Characterization of E2F3a function in HepG2 liver cancer cells. (Q53303550)

13 May 2018

title

Characterization of E2F3a function in HepG2 liver cancer cells. (English)

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13 May 2018

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Wei Li

1

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Guo-Xin Ni

2

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Ping Zhang

3

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Zheng-Xi Zhang

4

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Wei Li

5

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Qiang Wu

6

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13 May 2018

publication date

1 December 2010

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Journal of Cellular Biochemistry

published in

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13 May 2018

volume

111

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issue

5

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page(s)

1244-1251

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Complex transcriptional regulatory mechanisms control expression of the E2F3 locus

cites work

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Repression of the Arf tumor suppressor by E2F3 is required for normal cell cycle kinetics

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E2f3b plays an essential role in myogenic differentiation through isoform-specific gene regulation.

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Division and apoptosis of E2f-deficient retinal progenitors

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Emerging roles of E2Fs in cancer: an exit from cell cycle control

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E2f3a and E2f3b contribute to the control of cell proliferation and mouse development

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E2f1-3 switch from activators in progenitor cells to repressors in differentiating cells

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E2Fs mediate a fundamental cell-cycle deregulation in high-grade serous ovarian carcinomas

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Distinct roles for E2F proteins in cell growth control and apoptosis

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Regulation of transcription by proteins that control the cell cycle

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Amplification and overexpression of E2F3 in human bladder cancer

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Transcription factor E2F3 overexpressed in prostate cancer independently predicts clinical outcome

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New insights into apoptosis signaling by Apo2L/TRAIL.

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The Hallmarks of Cancer

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Identification of E2F-3B, an alternative form of E2F-3 lacking a conserved N-terminal region

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Inactivation of the Rb pathway and overexpression of both isoforms of E2F3 are obligate events in bladder tumours with 6p22 amplification.

21 January 2018

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Life and death decisions by the E2F transcription factors

21 January 2018

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Compensation and specificity of function within the E2F family.

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The retinoblastoma protein binds to a family of E2F transcription factors

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Identification of a novel E2F3 product suggests a mechanism for determining specificity of repression by Rb proteins

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Synergistic function of E2F7 and E2F8 is essential for cell survival and embryonic development.

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Identification of XAF1 as an antagonist of XIAP anti-Caspase activity

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Deregulated expression of E2F family members induces S-phase entry and overcomes p16INK4A-mediated growth suppression

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Cloning and characterization of mouse E2F8, a novel mammalian E2F family member capable of blocking cellular proliferation

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E2F3 is a mediator of DNA damage-induced apoptosis

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E2Fs regulate the expression of genes involved in differentiation, development, proliferation, and apoptosis.

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The retinoblastoma gene family: cousins with overlapping interests

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E2F3 amplification and overexpression is associated with invasive tumor growth and rapid tumor cell proliferation in urinary bladder cancer

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Role of E2F3 expression in modulating cellular proliferation rate in human bladder and prostate cancer cells

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E2F3a stimulates proliferation, p53-independent apoptosis and carcinogenesis in a transgenic mouse model.

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E2F1 has both oncogenic and tumor-suppressive properties in a transgenic model

21 January 2018

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E2F integrates cell cycle progression with DNA repair, replication, and G(2)/M checkpoints

21 January 2018

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Sibling rivalry in the E2F family

21 January 2018

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Mouse development with a single E2F activator.

21 January 2018

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The retinoblastoma protein and cell cycle control

21 January 2018

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Target gene specificity of E2F and pocket protein family members in living cells

21 January 2018

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The identification of E2F1-specific target genes

21 January 2018

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MicroRNA-195 suppresses tumorigenicity and regulates G1/S transition of human hepatocellular carcinoma cells.

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E2F3 contributes both to the inappropriate proliferation and to the apoptosis arising in Rb mutant embryos

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Identifiers

DOI

10.1002/JCB.22851

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13 May 2018

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13 May 2018