Neuronal apoptosis linked to EglN3 prolyl hydroxylase and familial pheochromocytoma genes: developmental culling and cancer (Q28267037)

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English	Neuronal apoptosis linked to EglN3 prolyl hydroxylase and familial pheochromocytoma genes: developmental culling and cancer	scientific article

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Neuronal apoptosis linked to EglN3 prolyl hydroxylase and familial pheochromocytoma genes: developmental culling and cancer (English)

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William G Kaelin

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Susanne Schlisio

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Susanne Schlisio

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11

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ORCID Public Data File 2021

author name string

Sungwoo Lee

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1

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Eijiro Nakamura

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2

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Haifeng Yang

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3

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Wenyi Wei

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4

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Michelle S Linggi

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5

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Mini P Sajan

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6

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Robert V Farese

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7

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Robert S Freeman

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8

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Bruce D Carter

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9

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language of work or name

English

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publication date

August 2005

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published in

Cancer Cell

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volume

8

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page(s)

155-67

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issue

2

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cites work

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Characterization of apoptosis in cultured rat sympathetic neurons after nerve growth factor withdrawal

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SM-20 is a novel mitochondrial protein that causes caspase-dependent cell death in nerve growth factor-dependent neurons

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A c-Jun dominant negative mutant protects sympathetic neurons against programmed cell death

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von Hippel-Lindau tumor suppressor: not only HIF's executioner

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NGF utilizes c-Ret via a novel GFL-independent, inter-RTK signaling mechanism to maintain the trophic status of mature sympathetic neurons.

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Temporal analysis of events associated with programmed cell death (apoptosis) of sympathetic neurons deprived of nerve growth factor

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12 October 2022

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Neuronal apoptosis linked to EglN3 prolyl hydroxylase and familial pheochromocytoma genes: developmental culling and cancer (Q28267037)

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Neuronal apoptosis linked to EglN3 prolyl hydroxylase and familial pheochromocytoma genes: developmental culling and cancer (Q28267037)

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