Nonsecreted insulin-like growth factor binding protein-3 (IGFBP-3) can induce apoptosis in human prostate cancer cells by IGF-independent mechanisms without being concentrated in the nucleus. (Q40263710)

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scientific article published on 22 June 2006

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English	Nonsecreted insulin-like growth factor binding protein-3 (IGFBP-3) can induce apoptosis in human prostate cancer cells by IGF-independent mechanisms without being concentrated in the nucleus.	scientific article published on 22 June 2006

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scholarly article

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25 December 2019

Nonsecreted insulin-like growth factor binding protein-3 (IGFBP-3) can induce apoptosis in human prostate cancer cells by IGF-independent mechanisms without being concentrated in the nucleus (English)

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based on heuristic

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prostate cancer

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based on heuristic

inferred from title

Malgorzata Wiench

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Nisan Bhattacharyya

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Klaus Pechhold

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Hanief Shahjee

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Giovanna Zappala

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Cem Elbi

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Bruce Raaka

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Jiang Hong

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Matthew M Rechler

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22 June 2006

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Journal of Biological Chemistry

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25 December 2019

Cellular actions of the insulin-like growth factor binding proteins

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Insulin-like growth factor (IGF)-binding protein-3 induces apoptosis and mediates the effects of transforming growth factor-beta1 on programmed cell death through a p53- and IGF-independent mechanism

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Plasma insulin-like growth factor-I and prostate cancer risk: a prospective study

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Insulin-like growth factor-I (IGF-I) and IGF binding protein-3 as predictors of advanced-stage prostate cancer.

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Transfection of human insulin-like growth factor-binding protein 3 gene inhibits cell growth and tumorigenicity: a cell culture model for lung cancer

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Combination therapy of insulin-like growth factor binding protein-3 and retinoid X receptor ligands synergize on prostate cancer cell apoptosis in vitro and in vivo

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Loss of HOXC6 expression induces apoptosis in prostate cancer cells

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EWS/FLI-1 silencing and gene profiling of Ewing cells reveal downstream oncogenic pathways and a crucial role for repression of insulin-like growth factor binding protein 3.

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Human papillomavirus type 16 E7 oncoprotein binds and inactivates growth-inhibitory insulin-like growth factor binding protein 3

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Transforming growth factor-beta-induced cell growth inhibition in human breast cancer cells is mediated through insulin-like growth factor-binding protein-3 action.

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Induction of the growth inhibitor IGF-binding protein 3 by p53

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Insulin-like growth factor binding protein-3 mediates cytokine-induced mesangial cell apoptosis

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Characterization of structural p53 mutants which show selective defects in apoptosis but not cell cycle arrest

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Antiproliferative effects of insulin-like growth factor-binding protein-3 in mesenchymal chondrogenic cell line RCJ3.1C5.18. relationship to differentiation stage

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The human insulin-like growth factor (IGF) binding protein-3 inhibits the growth of fibroblasts with a targeted disruption of the IGF-I receptor gene

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Insulin-like growth factor (IGF)-binding protein-3 mutants that do not bind IGF-I or IGF-II stimulate apoptosis in human prostate cancer cells

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Intranuclear localization of insulin-like growth factor binding protein-3 (IGFBP-3) during cell division in human keratinocytes

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Insulin-like growth factor-binding protein (IGFBP)-3 and IGFBP-5 share a common nuclear transport pathway in T47D human breast carcinoma cells.

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Direct functional interactions between insulin-like growth factor-binding protein-3 and retinoid X receptor-alpha regulate transcriptional signaling and apoptosis

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Signaling through the Smad pathway by insulin-like growth factor-binding protein-3 in breast cancer cells. Relationship to transforming growth factor-beta 1 signaling.

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10.1074/JBC.M509463200

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25 December 2019

1 reference

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25 December 2019

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