Phosphorylation of the pro-apoptotic protein BAD on serine 155, a novel site, contributes to cell survival (Q40853818)

24 September 2017

Phosphorylation of the pro-apoptotic protein BAD on serine 155, a novel site, contributes to cell survival (English)

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24 September 2017

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Virdee K

1

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24 September 2017

Parone PA

series ordinal

2

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24 September 2017

Tolkovsky AM

series ordinal

3

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24 September 2017

language of work or name

English

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publication date

1 September 2000

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24 September 2017

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24 September 2017

10

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24 September 2017

1151-1154

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24 September 2017

18

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The kit receptor promotes cell survival via activation of PI 3-kinase and subsequent Akt-mediated phosphorylation of Bad on Ser136

24 September 2017

1 reference

https://api.crossref.org/works/10.1016%2FS0960-9822%2800%2900702-8

7 January 2021

Akt phosphorylation of BAD couples survival signals to the cell-intrinsic death machinery

1 reference

https://api.crossref.org/works/10.1016%2FS0960-9822%2800%2900702-8

7 January 2021

Interleukin-3-induced phosphorylation of BAD through the protein kinase Akt

1 reference

https://api.crossref.org/works/10.1016%2FS0960-9822%2800%2900702-8

7 January 2021

Bad, a heterodimeric partner for Bcl-XL and Bcl-2, displaces Bax and promotes cell death

1 reference

https://api.crossref.org/works/10.1016%2FS0960-9822%2800%2900702-8

7 January 2021

Serine phosphorylation of death agonist BAD in response to survival factor results in binding to 14-3-3 not BCL-X(L)

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https://api.crossref.org/works/10.1016%2FS0960-9822%2800%2900702-8

7 January 2021

Phosphorylation and inactivation of BAD by mitochondria-anchored protein kinase A

1 reference

https://api.crossref.org/works/10.1016%2FS0960-9822%2800%2900702-8

7 January 2021

Cell survival promoted by the Ras-MAPK signaling pathway by transcription-dependent and -independent mechanisms

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https://api.crossref.org/works/10.1016%2FS0960-9822%2800%2900702-8

7 January 2021

Regulation of BAD phosphorylation at serine 112 by the Ras-mitogen-activated protein kinase pathway

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https://api.crossref.org/works/10.1016%2FS0960-9822%2800%2900702-8

7 January 2021

Rsk1 mediates a MEK-MAP kinase cell survival signal

1 reference

https://api.crossref.org/works/10.1016%2FS0960-9822%2800%2900702-8

7 January 2021

p21-activated kinase 1 phosphorylates the death agonist bad and protects cells from apoptosis

1 reference

https://api.crossref.org/works/10.1016%2FS0960-9822%2800%2900702-8

7 January 2021

Dissociation of cytokine-induced phosphorylation of Bad and activation of PKB/akt: involvement of MEK upstream of Bad phosphorylation

1 reference

https://api.crossref.org/works/10.1016%2FS0960-9822%2800%2900702-8

7 January 2021

Regulation of bad phosphorylation and association with Bcl-x(L) by the MAPK/Erk kinase

1 reference

https://api.crossref.org/works/10.1016%2FS0960-9822%2800%2900702-8

7 January 2021

Expression of constitutively active Raf-1 in the mitochondria restores antiapoptotic and leukemogenic potential of a transformation-deficient BCR/ABL mutant

1 reference

https://api.crossref.org/works/10.1016%2FS0960-9822%2800%2900702-8

7 January 2021

Bcl-2 targets the protein kinase Raf-1 to mitochondria

1 reference

https://api.crossref.org/works/10.1016%2FS0960-9822%2800%2900702-8

7 January 2021

Interaction of 14-3-3 with signaling proteins is mediated by the recognition of phosphoserine

1 reference

https://api.crossref.org/works/10.1016%2FS0960-9822%2800%2900702-8

7 January 2021

The structural basis for 14-3-3:phosphopeptide binding specificity

1 reference

https://api.crossref.org/works/10.1016%2FS0960-9822%2800%2900702-8

7 January 2021

BH3 domain of BAD is required for heterodimerization with BCL-XL and pro-apoptotic activity

1 reference

https://api.crossref.org/works/10.1016%2FS0960-9822%2800%2900702-8

7 January 2021

Autophagy is activated by apoptotic signalling in sympathetic neurons: an alternative mechanism of death execution

1 reference

https://api.crossref.org/works/10.1016%2FS0960-9822%2800%2900702-8

7 January 2021

Nerve growth factor-induced PKB/Akt activity is sustained by phosphoinositide 3-kinase dependent and independent signals in sympathetic neurons.

1 reference

https://api.crossref.org/works/10.1016%2FS0960-9822%2800%2900702-8

7 January 2021

10.1016/S0960-9822(00)00702-8

Identifiers

DOI

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24 September 2017

1 reference