Failure of stress-induced downregulation of Bcl-2 contributes to apoptosis resistance in senescent human diploid fibroblasts (Q79751169)

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25 December 2019

title

Failure of stress-induced downregulation of Bcl-2 contributes to apoptosis resistance in senescent human diploid fibroblasts (English)

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25 December 2019

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2

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25 December 2019

author name string

Ryu SJ

series ordinal

1

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25 December 2019

Park SC

series ordinal

3

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25 December 2019

publication date

9 February 2007

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Cell Death & Differentiation

25 December 2019

published in

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25 December 2019

volume

14

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25 December 2019

issue

5

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25 December 2019

page(s)

1020-1028

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https://scigraph.springernature.com/pub.10.1038/sj.cdd.4402091

25 December 2019

exact match

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A biomarker that identifies senescent human cells in culture and in aging skin in vivo

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Senescent phenotype can be reversed by reduction of caveolin status

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Change of the death pathway in senescent human fibroblasts in response to DNA damage is caused by an inability to stabilize p53.

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Comparison of replicative senescence and stress-induced premature senescence combining differential display and low-density DNA arrays

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Ceramide and reactive oxygen species generated by H2O2 induce caspase-3-independent degradation of Akt/protein kinase B.

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Na/Ca exchanger overexpression induces endoplasmic reticulum stress, caspase-12 release, and apoptosis

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Bax and adenine nucleotide translocator cooperate in the mitochondrial control of apoptosis

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Dexamethasone protected human glioblastoma U87MG cells from temozolomide induced apoptosis by maintaining Bax:Bcl-2 ratio and preventing proteolytic activities

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Lithium inhibits ceramide- and etoposide-induced protein phosphatase 2A methylation, Bcl-2 dephosphorylation, caspase-2 activation, and apoptosis

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Identifiers

DOI

10.1038/SJ.CDD.4402091

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:17290286%20AND%20SRC:MED&resulttype=core&format=json

25 December 2019

1 reference