H3-/IDH-wild type pediatric glioblastoma is comprised of molecularly and prognostically distinct subtypes with associated oncogenic drivers. (Q50733824)

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scientific article published on 11 April 2017

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English	H3-/IDH-wild type pediatric glioblastoma is comprised of molecularly and prognostically distinct subtypes with associated oncogenic drivers.	scientific article published on 11 April 2017

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scholarly article

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18 March 2018

http://europepmc.org/abstract/MED/28401334

H3-/IDH-wild type pediatric glioblastoma is comprised of molecularly and prognostically distinct subtypes with associated oncogenic drivers. (English)

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Europe PubMed Central

PubMed publication ID

18 March 2018

http://europepmc.org/abstract/MED/28401334

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http://europepmc.org/abstract/MED/28401334

Stefan M. Pfister

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Lukas Chavez

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Andreas von Deimling

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Andreas von Deimling

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David Capper

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Daniel Schrimpf

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Daniel Schrimpf

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Andrey Korshunov

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Andrey Korshunov

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David T. W. Jones

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David T W Jones

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Chris Jones

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http://europepmc.org/abstract/MED/28401334

Volker Hovestadt

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Volker Hovestadt

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http://europepmc.org/abstract/MED/28401334

Christof M Kramm

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Christof M Kramm

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http://europepmc.org/abstract/MED/28401334

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Marina Ryzhova

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http://europepmc.org/abstract/MED/28401334

Zheludkova Olga

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Olga Zheludkova

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http://europepmc.org/abstract/MED/28401334

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Ella Kumirova

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http://europepmc.org/abstract/MED/28401334

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Andrey Golanov

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http://europepmc.org/abstract/MED/28401334

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Antje Habel

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Tanvi Sharma

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http://europepmc.org/abstract/MED/28401334

Anna Burford

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publication date

11 April 2017

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Acta Neuropathologica

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507-516

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http://europepmc.org/abstract/MED/28401334

Investigational therapies up to Phase II which target PDGF receptors: potential anti-cancer therapeutics.

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MGMT methylation analysis of glioblastoma on the Infinium methylation BeadChip identifies two distinct CpG regions associated with gene silencing and outcome, yielding a prediction model for comparisons across datasets, tumor grades, and CIMP-status

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Recurrent MET fusion genes represent a drug target in pediatric glioblastoma

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Genomic analysis of diffuse intrinsic pontine gliomas identifies three molecular subgroups and recurrent activating ACVR1 mutations

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Histone H3F3A and HIST1H3B K27M mutations define two subgroups of diffuse intrinsic pontine gliomas with different prognosis and phenotypes

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K27M mutation in histone H3.3 defines clinically and biologically distinct subgroups of pediatric diffuse intrinsic pontine gliomas

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TERT promoter mutations occur frequently in gliomas and a subset of tumors derived from cells with low rates of self-renewal

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Distribution of TERT promoter mutations in pediatric and adult tumors of the nervous system

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Histologically distinct neuroepithelial tumors with histone 3 G34 mutation are molecularly similar and comprise a single nosologic entity

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Integrated analysis of pediatric glioblastoma reveals a subset of biologically favorable tumors with associated molecular prognostic markers

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21 January 2018

Next-generation (epi)genetic drivers of childhood brain tumours and the outlook for targeted therapies

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Alex's Lemonade Stand Foundation Infant and Childhood Primary Brain and Central Nervous System Tumors Diagnosed in the United States in 2007-2011.

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Molecular Classification of Ependymal Tumors across All CNS Compartments, Histopathological Grades, and Age Groups

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Integrated molecular genetic profiling of pediatric high-grade gliomas reveals key differences with the adult disease

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21 January 2018

PDGFRA amplification is common in pediatric and adult high-grade astrocytomas and identifies a poor prognostic group in IDH1 mutant glioblastoma

1 reference

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21 January 2018

Mesenchymal transition and PDGFRA amplification/mutation are key distinct oncogenic events in pediatric diffuse intrinsic pontine gliomas

1 reference

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Targeting MYCN in neuroblastoma by BET bromodomain inhibition

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ATRX and IDH1-R132H immunohistochemistry with subsequent copy number analysis and IDH sequencing as a basis for an "integrated" diagnostic approach for adult astrocytoma, oligodendroglioma and glioblastoma

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21 January 2018

Driver mutations in histone H3.3 and chromatin remodelling genes in paediatric glioblastoma

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Paediatric and adult glioblastoma: multiform (epi)genomic culprits emerge

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New Brain Tumor Entities Emerge from Molecular Classification of CNS-PNETs

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Hotspot mutations in H3F3A and IDH1 define distinct epigenetic and biological subgroups of glioblastoma

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21 January 2018

Recurrent activating ACVR1 mutations in diffuse intrinsic pontine glioma

1 reference

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Next-generation personalised medicine for high-risk paediatric cancer patients - The INFORM pilot study

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Somatic histone H3 alterations in pediatric diffuse intrinsic pontine gliomas and non-brainstem glioblastomas

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The genomic landscape of diffuse intrinsic pontine glioma and pediatric non-brainstem high-grade glioma

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Identifiers

10.1007/S00401-017-1710-1

1 reference

Europe PubMed Central

PubMed publication ID

18 March 2018

http://europepmc.org/abstract/MED/28401334

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

18 March 2018

http://europepmc.org/abstract/MED/28401334

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