Smad3 is required for enamel biomineralization (Q44451036)

3 December 2017

Smad3 is required for enamel biomineralization (English)

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3 December 2017

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Masahiko Yokozeki

1

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3 December 2017

Elaine Afanador

2

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3 December 2017

Masumi Nishi

3

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3 December 2017

Kazuyuki Kaneko

4

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3 December 2017

Hitoyata Shimokawa

5

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3 December 2017

Koutaro Yokote

6

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3 December 2017

Chuxia Deng

7

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3 December 2017

Kunihiro Tsuchida

8

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3 December 2017

Hiromu Sugino

9

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3 December 2017

Keiji Moriyama

10

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3 December 2017

language of work or name

English

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publication date

1 June 2003

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Biochemical and Biophysical Research Communications

3 December 2017

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3 December 2017

305

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3 December 2017

3

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3 December 2017

684-690

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Proteinases in developing dental enamel

3 December 2017

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https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900806-4

Expression, structure, and function of enamel proteinases

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900806-4

TGF-beta signal transduction

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900806-4

Regulation of cell proliferation by Smad proteins

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900806-4

Targeted disruption of SMAD3 results in impaired mucosal immunity and diminished T cell responsiveness to TGF-beta

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900806-4

Abnormal enamel development in a cystic fibrosis transgenic mouse model.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900806-4

Amelogenin-deficient mice display an amelogenesis imperfecta phenotype

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900806-4

A synthetic, chemically modified ribozyme eliminates amelogenin, the major translation product in developing mouse enamel in vivo.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900806-4

Enamel biomineralization defects result from alterations to amelogenin self-assembly

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900806-4

Targeted disruption of Smad3 reveals an essential role in transforming growth factor beta-mediated signal transduction

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900806-4

Mice lacking Smad3 show accelerated wound healing and an impaired local inflammatory response

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900806-4

Smad3 mutant mice develop metastatic colorectal cancer

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900806-4

TGF-beta/Smad3 signals repress chondrocyte hypertrophic differentiation and are required for maintaining articular cartilage

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900806-4

The loss of Smad3 results in a lower rate of bone formation and osteopenia through dysregulation of osteoblast differentiation and apoptosis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900806-4

Identification of a nonsense mutation in the amelogenin gene (AMELX) in a family with X-linked amelogenesis imperfecta (AIH1)

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900806-4

SSCP detection of a nonsense mutation in exon 5 of the amelogenin gene (AMGX) causing X-linked amelogenesis imperfecta (AIH1)

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900806-4

Mutation of the gene encoding the enamel-specific protein, enamelin, causes autosomal-dominant amelogenesis imperfecta

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900806-4

A nonsense mutation in the enamelin gene causes local hypoplastic autosomal dominant amelogenesis imperfecta (AIH2).

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900806-4

TGF-beta1 is essential for the homeostasis of the dentin-pulp complex

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900806-4

Overexpression of Smad2 reveals its concerted action with Smad4 in regulating TGF-beta-mediated epidermal homeostasis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900806-4

Cystic fibrosis transmembrane regulator gene (CFTR) is associated with abnormal enamel formation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900806-4

Reduced Smad3 protein expression and altered transforming growth factor-beta1-mediated signaling in cystic fibrosis epithelial cells

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900806-4

Signaling via hetero-oligomeric complexes of type I and type II serine/threonine kinase receptors

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900806-4

Expression of Smad1 and Smad2 during embryogenesis suggests a role in organ development.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900806-4

Smad2 role in mesoderm formation, left-right patterning and craniofacial development

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900806-4

Smad2 signaling in extraembryonic tissues determines anterior-posterior polarity of the early mouse embryo

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900806-4

Failure of egg cylinder elongation and mesoderm induction in mouse embryos lacking the tumor suppressor smad2.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900806-4

Postgastrulation Smad2-deficient embryos show defects in embryo turning and anterior morphogenesis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900806-4

Antagonistic effects of Smad2 versus Smad7 are sensitive to their expression level during tooth development

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900806-4

Enamelysin (matrix metalloproteinase 20)-deficient mice display an amelogenesis imperfecta phenotype.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900806-4

Amelogenesis imperfecta, dentinogenesis imperfecta and dentin dysplasia revisited: problems in classification

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900806-4

inferred from DOI database lookup

7 January 2021

based on heuristic

Identifiers

DOI

10.1016/S0006-291X(03)00806-4

1 reference

3 December 2017

1 reference