A polar mechanism coordinates different regions of alternative splicing within a single gene. (Q40390684)

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English	A polar mechanism coordinates different regions of alternative splicing within a single gene.	scientific article

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4 January 2020

A polar mechanism coordinates different regions of alternative splicing within a single gene (English)

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4 January 2020

alternative mRNA splicing, via spliceosome

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based on heuristic

inferred from title

Mikhail Gelfand

3

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4 January 2020

Federico Pelisch

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4 January 2020

Andrés F. Muro

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4 January 2020

Alberto Kornblihtt

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4 January 2020

Alexei D Neverov

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object named as

Alexei D Neverov

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author name string

Juan P Fededa

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4 January 2020

Ezequiel Petrillo

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4 January 2020

Sebastián Kadener

5

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4 January 2020

Guadalupe Nogués

6

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4 January 2020

Francisco E Baralle

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publication date

1 August 2005

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4 January 2020

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19

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393-404

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4 January 2020

3

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4 January 2020

Coordinate regulation of transcription and splicing by steroid receptor coregulators

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Coupling of transcription with alternative splicing: RNA pol II promoters modulate SF2/ASF and 9G8 effects on an exonic splicing enhancer

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The EIIIA segment of fibronectin is a ligand for integrins alpha 9beta 1 and alpha 4beta 1 providing a novel mechanism for regulating cell adhesion by alternative splicing.

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Alternatively spliced EDA segment regulates fibronectin-dependent cell cycle progression and mitogenic signal transduction

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A slow RNA polymerase II affects alternative splicing in vivo

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Intricate combinatorial patterns of exon splicing generate multiple regulated troponin T isoforms from a single gene.

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Genomic variants in exons and introns: identifying the splicing spoilers

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An extensive network of coupling among gene expression machines

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Alternative splicing: multiple control mechanisms and involvement in human disease

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Regulation of fibronectin EDA exon alternative splicing: possible role of RNA secondary structure for enhancer display.

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Role of the modular domains of SR proteins in subnuclear localization and alternative splicing specificity

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Impaired motor coordination in mice lacking the EDA exon of the fibronectin gene.

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The mRNA assembly line: transcription and processing machines in the same factory

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Evidence for the widespread coupling of alternative splicing and nonsense-mediated mRNA decay in humans

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P-TEFb, a cyclin-dependent kinase controlling elongation by RNA polymerase II

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Split genes and RNA splicing

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Exon recognition in vertebrate splicing

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Perturbation of transcription elongation influences the fidelity of internal exon inclusion in Saccharomyces cerevisiae.

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Regulation of the fibronectin EDA exon alternative splicing. Cooperative role of the exonic enhancer element and the 5' splicing site

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Influence of polymerase II processivity on alternative splicing depends on splice site strength.

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A novel bipartite splicing enhancer modulates the differential processing of the human fibronectin EDA exon

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The fibronectin gene as a model for splicing and transcription studies.

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Splicing of constitutive upstream introns is essential for the recognition of intra-exonic suboptimal splice sites in the thrombopoietin gene

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Integrating mRNA processing with transcription

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Initial sequencing and analysis of the human genome

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Multiple links between transcription and splicing

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Transcriptional activators differ in their abilities to control alternative splicing

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Listening to silence and understanding nonsense: exonic mutations that affect splicing

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Antagonistic effects of T-Ag and VP16 reveal a role for RNA pol II elongation on alternative splicing

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On the importance of being co-transcriptional

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Functional association between promoter structure and transcript alternative splicing.

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RNA folding affects the recruitment of SR proteins by mouse and human polypurinic enhancer elements in the fibronectin EDA exon

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Regulated splicing of the fibronectin EDA exon is essential for proper skin wound healing and normal lifespan

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Human fibronectin: cell specific alternative mRNA splicing generates polypeptide chains differing in the number of internal repeats.

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