Human keratinocytes acquire cellular cytotoxicity under UV-B irradiation. Implication of granzyme B and perforin (Q40307451)

19 September 2017

Human keratinocytes acquire cellular cytotoxicity under UV-B irradiation. Implication of granzyme B and perforin (English)

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19 September 2017

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Matthias Titeux

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Christine Jean

2

object named as

Christine Jean

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19 September 2017

Hélène Hernandez-Pigeon

1

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19 September 2017

Alexandra Charruyer

3

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19 September 2017

Marie-José Haure

4

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19 September 2017

Laure Tonasso

6

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19 September 2017

Anne Quillet-Mary

7

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19 September 2017

Caroline Baudouin

8

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19 September 2017

Marie Charveron

9

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19 September 2017

Guy Laurent

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19 September 2017

language of work or name

English

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publication date

8 March 2006

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Journal of Biological Chemistry

19 September 2017

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19 September 2017

281

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19 September 2017

19

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19 September 2017

13525-13532

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Skin aging and photoaging: an outlook

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Functional significance of the perforin/granzyme cell death pathway

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Cytotoxic lymphocytes require granzyme B for the rapid induction of DNA fragmentation and apoptosis in allogeneic target cells.

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Purification of three cytotoxic lymphocyte granule serine proteases that induce apoptosis through distinct substrate and target cell interactions

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Cytotoxicity mediated by T cells and natural killer cells is greatly impaired in perforin-deficient mice

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Perforin-independent expression of granzyme B and proteinase inhibitor 9 in human testis and placenta suggests a role for granzyme B-mediated proteolysis in reproduction

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Chemoprevention of UV light-induced skin tumorigenesis by inhibition of the epidermal growth factor receptor.

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Activation of extracellular signal-regulated kinase by ultraviolet is mediated through Src-dependent epidermal growth factor receptor phosphorylation. Its implication in an anti-apoptotic function

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Ultraviolet light and osmotic stress: activation of the JNK cascade through multiple growth factor and cytokine receptors

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Involvement of EGF receptor activation in the induction of cyclooxygenase-2 in HaCaT keratinocytes after UVB.

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Measurement of cytotoxic activity in experimental cancer.

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Identifiers

DOI

10.1074/JBC.M512694200

1 reference

19 September 2017

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