Entinostat is a histone deacetylase inhibitor selective for class 1 histone deacetylases and activates HIV production from latently infected primary T cells. (Q33838633)

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English	Entinostat is a histone deacetylase inhibitor selective for class 1 histone deacetylases and activates HIV production from latently infected primary T cells.	scientific article

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29 February 2020

Entinostat is a histone deacetylase inhibitor selective for class 1 histone deacetylases and activates HIV production from latently infected primary T cells (English)

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29 February 2020

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29 February 2020

Anthony L. Cunningham

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29 February 2020

Paul Urquhart Cameron

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29 February 2020

Sharon R. Lewin

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29 February 2020

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Ajantha E Solomon

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29 February 2020

Suha Saleh

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29 February 2020

Andrew N Harman

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29 February 2020

Melissa Churchill

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1 November 2013

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29 February 2020

2853-2862

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29 February 2020

Administration of vorinostat disrupts HIV-1 latency in patients on antiretroviral therapy

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Amino acid starvation induces reactivation of silenced transgenes and latent HIV-1 provirus via down-regulation of histone deacetylase 4 (HDAC4)

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Randomized phase II trial of erlotinib with and without entinostat in patients with advanced non-small-cell lung cancer who progressed on prior chemotherapy

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Class I histone deacetylase inhibitor entinostat suppresses regulatory T cells and enhances immunotherapies in renal and prostate cancer models

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HIV reservoirs and strategies for eradication

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Phase I study of the histone deacetylase inhibitor entinostat in combination with 13-cis retinoic acid in patients with solid tumours

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HDAC inhibitors in HIV.

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Expression and reactivation of HIV in a chemokine induced model of HIV latency in primary resting CD4+ T cells

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Inhibitors of histone deacetylases: correlation between isoform specificity and reactivation of HIV type 1 (HIV-1) from latently infected cells

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Disulfiram reactivates latent HIV-1 in a Bcl-2-transduced primary CD4+ T cell model without inducing global T cell activation

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Establishment of HIV-1 latency in resting CD4+ T cells depends on chemokine-induced changes in the actin cytoskeleton.

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Histone deacetylase inhibitor Scriptaid reactivates latent HIV-1 promoter by inducing histone modification in in vitro latency cell lines.

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Combinatorial latency reactivation for HIV-1 subtypes and variants

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The histone deacetylase inhibitor ITF2357 decreases surface CXCR4 and CCR5 expression on CD4(+) T-cells and monocytes and is superior to valproic acid for latent HIV-1 expression in vitro

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Expression of latent human immunodeficiency type 1 is induced by novel and selective histone deacetylase inhibitors

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Synergistic activation of HIV-1 expression by deacetylase inhibitors and prostratin: implications for treatment of latent infection

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HIV reservoir size and persistence are driven by T cell survival and homeostatic proliferation

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A limited group of class I histone deacetylases acts to repress human immunodeficiency virus type 1 expression

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Expression of latent HIV induced by the potent HDAC inhibitor suberoylanilide hydroxamic acid

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Human T cell transcription factor GATA-3 stimulates HIV-1 expression

1 reference

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1 reference

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CCR7 ligands CCL19 and CCL21 increase permissiveness of resting memory CD4+ T cells to HIV-1 infection: a novel model of HIV-1 latency.

1 reference

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29 October 2018

Identifiers

10.1097/QAD.0000000000000067

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29 February 2020

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29 February 2020

1 reference

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29 February 2020

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