Erg and AP-1 as determinants of glucocorticoid response in acute lymphoblastic leukemia (Q39301443)

10 September 2017

Erg and AP-1 as determinants of glucocorticoid response in acute lymphoblastic leukemia (English)

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10 September 2017

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10 September 2017

V Saha

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author name string

D W-C Chen

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10 September 2017

J-Z Liu

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10 September 2017

M Krstic-Demonacos

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10 September 2017

language of work or name

English

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publication date

6 August 2012

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10 September 2017

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32

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10 September 2017

3039-3048

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10 September 2017

25

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https://scigraph.springernature.com/pub.10.1038/onc.2012.321

10 September 2017

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Gene expression profiling of leukemic cells and primary thymocytes predicts a signature for apoptotic sensitivity to glucocorticoids

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Coordinated control of the gene expression machinery

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Inhibition of AP-1 by the glucocorticoid-inducible protein GILZ.

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AP-1 as a regulator of cell life and death

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Activation of the JNK pathway promotes phosphorylation and degradation of BimEL--a novel mechanism of chemoresistance in T-cell acute lymphoblastic leukemia

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Coordinate regulation of glucocorticoid receptor and c-jun gene expression is cell type-specific and exhibits differential hormonal sensitivity for down- and up-regulation

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Integrins and cytokines activate nuclear transcription factor-kappaB in human neutrophils

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The p38 and JNK pathways cooperate to trans-activate vitamin D receptor via c-Jun/AP-1 and sensitize human breast cancer cells to vitamin D(3)-induced growth inhibition

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JNK-dependent phosphorylation of c-Jun on serine 63 mediates nitric oxide-induced apoptosis of neuroblastoma cells

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High expression of the ETS transcription factor ERG predicts adverse outcome in acute T-lymphoblastic leukemia in adults.

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Low ERG and BAALC expression identifies a new subgroup of adult acute T-lymphoblastic leukemia with a highly favorable outcome.

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The Ets transcription factors interact with each other and with the c-Fos/c-Jun complex via distinct protein domains in a DNA-dependent and -independent manner

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TMPRSS2-ERG fusion, a common genomic alteration in prostate cancer activates C-MYC and abrogates prostate epithelial differentiation.

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A comparison of normalization methods for high density oligonucleotide array data based on variance and bias

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Clustering short time series gene expression data

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PCAF is an HIF-1alpha cofactor that regulates p53 transcriptional activity in hypoxia.

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The role of glucocorticoid receptor phosphorylation in Mcl-1 and NOXA gene expression.

21 January 2018

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ChIP-seq: using high-throughput sequencing to discover protein-DNA interactions

21 January 2018

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Pro-apoptotic Bim Induction in Response to Nerve Growth Factor Deprivation Requires Simultaneous Activation of Three Different Death Signaling Pathways

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Dexamethasone inhibits lung epithelial cell apoptosis induced by IFN-gamma and Fas.

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Steroid receptor regulated transcription of specific genes and gene networks

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Classification, subtype discovery, and prediction of outcome in pediatric acute lymphoblastic leukemia by gene expression profiling

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ETS transcription factors and their emerging roles in human cancer

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c-Myc does not prevent glucocorticoid-induced apoptosis of human leukemic lymphoblasts

31 March 2020

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Linear models and empirical bayes methods for assessing differential expression in microarray experiments

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TM4: a free, open-source system for microarray data management and analysis

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31 March 2020

Identifiers

DOI

10.1038/ONC.2012.321

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10 September 2017

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