Only amyloidogenic intermediates of transthyretin induce apoptosis (Q40727157)

23 September 2017

Only amyloidogenic intermediates of transthyretin induce apoptosis (English)

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23 September 2017

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Karin Andersson

1

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23 September 2017

Anders Olofsson

2

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23 September 2017

Ellen Holm Nielsen

3

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23 September 2017

Sven-Erik Svehag

4

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23 September 2017

Erik Lundgren

5

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23 September 2017

1 June 2002

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Biochemical and Biophysical Research Communications

23 September 2017

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23 September 2017

294

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23 September 2017

309-314

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23 September 2017

2

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Familial amyloidotic polyneuropathy

23 September 2017

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https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900465-5

Transthyretin amyloidosis

7 January 2021

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https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900465-5

Unifying features of systemic and cerebral amyloidosis

7 January 2021

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https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900465-5

Amyloid deposits and amyloidosis. The beta-fibrilloses (first of two parts).

7 January 2021

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https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900465-5

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900465-5

Hydrogen peroxide mediates amyloid beta protein toxicity

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900465-5

The Alzheimer's A beta peptide induces neurodegeneration and apoptotic cell death in transgenic mice

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900465-5

Scavenger receptor-mediated adhesion of microglia to beta-amyloid fibrils

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900465-5

Oxidative stress is found in amyloid deposits in systemic amyloidosis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900465-5

Histochemical detection of 4-hydroxynonenal protein in Alzheimer amyloid.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900465-5

Neuroscience. New leads on the 'how' of Alzheimer's.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900465-5

Diffusible, nonfibrillar ligands derived from Abeta1-42 are potent central nervous system neurotoxins

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900465-5

Protofibrillar intermediates of amyloid beta-protein induce acute electrophysiological changes and progressive neurotoxicity in cortical neurons.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900465-5

Characterization of two highly amyloidogenic mutants of transthyretin

7 January 2021

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https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900465-5

Rapid and efficient site-specific mutagenesis without phenotypic selection

7 January 2021

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https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900465-5

Vectors for selective expression of cloned DNAs by T7 RNA polymerase

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900465-5

Partial denaturation of transthyretin is sufficient for amyloid fibril formation in vitro

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900465-5

The acid-mediated denaturation pathway of transthyretin yields a conformational intermediate that can self-assemble into amyloid

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900465-5

An ultrastructural study of amyloid intermediates in A beta1-42 fibrillogenesis.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900465-5

Examination of the Structure of the Transthyretin Amyloid Fibril by Image Reconstruction from Electron Micrographs

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900465-5

Amyloid beta peptide induces necrosis rather than apoptosis.

7 January 2021

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https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900465-5

Neurodegeneration induced by beta-amyloid peptides in vitro: the role of peptide assembly state

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900465-5

Assembly of A beta amyloid protofibrils: an in vitro model for a possible early event in Alzheimer's disease.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900465-5

The characterization of soluble amyloid prepared in water

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900465-5

Amyloid peptides are toxic via a common oxidative mechanism

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900465-5

Inhibition of amyloid beta protein aggregation and neurotoxicity by rifampicin. Its possible function as a hydroxyl radical scavenger.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900465-5

Vitamin E protects nerve cells from amyloid beta protein toxicity

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900465-5

The role of iron in beta amyloid toxicity

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900465-5

The Aβ Peptide of Alzheimer's Disease Directly Produces Hydrogen Peroxide through Metal Ion Reduction†

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900465-5

Cu(II) potentiation of alzheimer abeta neurotoxicity. Correlation with cell-free hydrogen peroxide production and metal reduction.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900465-5

Deposition of transthyretin in early stages of familial amyloidotic polyneuropathy: evidence for toxicity of nonfibrillar aggregates

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900465-5

RAGE and amyloid-beta peptide neurotoxicity in Alzheimer's disease

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900465-5

Interaction of the receptor for advanced glycation end products (RAGE) with transthyretin triggers nuclear transcription factor kB (NF-kB) activation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900465-5

7 January 2021

Advances in amyloidosis

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900465-5

inferred from DOI database lookup

7 January 2021

based on heuristic

Identifiers

DOI

10.1016/S0006-291X(02)00465-5

1 reference

23 September 2017

1 reference