Mcl-1 is required for Akata6 B-lymphoma cell survival and is converted to a cell death molecule by efficient caspase-mediated cleavage. (Q40560316)

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28 December 2019

title

Mcl-1 is required for Akata6 B-lymphoma cell survival and is converted to a cell death molecule by efficient caspase-mediated cleavage (English)

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28 December 2019

10

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28 December 2019

Graham Packham

11

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28 December 2019

author name string

Jorg Michels

1

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28 December 2019

Jason W O'Neill

2

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28 December 2019

Claire L Dallman

3

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28 December 2019

Amalia Mouzakiti

4

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28 December 2019

Fay Habens

5

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28 December 2019

Matthew Brimmell

6

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28 December 2019

Kam Y J Zhang

7

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28 December 2019

Ruth W Craig

8

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28 December 2019

Eric G Marcusson

9

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28 December 2019

language of work or name

English

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publication date

1 June 2004

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28 December 2019

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28 December 2019

volume

23

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28 December 2019

page(s)

4818-4827

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28 December 2019

issue

28

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https://scigraph.springernature.com/pub.10.1038/sj.onc.1207648

28 December 2019

exact match

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Activators of the Epstein-Barr virus lytic program concomitantly induce apoptosis, but lytic gene expression protects from cell death

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MCL1, a gene expressed in programmed myeloid cell differentiation, has sequence similarity to BCL2

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The intracellular distribution and pattern of expression of Mcl-1 overlap with, but are not identical to, those of Bcl-2

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Bcl-2 family of proteins in indolent B-cell non-Hodgkin's lymphoma: Study of 116 cases

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In vivo localisation and stability of human Mcl-1 using green fluorescent protein (GFP) fusion proteins

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Reversal of EBV immortalization precedes apoptosis in IL-6-induced human B cell terminal differentiation

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The mechanism of tumor cell clearance by rituximab in vivo in patients with B-cell chronic lymphocytic leukemia: evidence of caspase activation and apoptosis induction

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Molecular cloning and DNA sequence analysis of cDNA encoding chicken homologue of the Bcl-2 oncoprotein

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7 January 2021

Cloning and characterization of zfBLP1, a Bcl-XL homologue from the zebrafish, Danio rerio.

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7 January 2021

Conversion of Bcl-2 to a Bax-like death effector by caspases

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7 January 2021

Caspases: the executioners of apoptosis

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MCL1 provides a window on the role of the BCL2 family in cell proliferation, differentiation and tumorigenesis

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7 January 2021

DNA damage response and MCL-1 destruction initiate apoptosis in adenovirus-infected cells

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7 January 2021

Antisense strategy shows that Mcl-1 rather than Bcl-2 or Bcl-x(L) is an essential survival protein of human myeloma cells.

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7 January 2021

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The protective effect of phorbol esters on Fas-mediated apoptosis in T cells. Transcriptional and postranscriptional regulation.

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Expression of Mcl-1 in mantle cell lymphoma is associated with high-grade morphology, a high proliferative state, and p53 overexpression

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7 January 2021

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7 January 2021

Elimination of Mcl-1 is required for the initiation of apoptosis following ultraviolet irradiation

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7 January 2021

Development and maintenance of B and T lymphocytes requires antiapoptotic MCL-1.

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Bisindolylmaleimide IX is a potent inducer of apoptosis in chronic lymphocytic leukaemic cells and activates cleavage of Mcl-1.

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High-grade malignant non-Hodgkin's lymphomas differ from low-grade lymphomas in the extent of apoptosis and their expression of bcl-2, mcl-1, bax and p53

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7 January 2021

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An Epstein-Barr virus-producer line Akata: Establishment of the cell line and analysis of viral DNA

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Identifiers

DOI

10.1038/SJ.ONC.1207648

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:15122313%20AND%20SRC:MED&resulttype=core&format=json

Dimensions Publication ID

28 December 2019

1009472794

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Fatcat ID

release_m7ssfa2drjgepfqjxq7sbkystu

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Fatcat

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24 November 2022

mapped directly with Wikidata item

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