Dementia in Parkinson's disease is associated with enhanced mitochondrial complex I deficiency. (Q48923189)

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7 March 2020

title

Dementia in Parkinson's disease is associated with enhanced mitochondrial complex I deficiency (English)

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7 March 2020

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4

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7 March 2020

Clive Ballard

5

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7 March 2020

Johannes Attems

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7 March 2020

Joseph M. Bateman

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7 March 2020

1

Ariana P Gatt

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7 March 2020

author name string

Olivia F Duncan

2

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language of work or name

English

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publication date

8 February 2016

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7 March 2020

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7 March 2020

volume

31

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issue

3

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7 March 2020

page(s)

352-359

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The epidemiology of dementia associated with Parkinson disease

7 March 2020

cites work

1 reference

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Association of a polymorphism in mitochondrial transcription factor A (TFAM) with Parkinson's disease dementia but not dementia with Lewy bodies.

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Cultivation in glucose-deprived medium stimulates mitochondrial biogenesis and oxidative metabolism in HepG2 hepatoma cells

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Complex I deficiency in Parkinson's disease frontal cortex

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Alzheimer's brains harbor somatic mtDNA control-region mutations that suppress mitochondrial transcription and replication

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Brain, skeletal muscle and platelet homogenate mitochondrial function in Parkinson's disease

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Parkinson's disease brain mitochondrial complex I has oxidatively damaged subunits and is functionally impaired and misassembled.

21 January 2018

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Brain mitochondrial dysfunction and oxidative damage in Parkinson's disease.

21 January 2018

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Human brain cortex: mitochondrial oxidative damage and adaptive response in Parkinson disease and in dementia with Lewy bodies.

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Pathophysiology of somatosensory abnormalities in Parkinson disease

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Parkinson's disease brain mitochondria have impaired respirasome assembly, age-related increases in distribution of oxidative damage to mtDNA and no differences in heteroplasmic mtDNA mutation abundance

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Is the mitochondrial outermembrane protein VDAC1 therapeutic target for Alzheimer's disease?

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Mitochondrial function in human brains is affected by pre- and post mortem factors.

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The transgenic overexpression of alpha-synuclein and not its related pathology associates with complex I inhibition.

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Impairment of mitochondria in adult mouse brain overexpressing predominantly full-length, N-terminally acetylated human α-synuclein

21 January 2018

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Mitochondrial import and accumulation of alpha-synuclein impair complex I in human dopaminergic neuronal cultures and Parkinson disease brain

21 January 2018

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alpha-synuclein promotes mitochondrial deficit and oxidative stress

21 January 2018

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Direct detection of alpha synuclein oligomers in vivo

21 January 2018

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Differences between subacute and chronic MPTP mice models: investigation of dopaminergic neuronal degeneration and alpha-synuclein inclusions.

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The mouse MPTP model: gene expression changes in dopaminergic neurons

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Alpha-synuclein expression in the substantia nigra of MPTP-lesioned non-human primates

21 January 2018

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Protection by the NDI1 gene against neurodegeneration in a rotenone rat model of Parkinson's disease

21 January 2018

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In vivo complementation of complex I by the yeast Ndi1 enzyme. Possible application for treatment of Parkinson disease

21 January 2018

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