Curcuma longa inhibits TNF-alpha induced expression of adhesion molecules on human umbilical vein endothelial cells (Q73215891)

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2 November 2019

title

Curcuma longa inhibits TNF-alpha induced expression of adhesion molecules on human umbilical vein endothelial cells (English)

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2 November 2019

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bisdemethoxycurcumin or Tetrahydrobisdemethoxy curcumin

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B Gupta

1

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2 November 2019

B Ghosh

series ordinal

2

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2 November 2019

publication date

1 November 1999

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International Journal of Immunopharmacology

2 November 2019

published in

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2 November 2019

volume

21

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2 November 2019

page(s)

745-757

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2 November 2019

issue

11

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Traffic signals for lymphocyte recirculation and leukocyte emigration: the multistep paradigm

2 November 2019

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Pentoxifylline in vivo and in vitro down-regulates the expression of the intercellular adhesion molecule-1 in monocytes

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Primary structure of ICAM-1 demonstrates interaction between members of the immunoglobulin and integrin supergene families

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Molecular structure of the human cytoplasmic beta-actin gene: interspecies homology of sequences in the introns

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A chromatographic study of the curcuminoids in Curcuma longa, L

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Pharmacology of Curcuma longa

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Curcumin is a non-competitive and selective inhibitor of phosphorylase kinase

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Transcriptional regulation of endothelial cell adhesion molecules: NF-kappa B and cytokine-inducible enhancers.

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The IkappaB kinase complex (IKK) contains two kinase subunits, IKKalpha and IKKbeta, necessary for IkappaB phosphorylation and NF-kappaB activation

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The proteasome pathway is required for cytokine-induced endothelial-leukocyte adhesion molecule expression

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Curcumin (Diferuloylmethane) inhibition of tumor necrosis factor (TNF)-mediated adhesion of monocytes to endothelial cells by suppression of cell surface expression of adhesion molecules and of nuclear factor-kappaB activation

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Identifiers

DOI

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2 November 2019

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