The pleiotropic effect of TRAIL on tumor-like synovial fibroblasts from rheumatoid arthritis patients is mediated by caspases (Q47903376)

6 February 2018

title

The pleiotropic effect of TRAIL on tumor-like synovial fibroblasts from rheumatoid arthritis patients is mediated by caspases (English)

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6 February 2018

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R Audo

1

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6 February 2018

B Combe

2

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6 February 2018

B Coulet

3

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6 February 2018

J Morel

4

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6 February 2018

M Hahne

5

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6 February 2018

publication date

1 May 2009

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6 February 2018

Cell Death & Differentiation

published in

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6 February 2018

volume

16

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6 February 2018

issue

9

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6 February 2018

page(s)

1227-1237

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6 February 2018

https://scigraph.springernature.com/pub.10.1038/cdd.2009.38

exact match

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cites work

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T cell life and death signalling via TNF-receptor family members

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TRAIL Promotes the Survival and Proliferation of Primary Human Vascular Endothelial Cells by Activating the Akt and ERK Pathways

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TRAIL promotes the survival, migration and proliferation of vascular smooth muscle cells

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TRAIL regulates normal erythroid maturation through an ERK-dependent pathway

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TRAIL induced survival and proliferation in cancer cells resistant towards TRAIL-induced apoptosis mediated by NF-kappaB.

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Tumor necrosis factor-related apoptosis-inducing ligand-mediated proliferation of tumor cells with receptor-proximal apoptosis defects

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TRAIL promotes metastasis of human pancreatic ductal adenocarcinoma

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Evolving concepts of rheumatoid arthritis

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7 January 2021

Tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) induces rheumatoid arthritis synovial fibroblast proliferation through mitogen-activated protein kinases and phosphatidylinositol 3-kinase/Akt

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TRAIL-R2 (DR5) mediates apoptosis of synovial fibroblasts in rheumatoid arthritis

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Rheumatoid arthritis synovial fluid fibroblasts express TRAIL-R2 (DR5) that is functionally active.

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Apoptosis is not the major death mechanism induced by celecoxib on rheumatoid arthritis synovial fibroblasts

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Trichostatin A sensitises rheumatoid arthritis synovial fibroblasts for TRAIL-induced apoptosis

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Tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) is an inhibitor of autoimmune inflammation and cell cycle progression

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Defective thymocyte apoptosis and accelerated autoimmune diseases in TRAIL-/- mice

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TRAIL: a multifunctional cytokine

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Phosphatidylinositol 3-kinase inhibition broadly sensitizes glioblastoma cells to death receptor- and drug-induced apoptosis

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Akt phosphorylation and stabilization of X-linked inhibitor of apoptosis protein (XIAP).

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Cell survival, cell death and cell cycle pathways are interconnected: implications for cancer therapy

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Bortezomib abolishes tumor necrosis factor-related apoptosis-inducing ligand resistance via a p21-dependent mechanism in human bladder and prostate cancer cells

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Multiple effects of TRAIL in human carcinoma cells: induction of apoptosis, senescence, proliferation, and cytokine production

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Caspases at the crossroads of immune-cell life and death

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Caspase-8 interacts with the p85 subunit of phosphatidylinositol 3-kinase to regulate cell adhesion and motility

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Molecular determinants of kinase pathway activation by Apo2 ligand/tumor necrosis factor-related apoptosis-inducing ligand

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7 January 2021

Many cuts to ruin: a comprehensive update of caspase substrates

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New roles for p21 and p27 cell-cycle inhibitors: a function for each cell compartment?

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The american rheumatism association 1987 revised criteria for the classification of rheumatoid arthritis

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Signal transduction pathways involved in rheumatoid arthritis synovial fibroblast interleukin-18-induced vascular cell adhesion molecule-1 expression

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7 January 2021

Identifiers

DOI

10.1038/CDD.2009.38

1 reference

6 February 2018

PubMed ID

19407827

1 reference

6 February 2018