Granzyme B induces BID-mediated cytochrome c release and mitochondrial permeability transition (Q40837904)

24 September 2017

Granzyme B induces BID-mediated cytochrome c release and mitochondrial permeability transition (English)

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24 September 2017

Alimonti JB

1

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24 September 2017

Shi L

series ordinal

2

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24 September 2017

Baijal PK

series ordinal

3

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24 September 2017

Greenberg AH

series ordinal

4

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24 September 2017

language of work or name

English

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publication date

12 December 2000

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Journal of Biological Chemistry

24 September 2017

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24 September 2017

276

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24 September 2017

10

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24 September 2017

6974-6982

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A natural killer cell granule protein that induces DNA fragmentation and apoptosis

24 September 2017

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Purification of three cytotoxic lymphocyte granule serine proteases that induce apoptosis through distinct substrate and target cell interactions

21 January 2018

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Granzyme B (GraB) autonomously crosses the cell membrane and perforin initiates apoptosis and GraB nuclear localization.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Cytotoxic T lymphocyte-assisted suicide. Caspase 3 activation is primarily the result of the direct action of granzyme B.

21 January 2018

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https://api.crossref.org/works/10.1074%2FJBC.M008444200

Caspase structure, proteolytic substrates, and function during apoptotic cell death

21 January 2018

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https://api.crossref.org/works/10.1074%2FJBC.M008444200

The role of caspases in development, immunity, and apoptotic signal transduction: lessons from knockout mice.

21 January 2018

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Mitochondria and apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

BCL-2 family members and the mitochondria in apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Mitochondria and cell death. Mechanistic aspects and methodological issues

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

The mitochondrial permeability transition pore and its role in cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

The mitochondrion in cell death control: certainties and incognita

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Apaf-1, a human protein homologous to C. elegans CED-4, participates in cytochrome c-dependent activation of caspase-3

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Caspase-9 and APAF-1 form an active holoenzyme

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Differential requirement for caspase 9 in apoptotic pathways in vivo.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Apaf1 is required for mitochondrial pathways of apoptosis and brain development

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Regulated targeting of BAX to mitochondria.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Cell damage-induced conformational changes of the pro-apoptotic protein Bak in vivo precede the onset of apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Akt phosphorylation of BAD couples survival signals to the cell-intrinsic death machinery

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Serine phosphorylation of death agonist BAD in response to survival factor results in binding to 14-3-3 not BCL-X(L)

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

The proapoptotic activity of the Bcl-2 family member Bim is regulated by interaction with the dynein motor complex

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Proapoptotic Bcl-2 relative Bim required for certain apoptotic responses, leukocyte homeostasis, and to preclude autoimmunity

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Noxa, a BH3-only member of the Bcl-2 family and candidate mediator of p53-induced apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

BH3 domain of BAD is required for heterodimerization with BCL-XL and pro-apoptotic activity

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Granzyme B short-circuits the need for caspase 8 activity during granule-mediated cytotoxic T-lymphocyte killing by directly cleaving Bid.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

The permeability transition pore complex: a target for apoptosis regulation by caspases and bcl-2-related proteins

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Bax and adenine nucleotide translocator cooperate in the mitochondrial control of apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Bax interacts with the permeability transition pore to induce permeability transition and cytochrome c release in isolated mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Granzyme B mimics apical caspases. Description of a unified pathway for trans-activation of executioner caspase-3 and -7.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Mitochondria-dependent and -independent regulation of Granzyme B-induced apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

DFF45/ICAD can be directly processed by granzyme B during the induction of apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

BNIP3 and genetic control of necrosis-like cell death through the mitochondrial permeability transition pore

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Commitment to apoptosis by GD3 ganglioside depends on opening of the mitochondrial permeability transition pore.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Transient and long-lasting openings of the mitochondrial permeability transition pore can be monitored directly in intact cells by changes in mitochondrial calcein fluorescence

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Serial killers: ordering caspase activation events in apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Granule-mediated killing: pathways for granzyme B-initiated apoptosis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Definition and redesign of the extended substrate specificity of granzyme B.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Reduced apoptosis and cytochrome c-mediated caspase activation in mice lacking caspase 9

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

An APAF-1.cytochrome c multimeric complex is a functional apoptosome that activates procaspase-9

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Death and survival signals determine active/inactive conformations of pro-apoptotic BAX, BAD, and BID molecules

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

The mitochondrial death/life regulator in apoptosis and necrosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

21 January 2018

Bcl-2 family proteins

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Caspase cleaved BID targets mitochondria and is required for cytochrome c release, while BCL-XL prevents this release but not tumor necrosis factor-R1/Fas death

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

BID: a novel BH3 domain-only death agonist

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Bid-deficient mice are resistant to Fas-induced hepatocellular apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

The pro-apoptotic proteins, Bid and Bax, cause a limited permeabilization of the mitochondrial outer membrane that is enhanced by cytosol

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Bcl-xL regulates the membrane potential and volume homeostasis of mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Solution structure of BID, an intracellular amplifier of apoptotic signaling

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Bid-induced conformational change of Bax is responsible for mitochondrial cytochrome c release during apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Solution structure of the proapoptotic molecule BID: a structural basis for apoptotic agonists and antagonists

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Bax-induced cytochrome C release from mitochondria is independent of the permeability transition pore but highly dependent on Mg2+ ions

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Ion channel activity of the BH3 only Bcl-2 family member, BID.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Differential modulation of apoptosis sensitivity in CD95 type I and type II cells.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

How do cytotoxic lymphocytes kill their targets?

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M008444200

Cell Death in Development

21 January 2018

1 reference

5 June 2022

https://opencitations.net/index/coci/api/v1/citations/10.1016/S0092-8674(00)80564-4

Identifiers

DOI

10.1074/JBC.M008444200

1 reference

24 September 2017

1 reference