The histone deacetylase inhibitor trichostatin A induces GADD45 gamma expression via Oct and NF-Y binding sites (Q40088653)

17 September 2017

The histone deacetylase inhibitor trichostatin A induces GADD45 gamma expression via Oct and NF-Y binding sites (English)

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17 September 2017

1

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17 September 2017

Herrero A

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17 September 2017

Calvo V

series ordinal

3

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17 September 2017

language of work or name

English

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publication date

27 August 2007

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17 September 2017

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17 September 2017

27

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17 September 2017

9

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17 September 2017

1263-1272

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https://scigraph.springernature.com/pub.10.1038/sj.onc.1210735

17 September 2017

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Induction and superinduction of growth arrest and DNA damage gene 45 (GADD45) alpha and beta messenger RNAs by histone deacetylase inhibitors trichostatin A (TSA) and butyrate in SW620 human colon carcinoma cells

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Gadd45gamma expression is reduced in anaplastic thyroid cancer and its reexpression results in apoptosis

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Genes modulated by histone acetylation as new effectors of butyrate activity

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Induction of gadd45beta by NF-kappaB downregulates pro-apoptotic JNK signalling.

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Cytokine response gene 6 induces p21 and regulates both cell growth and arrest

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Histone H2B gene transcription during Xenopus early development requires functional cooperation between proteins bound to the CCAAT and octamer motifs

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p53-independent induction of Gadd45 by histone deacetylase inhibitor: coordinate regulation by transcription factors Oct-1 and NF-Y

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Trichostatin A induces transforming growth factor beta type II receptor promoter activity and acetylation of Sp1 by recruitment of PCAF/p300 to a Sp1.NF-Y complex

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Sp1 and NF-Y synergistically mediate the effect of vitamin D(3) in the p27(Kip1) gene promoter that lacks vitamin D response elements

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Gadd45gamma is androgen-responsive and growth-inhibitory in prostate cancer cells

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Transcription factors Oct-1 and NF-YA regulate the p53-independent induction of the GADD45 following DNA damage

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Histone-deacetylase inhibitors: novel drugs for the treatment of cancer

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A novel oncostatin M-inducible gene OIG37 forms a gene family with MyD118 and GADD45 and negatively regulates cell growth

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Histone deacetylase activity represses gamma interferon-inducible HLA-DR gene expression following the establishment of a DNase I-hypersensitive chromatin conformation

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Histone deacetylase inhibitors activate INK4d gene through Sp1 site in its promoter

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CR6: A third member in the MyD118 and Gadd45 gene family which functions in negative growth control

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Loss of expression of GADD45 gamma, a growth inhibitory gene, in human pituitary adenomas: implications for tumorigenesis

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Myc represses the growth arrest gene gadd45

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Genomic instability in Gadd45a-deficient mice

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GADD45-induced cell cycle G2-M arrest associates with altered subcellular distribution of cyclin B1 and is independent of p38 kinase activity

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Myeloid differentiation (MyD) primary response genes in hematopoiesis

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Gadd45beta is important for perpetuating cognate and inflammatory signals in T cells

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GADD45gamma mediates the activation of the p38 and JNK MAP kinase pathways and cytokine production in effector TH1 cells

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Identifiers

DOI

10.1038/SJ.ONC.1210735

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Dimensions Publication ID

17 September 2017

1048575434

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Fatcat ID

release_xi3am5aqvvfvhfiaffsdog7fq4

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Fatcat

https://api.fatcat.wiki/v0/release/xi3am5aqvvfvhfiaffsdog7fq4

24 November 2022

mapped directly with Wikidata item

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