Anisomycin suppresses Jurkat T cell growth by the cell cycle-regulating proteins (Q43867719)

27 November 2017

title

Anisomycin suppresses Jurkat T cell growth by the cell cycle-regulating proteins (English)

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27 November 2017

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Chunyan Yu

1

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27 November 2017

Feiyue Xing

2

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27 November 2017

Zhengle Tang

3

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27 November 2017

Christian Bronner

4

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27 November 2017

Xijian Lu

5

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27 November 2017

Jingfang Di

6

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27 November 2017

Shan Zeng

7

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27 November 2017

Jing Liu

8

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27 November 2017

publication date

1 January 2013

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27 November 2017

published in

Pharmacological Reports

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27 November 2017

volume

65

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27 November 2017

issue

2

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27 November 2017

page(s)

435-444

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Down-regulation of nuclear protein ICBP90 by p53/p21Cip1/WAF1-dependent DNA-damage checkpoint signals contributes to cell cycle arrest at G1/S transition

27 November 2017

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Expression of immediate early gene pip92 during anisomycin-induced cell death is mediated by the JNK- and p38-dependent activation of Elk1.

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The protein synthesis inhibitor anisomycin induces macrophage apoptosis in rabbit atherosclerotic plaques through p38 mitogen-activated protein kinase.

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7 January 2021

ICBP90 mediates the ERK1/2 signaling to regulate the proliferation of Jurkat T cells.

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Tyrosine phosphorylation of the p21 cyclin-dependent kinase inhibitor facilitates the development of proneural glioma.

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Anisomycin superinduces annexin V mRNA expression through the ERK1/2 but not the p38 MAP kinase pathway

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Anisomycin inhibits the behaviors of T cells and the allogeneic skin transplantation in mice

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7 January 2021

p21 is a universal inhibitor of cyclin kinases

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Gatifloxacin induces S and G2-phase cell cycle arrest in pancreatic cancer cells via p21/p27/p53

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7 January 2021

10.1016/S1734-1140(13)71019-3

Identifiers

DOI

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release_5ov3nqyteng2vp5uhyj4ye5rsy

27 November 2017

Fatcat ID

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Fatcat

https://api.fatcat.wiki/v0/release/5ov3nqyteng2vp5uhyj4ye5rsy

24 November 2022

mapped directly with Wikidata item

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