Nitric oxide sensitizes prostate carcinoma cell lines to TRAIL-mediated apoptosis via inactivation of NF-kappa B and inhibition of Bcl-xl expression. (Q46074399)

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scientific article published in June 2004

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English	Nitric oxide sensitizes prostate carcinoma cell lines to TRAIL-mediated apoptosis via inactivation of NF-kappa B and inhibition of Bcl-xl expression.	scientific article published in June 2004

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26 December 2019

Nitric oxide sensitizes prostate carcinoma cell lines to TRAIL-mediated apoptosis via inactivation of NF-kappa B and inhibition of Bcl-xl expression (English)

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26 December 2019

prostate carcinoma

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4

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26 December 2019

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26 December 2019

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Sara Huerta-Yepez

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26 December 2019

Ali Jazirehi

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Fumiya Hongo

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Genhong Cheng

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Benjamin Bonavida

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1 June 2004

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4993-5003

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26 December 2019

https://scigraph.springernature.com/pub.10.1038/sj.onc.1207655

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Apoptosis control by death and decoy receptors

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NF-kappaB-mediated up-regulation of Bcl-x and Bfl-1/A1 is required for CD40 survival signaling in B lymphocytes

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Redox potential controls the structure and DNA binding activity of the paired domain

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Death receptor ligands, in particular TRAIL, to overcome drug resistance

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Alteration of NF-kappa B p50 DNA binding kinetics by S-nitrosylation

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Nitric oxide inhibits the transcription repressor Yin-Yang 1 binding activity at the silencer region of the Fas promoter: a pivotal role for nitric oxide in the up-regulation of Fas gene expression in human tumor cells

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Nitric oxide disrupts H2O2-dependent activation of nuclear factor kappa B. Role in sensitization of human tumor cells to tumor necrosis factor-alpha -induced cytotoxicity

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Inhibition of NF-kappa B by S-nitrosylation

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Inhibition of NF-kappaB DNA binding by nitric oxide

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1 reference

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7 January 2021

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Redox signaling: nitrosylation and related target interactions of nitric oxide

1 reference

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7 January 2021

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1 reference

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1 reference

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Identifiers

10.1038/SJ.ONC.1207655

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26 December 2019

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26 December 2019

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