Suppression of NF-kappaB and NF-kappaB-regulated gene expression by sulforaphane and PEITC through IkappaBalpha, IKK pathway in human prostate cancer PC-3 cells. (Q40430279)

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English	Suppression of NF-kappaB and NF-kappaB-regulated gene expression by sulforaphane and PEITC through IkappaBalpha, IKK pathway in human prostate cancer PC-3 cells.	scientific article

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11 January 2020

Suppression of NF-kappaB and NF-kappaB-regulated gene expression by sulforaphane and PEITC through IkappaBalpha, IKK pathway in human prostate cancer PC-3 cells (English)

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11 January 2020

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prostate cancer

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3

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11 January 2020

Ah-Ng Tony Kong

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Ah-Ng Tony Kong

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11 January 2020

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Changjiang Xu

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11 January 2020

Guoxiang Shen

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11 January 2020

Céline Gélinas

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1 June 2005

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11 January 2020

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11 January 2020

4486-4495

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11 January 2020

https://scigraph.springernature.com/pub.10.1038/sj.onc.1208656

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NF-kappaB activation in cancer: a challenge for ubiquitination- and proteasome-based therapeutic approach

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Potent induction of carcinogen defence enzymes with sulforaphane, a putative prostate cancer chemopreventive agent.

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Inhibitors of protein kinase C (PKC) prevent activated transcription: role of events downstream of NF-kappaB DNA binding

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Tyrosine phosphorylation of I kappa B alpha activates NF kappa B through a redox-regulated and c-Src-dependent mechanism following hypoxia/reoxygenation.

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The IKK/NF-kappaB activation pathway-a target for prevention and treatment of cancer

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Nuclear factor kappa B is a molecular target for sulforaphane-mediated anti-inflammatory mechanisms.

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In vivo pharmacokinetics and regulation of gene expression profiles by isothiocyanate sulforaphane in the rat.

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Blockade of NF-kappaB activity in human prostate cancer cells is associated with suppression of angiogenesis, invasion, and metastasis

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Modulatory properties of various natural chemopreventive agents on the activation of NF-kappaB signaling pathway

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Signal transduction events elicited by cancer prevention compounds

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Inhibition of NF-kappaB essentially contributes to arsenic-induced apoptosis.

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Inhibition of 12-O-tetradecanoylphorbol-13-acetate-induced NF-kappaB activation by tea polyphenols, (-)-epigallocatechin gallate and theaflavins

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Activation of the heterodimeric IkappaB kinase alpha (IKKalpha)-IKKbeta complex is directional: IKKalpha regulates IKKbeta under both basal and stimulated conditions

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Aberrant rel/nfkb genes and activity in human cancer

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Activation by IKKalpha of a second, evolutionary conserved, NF-kappa B signaling pathway

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Suppression of constitutive and tumor necrosis factor alpha-induced nuclear factor (NF)-kappaB activation and induction of apoptosis by apigenin in human prostate carcinoma PC-3 cells: correlation with down-regulation of NF-kappaB-responsive genes

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Sulforaphane induces caspase-mediated apoptosis in cultured PC-3 human prostate cancer cells and retards growth of PC-3 xenografts in vivo

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Cell cycle arrest, apoptosis induction and inhibition of nuclear factor kappa B activation in anti-proliferative activity of benzyl isothiocyanate against human pancreatic cancer cells

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Vegetables, fruit, and cancer prevention: a review

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Mechanisms of constitutive NF-kappaB activation in human prostate cancer cells

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NF-kappaB activation in human prostate cancer: important mediator or epiphenomenon?

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Flavopiridol inhibits NF-kappaB activation induced by various carcinogens and inflammatory agents through inhibition of IkappaBalpha kinase and p65 phosphorylation: abrogation of cyclin D1, cyclooxygenase-2, and matrix metalloprotease-9.

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Protein farnesyltransferase inhibitor (SCH 66336) abolishes NF-kappaB activation induced by various carcinogens and inflammatory stimuli leading to suppression of NF-kappaB-regulated gene expression and up-regulation of apoptosis

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Hydrogen peroxide activates NF-kappa B through tyrosine phosphorylation of I kappa B alpha and serine phosphorylation of p65: evidence for the involvement of I kappa B alpha kinase and Syk protein-tyrosine kinase

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Identification of a p65 peptide that selectively inhibits NF-kappa B activation induced by various inflammatory stimuli and its role in down-regulation of NF-kappaB-mediated gene expression and up-regulation of apoptosis

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Chemoprevention of colonic aberrant crypt foci in Fischer rats by sulforaphane and phenethyl isothiocyanate

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