Aplysia CREB2 represses long-term facilitation: relief of repression converts transient facilitation into long-term functional and structural change (Q39469791)

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scientific article published on December 1995

Language	Label	Description	Also known as
English	Aplysia CREB2 represses long-term facilitation: relief of repression converts transient facilitation into long-term functional and structural change	scientific article published on December 1995

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scholarly article

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Europe PubMed Central

12 September 2017

Aplysia CREB2 represses long-term facilitation: relief of repression converts transient facilitation into long-term functional and structural change (English)

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Europe PubMed Central

12 September 2017

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object named as

Kandel ER

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Europe PubMed Central

12 September 2017

author name string

Bartsch D

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Europe PubMed Central

12 September 2017

Ghirardi M

2

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Europe PubMed Central

12 September 2017

Skehel PA

3

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Europe PubMed Central

12 September 2017

Karl KA

4

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Europe PubMed Central

12 September 2017

Herder SP

5

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Europe PubMed Central

12 September 2017

Chen M

6

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Europe PubMed Central

12 September 2017

Bailey CH

7

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Europe PubMed Central

12 September 2017

language of work or name

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publication date

1 December 1995

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Europe PubMed Central

12 September 2017

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Europe PubMed Central

12 September 2017

83

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Europe PubMed Central

12 September 2017

979-992

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Europe PubMed Central

12 September 2017

6

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Europe PubMed Central

12 September 2017

The cAMP response element binding protein, CREB, is a potent inhibitor of diverse transcriptional activators

1 reference

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Inhibitor of protein synthesis blocks long-term behavioral sensitization in the isolated gill-withdrawal reflex of Aplysia.

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Long-term habituation of a defensive withdrawal reflex in aplysia

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Roles of PKA and PKC in facilitation of evoked and spontaneous transmitter release at depressed and nondepressed synapses in Aplysia sensory neurons

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Memory function in normal aging.

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Flashbulb memories

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Long-Term Sensitization of a Defensive Withdrawal Reflex in Aplysia

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A novel genetic system to detect protein-protein interactions

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Ultrastructure of the synapses of sensory neurons that mediate the gill-withdrawal reflex inAplysia

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Deficient long-term memory in mice with a targeted mutation of the cAMP-responsive element-binding protein.

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A novel intermediate stage in the transition between short- and long-term facilitation in the sensory to motor neuron synapse of aplysia.

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C/EBP is an immediate-early gene required for the consolidation of long-term facilitation in Aplysia.

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Transcriptional repression by a novel member of the bZIP family of transcription factors

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The price of repression

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Differential regulation of chromogranin B and synapsin I gene promoter activity by cAMP and cAMP-dependent protein kinase

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Habituation and Dishabituation of the Gill-Withdrawal Reflex in Aplysia

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cAMP evokes long-term facilitation in Aplysia sensory neurons that requires new protein synthesis

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Synaptic plasticity in vitro: cell culture of identified Aplysia neurons mediating short-term habituation and sensitization.

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Synaptic Facilitation and Behavioral Sensitization in Aplysia : Possible Role of Serotonin and Cyclic AMP

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Inducibility and negative autoregulation of CREM: an alternative promoter directs the expression of ICER, an early response repressor.

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Morphological basis of long-term habituation and sensitization in Aplysia.

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Neuromodulatory systems and memory storage: role of the amygdala

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Forgotten mnemonists

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Injection of the cAMP-responsive element into the nucleus of Aplysia sensory neurons blocks long-term facilitation

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Time course of structural changes at identified sensory neuron synapses during long-term sensitization in Aplysia.

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Induction of a dominant negative CREB transgene specifically blocks long-term memory in Drosophila

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CREB as a memory modulator: induced expression of a dCREB2 activator isoform enhances long-term memory in Drosophila

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Intracellular injection of cAMP induces a long-term reduction of neuronal K+ currents

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Activation of cAMP-responsive genes by stimuli that produce long-term facilitation in Aplysia sensory neurons.

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The leucine zipper: a hypothetical structure common to a new class of DNA binding proteins

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Rubinstein-Taybi syndrome caused by mutations in the transcriptional co-activator CBP

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More is better: Activators and repressors from the same gene

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A critical period for macromolecular synthesis in long-term heterosynaptic facilitation in Aplysia.

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Structural changes accompanying memory storage

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Inhibitors of protein and RNA synthesis block structural changes that accompany long-term heterosynaptic plasticity in Aplysia

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Characterization of a Y-Box factor from Aplysia californica.

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Spatially resolved dynamics of cAMP and protein kinase A subunits in Aplysia sensory neurons.

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Facilitatory transmitter causes a selective and prolonged increase in adenosine 3':5'-monophosphate in sensory neurons mediating the gill and siphon withdrawal reflex in Aplysia

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10.1016/0092-8674(95)90213-9

1 reference

Europe PubMed Central

12 September 2017

release_l6mdjmraf5esrdz6exnxlvvq3y

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1 reference

Europe PubMed Central

12 September 2017

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