Identification of a novel phosphorylation site, Ser-170, as a regulator of bad pro-apoptotic activity (Q28511401)

GOA release 2020-03-11

8

Ruedi Aebersold

1 reference

25 January 2017

7

David R. Goodlett

1 reference

25 January 2017

Shaynoor Dramsi

1

1 reference

25 January 2017

Michael P. Scheid

2

1 reference

25 January 2017

Arpita Maiti

3

1 reference

25 January 2017

Payman Hojabrpour

4

1 reference

25 January 2017

Xianming Chen

5

1 reference

25 January 2017

Kathryn Schubert

6

1 reference

25 January 2017

Vincent Duronio

Journal of Biological Chemistry

9

1 reference

25 January 2017

language of work or name

English

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publication date

22 February 2002

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25 January 2017

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25 January 2017

277

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25 January 2017

6399–6405

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25 January 2017

8

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25 January 2017

BCL-2 family members and the mitochondria in apoptosis

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M109990200

BCL-2 family: regulators of cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M109990200

BH3-Only proteins-essential initiators of apoptotic cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M109990200

Noxa, a BH3-only member of the Bcl-2 family and candidate mediator of p53-induced apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M109990200

21 January 2018

The cell-death machine

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M109990200

Epigenetic inheritance in the mouse

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M109990200

21 January 2018

Bcl-2 family proteins

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M109990200

BH3-only proteins that bind pro-survival Bcl-2 family members fail to induce apoptosis in the absence of Bax and Bak

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M109990200

Bad, a heterodimeric partner for Bcl-XL and Bcl-2, displaces Bax and promotes cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M109990200

Serine phosphorylation of death agonist BAD in response to survival factor results in binding to 14-3-3 not BCL-X(L)

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M109990200

BAD Ser-155 phosphorylation regulates BAD/Bcl-XL interaction and cell survival

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M109990200

Growth factors inactivate the cell death promoter BAD by phosphorylation of its BH3 domain on Ser155.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M109990200

14-3-3 proteins and survival kinases cooperate to inactivate BAD by BH3 domain phosphorylation

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M109990200

Regulation of BAD by cAMP-dependent protein kinase is mediated via phosphorylation of a novel site, Ser155

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M109990200

Phosphorylation of the pro-apoptotic protein BAD on serine 155, a novel site, contributes to cell survival

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M109990200

p90(RSK) blocks bad-mediated cell death via a protein kinase C-dependent pathway

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M109990200

Akt phosphorylation of BAD couples survival signals to the cell-intrinsic death machinery

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M109990200

BH3 domain of BAD is required for heterodimerization with BCL-XL and pro-apoptotic activity

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M109990200

Protein phosphatase 1alpha is a Ras-activated Bad phosphatase that regulates interleukin-2 deprivation-induced apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M109990200

Ca2+-induced apoptosis through calcineurin dephosphorylation of BAD

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M109990200

Protein phosphatase 2A activates the proapoptotic function of BAD in interleukin- 3-dependent lymphoid cells by a mechanism requiring 14-3-3 dissociation

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M109990200

The growth-promoting activity of the Bad protein in chicken embryo fibroblasts requires binding to protein 14-3-3

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M109990200

BAD/BCL-[X(L)] heterodimerization leads to bypass of G0/G1 arrest

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M109990200

Regulation of bad phosphorylation and association with Bcl-x(L) by the MAPK/Erk kinase

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M109990200

Dissociation of cytokine-induced phosphorylation of Bad and activation of PKB/akt: involvement of MEK upstream of Bad phosphorylation

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M109990200

Cell survival promoted by the Ras-MAPK signaling pathway by transcription-dependent and -independent mechanisms

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M109990200

Regulation of BAD phosphorylation at serine 112 by the Ras-mitogen-activated protein kinase pathway

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M109990200

Rsk1 mediates a MEK-MAP kinase cell survival signal

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M109990200

Interleukin-3-induced phosphorylation of BAD through the protein kinase Akt

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M109990200

The kit receptor promotes cell survival via activation of PI 3-kinase and subsequent Akt-mediated phosphorylation of Bad on Ser136

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M109990200

Role of phosphatidylinositol 3-OH-kinase activity in the inhibition of apoptosis in haemopoietic cells: phosphatidylinositol 3-OH-kinase inhibitors reveal a difference in signalling between interleukin-3 and granulocyte-macrophage colony stimulating

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M109990200

Bad can act as a key regulator of T cell apoptosis and T cell development.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M109990200