of pages

6

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Trends in Biochemical Sciences

inferred from page(s)

published in

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3 November 2019

volume

26

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3 November 2019

issue

1

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3 November 2019

page(s)

61-66

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publication type of scholarly work

3 November 2019

review article

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The Bcl-2 protein family: arbiters of cell survival

cites work

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7 January 2021

Mitochondria and apoptosis

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7 January 2021

BCL-2 family members and the mitochondria in apoptosis

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Biochemical pathways of caspase activation during apoptosis

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7 January 2021

Bcl-2 proteins: regulators of apoptosis or of mitochondrial homeostasis?

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7 January 2021

The biochemistry of apoptosis

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IAP family proteins--suppressors of apoptosis

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7 January 2021

Identification of DIABLO, a mammalian protein that promotes apoptosis by binding to and antagonizing IAP proteins

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7 January 2021

Smac, a mitochondrial protein that promotes cytochrome c-dependent caspase activation by eliminating IAP inhibition

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7 January 2021

Bcl-2 gene promotes haemopoietic cell survival and cooperates with c-myc to immortalize pre-B cells

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7 January 2021

Bcl-2-family proteins: the role of the BH3 domain in apoptosis

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7 January 2021

X-ray and NMR structure of human Bcl-xL, an inhibitor of programmed cell death

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7 January 2021

Structure of Bcl-xL-Bak peptide complex: recognition between regulators of apoptosis

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7 January 2021

Structure of Bax: coregulation of dimer formation and intracellular localization

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7 January 2021

Bcl-xL regulates apoptosis by heterodimerization-dependent and -independent mechanisms

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7 January 2021

The C. elegans Protein EGL-1 Is Required for Programmed Cell Death and Interacts with the Bcl-2–like Protein CED-9

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7 January 2021

Proapoptotic Bcl-2 relative Bim required for certain apoptotic responses, leukocyte homeostasis, and to preclude autoimmunity

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7 January 2021

Bid-deficient mice are resistant to Fas-induced hepatocellular apoptosis

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7 January 2021

The cell death-promoting gene DP5, which interacts with the BCL2 family, is induced during neuronal apoptosis following exposure to amyloid beta protein

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7 January 2021

Noxa, a BH3-only member of the Bcl-2 family and candidate mediator of p53-induced apoptosis

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7 January 2021

Bid, a Bcl2 Interacting Protein, Mediates Cytochrome c Release from Mitochondria in Response to Activation of Cell Surface Death Receptors

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7 January 2021

The proapoptotic activity of the Bcl-2 family member Bim is regulated by interaction with the dynein motor complex

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7 January 2021

Serine phosphorylation of death agonist BAD in response to survival factor results in binding to 14-3-3 not BCL-X(L)

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7 January 2021

14-3-3 proteins and survival kinases cooperate to inactivate BAD by BH3 domain phosphorylation

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7 January 2021

Bax-deficient mice with lymphoid hyperplasia and male germ cell death

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7 January 2021

Withdrawal of IL-7 induces Bax translocation from cytosol to mitochondria through a rise in intracellular pH.

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7 January 2021

Bid induces the oligomerization and insertion of Bax into the outer mitochondrial membrane

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7 January 2021

Movement of Bax from the Cytosol to Mitochondria during Apoptosis

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7 January 2021

Bcl-2 and Fas/APO-1 regulate distinct pathways to lymphocyte apoptosis

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7 January 2021

Translocation of C. elegans CED-4 to nuclear membranes during programmed cell death

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7 January 2021

Bcl-2 family members do not inhibit apoptosis by binding the caspase activator Apaf-1

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7 January 2021

Pro-apoptotic apoptosis protease-activating factor 1 (Apaf-1) has a cytoplasmic localization distinct from Bcl-2 or Bcl-x(L).

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7 January 2021

C. elegans cell survival gene ced-9 encodes a functional homolog of the mammalian proto-oncogene bcl-2.

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7 January 2021

Apoptotic protease activating factor 1 (Apaf-1)-independent cell death suppression by Bcl-2.

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7 January 2021

Induction of apoptotic program in cell-free extracts: requirement for dATP and cytochrome c

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7 January 2021

Apaf1 is required for mitochondrial pathways of apoptosis and brain development

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7 January 2021

Caspases: their intracellular localization and translocation during apoptosis

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7 January 2021

Molecular characterization of mitochondrial apoptosis-inducing factor

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7 January 2021

Cytochrome c deficiency causes embryonic lethality and attenuates stress-induced apoptosis

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7 January 2021

The mitochondrial permeability transition pore and its role in cell death

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7 January 2021

Bcl-xL prevents cell death following growth factor withdrawal by facilitating mitochondrial ATP/ADP exchange

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7 January 2021

Outer mitochondrial membrane permeability can regulate coupled respiration and cell survival

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7 January 2021

Bcl-xL Regulates the Membrane Potential and Volume Homeostasis of Mitochondria

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7 January 2021

Preservation of mitochondrial structure and function after Bid- or Bax-mediated cytochrome c release

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7 January 2021

Changes in intramitochondrial and cytosolic pH: early events that modulate caspase activation during apoptosis

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7 January 2021

Bax and adenine nucleotide translocator cooperate in the mitochondrial control of apoptosis

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7 January 2021

Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC

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7 January 2021

Bcl-2 retards cell cycle entry through p27(Kip1), pRB relative p130, and altered E2F regulation

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7 January 2021

10.1016/S0968-0004(00)01740-0

Identifiers

DOI

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:11165519%20AND%20SRC:MED&resulttype=core&format=json

3 November 2019

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