Mechanisms of white matter damage in multiple sclerosis (Q38182524)

30 August 2017

Mechanisms of white matter damage in multiple sclerosis (English)

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30 August 2017

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Hans Lassmann

1

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30 August 2017

28 January 2014

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30 August 2017

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30 August 2017

62

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30 August 2017

11

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30 August 2017

1816-1830

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Preferential loss of myelin-associated glycoprotein reflects hypoxia-like white matter damage in stroke and inflammatory brain diseases.

30 August 2017

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Extensive cortical remyelination in patients with chronic multiple sclerosis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Antigen presentation in EAE: role of microglia, macrophages and dendritic cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Meningeal T cells associate with diffuse axonal loss in multiple sclerosis spinal cords.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Clonal expansions of CD8(+) T cells dominate the T cell infiltrate in active multiple sclerosis lesions as shown by micromanipulation and single cell polymerase chain reaction

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Tracking iron in multiple sclerosis: a combined imaging and histopathological study at 7 Tesla.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Relapsing and remitting multiple sclerosis: pathology of the newly forming lesion

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Myelination: do astrocytes play a role?

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Elevated protein carbonylation in the brain white matter and gray matter of patients with multiple sclerosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Astrocyte senescence as a component of Alzheimer's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Subpial demyelination in the cerebral cortex of multiple sclerosis patients.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Myelin-laden macrophages are anti-inflammatory, consistent with foam cells in multiple sclerosis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Oligodendrocytes: biology and pathology

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Demyelination versus remyelination in progressive multiple sclerosis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Homogeneity of active demyelinating lesions in established multiple sclerosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Demyelinating myelin oligodendrocyte glycoprotein-specific autoantibody response is focused on one dominant conformational epitope region in rodents

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

The astrocyte in multiple sclerosis revisited

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Reduced astrocytic NF-κB activation by laquinimod protects from cuprizone-induced demyelination

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Monocyte/macrophage differentiation in early multiple sclerosis lesions.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Mitochondrial DNA deletions and neurodegeneration in multiple sclerosis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Premyelinating oligodendrocytes in chronic lesions of multiple sclerosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Re-expression of PSA-NCAM by demyelinated axons: an inhibitor of remyelination in multiple sclerosis?

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Meningeal inflammation plays a role in the pathology of primary progressive multiple sclerosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Axon degeneration mechanisms: commonality amid diversity

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Oligodendrocyte progenitor cell susceptibility to injury in multiple sclerosis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

The relevance of animal models in multiple sclerosis research

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Nrf2-induced antioxidant protection: a promising target to counteract ROS-mediated damage in neurodegenerative disease?

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Mitochondrial dysfunction as a cause of axonal degeneration in multiple sclerosis patients.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Size-selective neuronal changes in the anterior optic pathways suggest a differential susceptibility to injury in multiple sclerosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Axonal damage in acute multiple sclerosis lesions.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

NADPH oxidase expression in active multiple sclerosis lesions in relation to oxidative tissue damage and mitochondrial injury

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Disease-specific molecular events in cortical multiple sclerosis lesions.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Autoreactive CD8+ T cells in multiple sclerosis: a new target for therapy?

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Opposing effects of HLA class I molecules in tuning autoreactive CD8+ T cells in multiple sclerosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

The relation between inflammation and neurodegeneration in multiple sclerosis brains.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Identification of autoantibodies associated with myelin damage in multiple sclerosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Understanding pathogenesis and therapy of multiple sclerosis via animal models: 70 years of merits and culprits in experimental autoimmune encephalomyelitis research.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Placebo-controlled phase 3 study of oral BG-12 for relapsing multiple sclerosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Elevated myeloperoxidase activity in white matter in multiple sclerosis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Laquinimod suppress antigen presentation in relapsing-remitting multiple sclerosis: in-vitro high-throughput gene expression study

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Transcription factor NF-kappaB and inhibitor I kappaBalpha are localized in macrophages in active multiple sclerosis lesions.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Degenerative and spontaneous regenerative processes after spinal cord injury

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Oxidative damage in multiple sclerosis lesions

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

The effect of age on the non-haemin iron in the human brain.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Iron and neurodegeneration in the multiple sclerosis brain

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Occurrence of a multiple sclerosis-like illness in women who have a Leber's hereditary optic neuropathy mitochondrial DNA mutation

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Selective upregulation of scavenger receptors in and around demyelinating areas in multiple sclerosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Aquaporin-4-binding autoantibodies in patients with neuromyelitis optica impair glutamate transport by down-regulating EAAT2.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Dysferlin is a new marker for leaky brain blood vessels in multiple sclerosis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Meningeal inflammation is widespread and linked to cortical pathology in multiple sclerosis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

A pathogenic role for myelin-specific CD8(+) T cells in a model for multiple sclerosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Microglia and multiple sclerosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

A combined post-mortem magnetic resonance imaging and quantitative histological study of multiple sclerosis pathology

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Neuropathology of white matter disease in Leber's hereditary optic neuropathy.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Diagnosis of inflammatory demyelination in biopsy specimens: a practical approach

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Cortical demyelination and diffuse white matter injury in multiple sclerosis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Widespread demyelination in the cerebellar cortex in multiple sclerosis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Progressive multiple sclerosis: pathology and pathogenesis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Review: the architecture of inflammatory demyelinating lesions: implications for studies on pathogenesis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

The immunopathology of multiple sclerosis: an overview

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Endoplasmic reticulum stress in disorders of myelinating cells.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Fumaric acid esters exert neuroprotective effects in neuroinflammation via activation of the Nrf2 antioxidant pathway.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Differential effects of Th1, monocyte/macrophage and Th2 cytokine mixtures on early gene expression for molecules associated with metabolism, signaling and regulation in central nervous system mixed glial cell cultures

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Secretory products of multiple sclerosis B cells are cytotoxic to oligodendroglia in vitro

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Expression of inducible nitric oxide synthase and nitrotyrosine in multiple sclerosis lesions

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Microglial dystrophy in the aged and Alzheimer's disease brain is associated with ferritin immunoreactivity.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Defining the clinical course of multiple sclerosis: Results of an international survey

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Heterogeneity of multiple sclerosis lesions: implications for the pathogenesis of demyelination

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

A quantitative analysis of oligodendrocytes in multiple sclerosis lesions. A study of 113 cases

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Inflammatory cortical demyelination in early multiple sclerosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Meningeal B-cell follicles in secondary progressive multiple sclerosis associate with early onset of disease and severe cortical pathology.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Mitochondrial defects in acute multiple sclerosis lesions

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Mitochondrial changes within axons in multiple sclerosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Gap junction pathology in multiple sclerosis lesions and normal-appearing white matter.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Oligodendrocytes are damaged by neuromyelitis optica immunoglobulin G via astrocyte injury.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Lesion genesis in a subset of patients with multiple sclerosis: a role for innate immunity?

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Neurofascin as a novel target for autoantibody-mediated axonal injury

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Aquaporin-4 astrocytopathy in Baló’s disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

A new focal EAE model of cortical demyelination: multiple sclerosis-like lesions with rapid resolution of inflammation and extensive remyelination.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Presence of six different lesion types suggests diverse mechanisms of tissue injury in neuromyelitis optica

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Fumaric Acid and its esters: an emerging treatment for multiple sclerosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

How factors secreted from astrocytes impact myelin repair

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

How mitochondria produce reactive oxygen species

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Naive CD8 T-cells initiate spontaneous autoimmunity to a sequestered model antigen of the central nervous system.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Oligodendrocytes enforce immune tolerance of the uninfected brain by purging the peripheral repertoire of autoreactive CD8+ T cells.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Astrocytes in multiple sclerosis: a product of their environment

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Debris clearance by microglia: an essential link between degeneration and regeneration.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Cytotoxic T lymphocytes in autoimmune and degenerative CNS diseases.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Cerebral white matter is highly vulnerable to ischemia

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Remyelination can be extensive in multiple sclerosis despite a long disease course

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Remyelination is extensive in a subset of multiple sclerosis patients.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Microglia in neurodegenerative disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Imaging cortical lesions in multiple sclerosis with ultra-high-field magnetic resonance imaging.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Immunopathology of secondary-progressive multiple sclerosis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Multiple sclerosis. Pathology of recurrent lesions.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

C3-dependent mechanism of microglial priming relevant to multiple sclerosis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Ceruloplasmin protects injured spinal cord from iron-mediated oxidative damage.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Mitochondrial sirtuins--a new therapeutic target for repair and protection in multiple sclerosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Cutting edge: Multiple sclerosis-like lesions induced by effector CD8 T cells recognizing a sequestered antigen on oligodendrocytes.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Iron efflux from oligodendrocytes is differentially regulated in gray and white matter.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Detection of ectopic B-cell follicles with germinal centers in the meninges of patients with secondary progressive multiple sclerosis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Inflammation induced by innate immunity in the central nervous system leads to primary astrocyte dysfunction followed by demyelination.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

SIRT1 activation confers neuroprotection in experimental optic neuritis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Microglial nodules in early multiple sclerosis white matter are associated with degenerating axons.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Astrocytes: biology and pathology

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Potassium channel KIR4.1 as an immune target in multiple sclerosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Differential upregulation of heme oxygenase-1 (HSP32) in glial cells after oxidative stress and in demyelinating disorders

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Autoimmunity to myelin oligodendrocyte glycoprotein in rats mimics the spectrum of multiple sclerosis pathology.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Cortical demyelination can be modeled in specific rat models of autoimmune encephalomyelitis and is major histocompatibility complex (MHC) haplotype-related.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Genetic inactivation of mitochondria-targeted redox enzyme p66ShcA preserves neuronal viability and mitochondrial integrity in response to oxidative challenges

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Virtual hypoxia and chronic necrosis of demyelinated axons in multiple sclerosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

CCR1+/CCR5+ mononuclear phagocytes accumulate in the central nervous system of patients with multiple sclerosis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

In vitro and in vivo models of multiple sclerosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Nrf2 and DJ1 are consistently upregulated in inflammatory multiple sclerosis lesions.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

NAD(P)H:quinone oxidoreductase 1 expression in multiple sclerosis lesions

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Severe oxidative damage in multiple sclerosis lesions coincides with enhanced antioxidant enzyme expression.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Preactive multiple sclerosis lesions offer novel clues for neuroprotective therapeutic strategies.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Inhibiting poly(ADP-ribose) polymerase: a potential therapy against oligodendrocyte death.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Molecular characterization of antibody specificities against myelin/oligodendrocyte glycoprotein in autoimmune demyelination

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Pathogenic implications of iron accumulation in multiple sclerosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Astrocytes--friends or foes in multiple sclerosis?

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Enhanced number and activity of mitochondria in multiple sclerosis lesions

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Protective effects and mechanisms of sirtuins in the nervous system

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Molecular changes in white matter adjacent to an active demyelinating lesion in early multiple sclerosis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Injury and differentiation following inhibition of mitochondrial respiratory chain complex IV in rat oligodendrocytes

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Anti–Myelin Oligodendrocyte Glycoprotein Antibodies in Pediatric Patients With Optic Neuritis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

Expression of αB-crystallin in glia cells during lesional development in multiple sclerosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

7 January 2021

The pathology of multiple sclerosis is location-dependent: no significant complement activation is detected in purely cortical lesions

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

7 January 2021

Multiple sclerosis: a quantitative and qualitative study of immunoglobulin-containing cells in the central nervous system

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

7 January 2021

Glycolytic oligodendrocytes maintain myelin and long-term axonal integrity

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

7 January 2021

Cortical lesions in multiple sclerosis

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

7 January 2021

Differentiation block of oligodendroglial progenitor cells as a cause for remyelination failure in chronic multiple sclerosis

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

7 January 2021

Oxidative damage to mitochondrial DNA and activity of mitochondrial enzymes in chronic active lesions of multiple sclerosis

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

7 January 2021

The neurotoxicant, cuprizone, as a model to study demyelination and remyelination in the central nervous system.

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

7 January 2021

[Astrocytopathy in neuromyelitis optica, multiple sclerosis and Baló's disease]

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

7 January 2021

Cortical demyelination in PML and MS: Similarities and differences

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

7 January 2021

A reversible form of axon damage in experimental autoimmune encephalomyelitis and multiple sclerosis

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

7 January 2021

Overexpression of SIRT1 protein in neurons protects against experimental autoimmune encephalomyelitis through activation of multiple SIRT1 targets

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

7 January 2021

Patterns of oligodendroglia pathology in multiple sclerosis

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

7 January 2021

Neuromyelitis optica: a demyelinating disease characterized by acute destruction and regeneration of perivascular astrocytes.

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

7 January 2021

Multiple sclerosis: remyelination of nascent lesions

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

7 January 2021

Myelin antigen-specific CD8+ T cells are encephalitogenic and produce severe disease in C57BL/6 mice

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

7 January 2021

Semaphorin 3A and 3F: key players in myelin repair in multiple sclerosis?

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

7 January 2021

Oligodendrocyte survival, loss and birth in lesions of chronic-stage multiple sclerosis

1 reference

https://api.crossref.org/works/10.1002%2FGLIA.22597

7 January 2021

Identifiers

DOI

10.1002/GLIA.22597

1 reference

30 August 2017

1 reference

PubMed ID

24470325