Demethylation of a LINE-1 antisense promoter in the cMet locus impairs Met signalling through induction of illegitimate transcription (Q39690744)

14 September 2017

Demethylation of a LINE-1 antisense promoter in the cMet locus impairs Met signalling through induction of illegitimate transcription (English)

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14 September 2017

B Weber

1

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14 September 2017

S Kimhi

2

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14 September 2017

G Howard

3

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14 September 2017

A Eden

4

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14 September 2017

F Lyko

5

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14 September 2017

language of work or name

English

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publication date

21 June 2010

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14 September 2017

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29

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43

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14 September 2017

5775-5784

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https://scigraph.springernature.com/pub.10.1038/onc.2010.227

14 September 2017

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General transcription factors bind promoters repressed by Polycomb group proteins.

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Comparison of biological effects of non-nucleoside DNA methylation inhibitors versus 5-aza-2'-deoxycytidine.

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Isolation of cancer-specific chimeric transcripts induced by hypomethylation of the LINE-1 antisense promoter

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Activation and transposition of endogenous retroviral elements in hypomethylation induced tumors in mice.

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Targeting DNA methylation

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Igf2 imprinting does not require its own DNA methylation or H19 RNA.

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Toxicity of 5-aza-2'-deoxycytidine to mammalian cells is mediated primarily by covalent trapping of DNA methyltransferase rather than DNA demethylation

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Decitabine improves patient outcomes in myelodysplastic syndromes: results of a phase III randomized study

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Repression of the human dihydrofolate reductase gene by a non-coding interfering transcript

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Transcriptional interference: an unexpected layer of complexity in gene regulation

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Reactivation of epigenetically silenced genes by DNA methyltransferase inhibitors: basic concepts and clinical applications

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Characterization of DNA demethylation effects induced by 5-Aza-2'-deoxycytidine in patients with myelodysplastic syndrome.

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Many human genes are transcribed from the antisense promoter of L1 retrotransposon.

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DNA methylation-mediated down-regulation of DNA methyltransferase-1 (DNMT1) is coincident with, but not essential for, global hypomethylation in human placenta

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DNA methylation inhibitor 5-Aza-2'-deoxycytidine induces reversible genome-wide DNA damage that is distinctly influenced by DNA methyltransferases 1 and 3B.

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DNMT1 and DNMT3b cooperate to silence genes in human cancer cells

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Promoter hypomethylation of the LINE-1 retrotransposable elements activates sense/antisense transcription and marks the progression of chronic myeloid leukemia

21 January 2018

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Covalent bond formation between a DNA-cytosine methyltransferase and DNA containing 5-azacytosine

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Comparing the DNA hypermethylome with gene mutations in human colorectal cancer

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Randomized controlled trial of azacitidine in patients with the myelodysplastic syndrome: a study of the cancer and leukemia group B.

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Antisense promoter of human L1 retrotransposon drives transcription of adjacent cellular genes

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Azacytidine causes complex DNA methylation responses in myeloid leukemia

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Biochemical mechanisms of drug resistance IV. Development of resistance to 5-azacytidine and simultaneous depression of pyrimidine metabolism in leukemic mice

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Transcription of IAP endogenous retroviruses is constrained by cytosine methylation

21 January 2018

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A simple method for estimating global DNA methylation using bisulfite PCR of repetitive DNA elements

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Combined DNA methyltransferase and histone deacetylase inhibition in the treatment of myeloid neoplasms

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DNA methylation changes after 5-aza-2'-deoxycytidine therapy in patients with leukemia

31 March 2020

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31 March 2020

Identifiers

DOI

10.1038/ONC.2010.227

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14 September 2017

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