Interleukin-1R signaling is essential for induction of proapoptotic CD8 T cells, viral clearance, and pathology during lymphocytic choriomeningitis virus infection in mice (Q41981686)

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scientific article published on 6 June 2012

Language	Label	Description	Also known as
English	Interleukin-1R signaling is essential for induction of proapoptotic CD8 T cells, viral clearance, and pathology during lymphocytic choriomeningitis virus infection in mice	scientific article published on 6 June 2012

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scholarly article

1 reference

Europe PubMed Central

14 October 2017

Interleukin-1R signaling is essential for induction of proapoptotic CD8 T cells, viral clearance, and pathology during lymphocytic choriomeningitis virus infection in mice (English)

1 reference

Europe PubMed Central

14 October 2017

0 references

lymphocytic choriomeningitis

1 reference

based on heuristic

inferred from title

author name string

Lars T Joeckel

1

1 reference

Europe PubMed Central

14 October 2017

Reinhard Wallich

2

1 reference

Europe PubMed Central

14 October 2017

Sunil S Metkar

3

1 reference

Europe PubMed Central

14 October 2017

Christopher J Froelich

4

1 reference

Europe PubMed Central

14 October 2017

Markus M Simon

5

1 reference

Europe PubMed Central

14 October 2017

Christoph Borner

6

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Europe PubMed Central

14 October 2017

language of work or name

0 references

publication date

6 June 2012

1 reference

Europe PubMed Central

14 October 2017

Journal of Virology

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Europe PubMed Central

14 October 2017

86

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Europe PubMed Central

14 October 2017

8713-8719

1 reference

Europe PubMed Central

14 October 2017

16

1 reference

Europe PubMed Central

14 October 2017

Granzyme B-dependent proteolysis acts as a switch to enhance the proinflammatory activity of IL-1α.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3421753

5 June 2018

Perforin rapidly induces plasma membrane phospholipid flip-flop

1 reference

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Mouse granzyme K has pro-inflammatory potential.

1 reference

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Granzyme B-induced and caspase 3-dependent cleavage of gelsolin by mouse cytotoxic T cells modifies cytoskeleton dynamics.

1 reference

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5 June 2018

Anti-inflammatory Agents: Present and Future

1 reference

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The IL-1 family: regulators of immunity

1 reference

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Differential expression of granzyme B and C in murine cytotoxic lymphocytes

1 reference

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IL-1 acts directly on CD4 T cells to enhance their antigen-driven expansion and differentiation

1 reference

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Immunological and inflammatory functions of the interleukin-1 family

1 reference

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Human and mouse granzyme A induce a proinflammatory cytokine response.

1 reference

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Granzyme B delivery via perforin is restricted by size, but not by heparan sulfate-dependent endocytosis

1 reference

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Granzyme B-induced cell death exerted by ex vivo CTL: discriminating requirements for cell death and some of its signs

1 reference

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Immunobiology and pathogenesis of viral hepatitis

1 reference

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Granzyme B is expressed in mouse mast cells in vivo and in vitro and causes delayed cell death independent of perforin.

1 reference

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1 reference

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A crucial role for interleukin (IL)-1 in the induction of IL-17-producing T cells that mediate autoimmune encephalomyelitis.

1 reference

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Signal 3 tolerant CD8 T cells degranulate in response to antigen but lack granzyme B to mediate cytolysis

1 reference

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Quiescent and activated mouse granulocytes do not express granzyme A and B or perforin: similarities or differences with human polymorphonuclear leukocytes?

1 reference

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Granzyme B and the downstream granzymes C and/or F are important for cytotoxic lymphocyte functions.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3421753

5 June 2018

Apoptotic pathways are selectively activated by granzyme A and/or granzyme B in CTL-mediated target cell lysis

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3421753

5 June 2018

Perforin and Fas act together in the induction of apoptosis, and both are critical in the clearance of lymphocytic choriomeningitis virus infection

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3421753

5 June 2018

Human T regulatory cells can use the perforin pathway to cause autologous target cell death

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3421753

5 June 2018

T cell responses to viral infections: lessons from lymphocytic choriomeningitis virus

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3421753

5 June 2018

Concerted action of the FasL/Fas and perforin/granzyme A and B pathways is mandatory for the development of early viral hepatitis but not for recovery from viral infection

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3421753

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Targeted disruption of the MyD88 gene results in loss of IL-1- and IL-18-mediated function

1 reference

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Chemokines and T lymphocytes: more than an attraction.

1 reference

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To kill or to cure: options in host defense against viral infection.

1 reference

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Molecular mechanisms of lymphocyte-mediated cytotoxicity and their role in immunological protection and pathogenesis in vivo

1 reference

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Cytotoxicity mediated by T cells and natural killer cells is greatly impaired in perforin-deficient mice

1 reference

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Molecular and cellular pathology of hepatitis B.

1 reference

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T cell-mediated hepatitis in mice infected with lymphocytic choriomeningitis virus. Liver cell destruction by H-2 class I-restricted virus-specific cytotoxic T cells as a physiological correlate of the 51Cr-release assay?

1 reference

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Breakdown of blood-brain barrier function in the murine lymphocytic choriomeningitis virus infection mediated by virus-specific CD8+ T cells

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A role for granzyme M in TLR4-driven inflammation and endotoxicosis.

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T-cell intrinsic expression of MyD88 is required for sustained expansion of the virus-specific CD8+ T-cell population in LCMV-infected mice

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IRAK-4 kinase activity is required for IRAK-4-dependent innate and adaptive immune responses.

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Heterogeneity of effector phenotype for acute phase and memory influenza A virus-specific CTL.

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Acquisition of murine NK cell cytotoxicity requires the translation of a pre-existing pool of granzyme B and perforin mRNAs.

1 reference

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The genes for perforin, granzymes A-C and IFN-gamma are differentially expressed in single CD8(+) T cells during primary activation

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based on heuristic

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Mechanisms of liver cell injury in acute and chronic hepatitis B

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https://pubmed.ncbi.nlm.nih.gov/22674984

12 December 2020

based on heuristic

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Identifiers

10.1128/JVI.00682-12

1 reference

Europe PubMed Central

14 October 2017

1 reference

Europe PubMed Central

14 October 2017

1 reference

Europe PubMed Central

14 October 2017

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