Gout-associated uric acid crystals activate the NALP3 inflammasome (Q28131797)

2

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Fabio Martinon

1

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author name string

Annick Mayor

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Aubry Tardivel

Molecular identification of a danger signal that alerts the immune system to dying cells

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language of work or name

English

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publication date

9 March 2006

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published in

Nature

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volume

440

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issue

7081

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page(s)

237-41

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cites work

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Blocking IL-1 in systemic inflammation

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Pyrin binds the PSTPIP1/CD2BP1 protein, defining familial Mediterranean fever and PAPA syndrome as disorders in the same pathway

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Targeted disruption of pyrin, the FMF protein, causes heightened sensitivity to endotoxin and a defect in macrophage apoptosis

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New insights into the mechanism of IL-1beta maturation

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The inflammasome: a molecular platform triggering activation of inflammatory caspases and processing of proIL-beta

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Differential activation of the inflammasome by caspase-1 adaptors ASC and Ipaf

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NALPs: a novel protein family involved in inflammation

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Identification of bacterial muramyl dipeptide as activator of the NALP3/cryopyrin inflammasome.

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NLRs join TLRs as innate sensors of pathogens

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ASC is essential for LPS-induced activation of procaspase-1 independently of TLR-associated signal adaptor molecules

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Mechanisms of inflammation in gout

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Prevention of cold-associated acute inflammation in familial cold autoinflammatory syndrome by interleukin-1 receptor antagonist

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Interleukin-1-receptor antagonist in the Muckle-Wells syndrome.

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Update on colchicine and its mechanism of action

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THE EFFECT OF PRETREATMENT WITH COLCHICINE ON THE INFLAMMATORY RESPONSE TO MICROCRYSTALLINE URATE: A MODEL FOR GOUTY INFLAMMATION

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Suppression of metabolic accompaniments of phagocytosis by colchicine

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Innate immunity conferred by Toll-like receptors 2 and 4 and myeloid differentiation factor 88 expression is pivotal to monosodium urate monohydrate crystal-induced inflammation

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Innate immune recognition

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The danger model: a renewed sense of self

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Mutation of a new gene encoding a putative pyrin-like protein causes familial cold autoinflammatory syndrome and Muckle-Wells syndrome

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NALP3 forms an IL-1beta-processing inflammasome with increased activity in Muckle-Wells autoinflammatory disorder

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Identifiers

DOI

10.1038/NATURE04516

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Consolidated OpenCitations Corpus – April 2017

Dimensions Publication ID

ADS bibcode

2006Natur.440..237M

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1039236419

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1 reference

Consolidated OpenCitations Corpus – April 2017

PubMed publication ID

16407889

1 reference

Consolidated OpenCitations Corpus – April 2017