Mechanisms for generating the autonomous cAMP-dependent protein kinase required for long-term facilitation in Aplysia (Q74458788)

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10 November 2019

title

Mechanisms for generating the autonomous cAMP-dependent protein kinase required for long-term facilitation in Aplysia (English)

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10 November 2019

author name string

D G Chain

1

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10 November 2019

A Casadio

2

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10 November 2019

S Schacher

3

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10 November 2019

A N Hegde

4

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10 November 2019

M Valbrun

5

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10 November 2019

N Yamamoto

6

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10 November 2019

A L Goldberg

7

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10 November 2019

D Bartsch

8

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10 November 2019

E R Kandel

9

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10 November 2019

J H Schwartz

10

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10 November 2019

publication date

1 January 1999

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10 November 2019

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10 November 2019

volume

22

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10 November 2019

issue

1

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10 November 2019

page(s)

147-156

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C/EBP is an immediate-early gene required for the consolidation of long-term facilitation in Aplysia.

10 November 2019

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Spatially resolved dynamics of cAMP and protein kinase A subunits in Aplysia sensory neurons.

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Aplysia CREB2 represses long-term facilitation: relief of repression converts transient facilitation into long-term functional and structural change

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CREB1 encodes a nuclear activator, a repressor, and a cytoplasmic modulator that form a regulatory unit critical for long-term facilitation

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5-HT modulates protein synthesis and the expression of specific proteins during long-term facilitation in Aplysia sensory neurons.

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Protein synthesis during acquisition of long-term facilitation is needed for the persistent loss of regulatory subunits of the Aplysia cAMP-dependent protein kinase

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A regulatory subunit of the cAMP-dependent protein kinase down-regulated in aplysia sensory neurons during long-term sensitization.

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Facilitatory transmitter causes a selective and prolonged increase in adenosine 3':5'-monophosphate in sensory neurons mediating the gill and siphon withdrawal reflex in Aplysia

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Intracellular injection of t he catalytic subunit of cyclic AMP-dependent protein kinase simulates facilitation of transmitter release underlying behavioral sensitization in Aplysia

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7 January 2021

Persistent activation of cAMP-dependent protein kinase by regulated proteolysis suggests a neuron-specific function of the ubiquitin system in Aplysia.

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Serotonin produces long-term changes in the excitability of Aplysia sensory neurons in culture that depend on new protein synthesis

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Injection of the cAMP-responsive element into the nucleus of Aplysia sensory neurons blocks long-term facilitation

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7 January 2021

Ethanol causes translocation of cAMP-dependent protein kinase catalytic subunit to the nucleus

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Monosynaptic connections made by the sensory neurons of the gill- and siphon-withdrawal reflex in Aplysia participate in the storage of long-term memory for sensitization

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7 January 2021

A novel intermediate stage in the transition between short- and long-term facilitation in the sensory to motor neuron synapse of aplysia.

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7 January 2021

A molecular mechanism for long-term sensitization in Aplysia.

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7 January 2021

Distribution of cAMP and cAMP-dependent protein kinases in Aplysia sensory neurons.

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7 January 2021

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Regulatory subunits of cAMP-dependent protein kinases are degraded after conjugation to ubiquitin: a molecular mechanism underlying long-term synaptic plasticity

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7 January 2021

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Repeated pulses of serotonin required for long-term facilitation activate mitogen-activated protein kinase in sensory neurons of Aplysia.

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A critical period for macromolecular synthesis in long-term heterosynaptic facilitation in Aplysia.

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7 January 2021

Serotonin induces temporally and mechanistically distinct phases of persistent PKA activity in Aplysia sensory neurons.

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7 January 2021

Phosphorylation-dependent subcellular translocation of a Ca2+/calmodulin-dependent protein kinase produces an autonomous enzyme in Aplysia neurons

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7 January 2021

Neurite regeneration by Aplysia neurons in dissociated cell culture: modulation by Aplysia hemolymph and the presence of the initial axonal segment

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Identifiers

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10 November 2019

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