AP-1 as a regulator of cell life and death (Q29616558)

Europe PubMed Central

title

AP-1 as a regulator of cell life and death (English)

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=11988758

30 April 2017

author name string

Shaulian E

series ordinal

1

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30 April 2017

Karin M

series ordinal

2

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=11988758

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=11988758

30 April 2017

language of work or name

1 reference

30 April 2017

publication date

May 2002

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=11988758

30 April 2017

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=11988758

30 April 2017

volume

4

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=11988758

30 April 2017

page(s)

E131-6

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=11988758

30 April 2017

issue

5

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=11988758

https://scigraph.springernature.com/pub.10.1038/ncb0502-e131

30 April 2017

exact match

0 references

cites work

jun-B inhibits and c-fos stimulates the transforming and trans-activating activities of c-jun

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Jun-B differs in its biological properties from, and is a negative regulator of, c-Jun.

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

The regulation of AP-1 activity by mitogen-activated protein kinases

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

AP-1 function and regulation

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Control of cell cycle progression by c-Jun is p53 dependent

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

JunB suppresses cell proliferation by transcriptional activation of p16(INK4a) expression

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

The mammalian UV response: c-Jun induction is required for exit from p53-imposed growth arrest

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

c-Jun interacts with the corepressor TG-interacting factor (TGIF) to suppress Smad2 transcriptional activity

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Mammalian MAP kinase signalling cascades

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Serum-regulated transcription by serum response factor (SRF): a novel role for the DNA binding domain.

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

BMK1/ERK5 regulates serum-induced early gene expression through transcription factor MEF2C

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Induction of c-fos expression through JNK-mediated TCF/Elk-1 phosphorylation.

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Transcription factor ATF2 regulation by the JNK signal transduction pathway

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Rapid and preferential activation of the c-jun gene during the mammalian UV response

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Requirement for fos gene expression in the transcriptional activation of collagenase by other oncogenes and phorbol esters

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Identification of an oncoprotein- and UV-responsive protein kinase that binds and potentiates the c-Jun activation domain

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

JNK1: a protein kinase stimulated by UV light and Ha-Ras that binds and phosphorylates the c-Jun activation domain

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Activation of the transcription factor MEF2C by the MAP kinase p38 in inflammation

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

AP-1 in cell proliferation and survival

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

The jun and fos protein families are both required for cell cycle progression in fibroblasts

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Fos family members induce cell cycle entry by activating cyclin D1.

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Amino-terminal phosphorylation of c-Jun regulates stress-induced apoptosis and cellular proliferation

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

A null mutation at the c-jun locus causes embryonic lethality and retarded cell growth in culture

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Functions of c-Jun in liver and heart development

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

c-Jun and JunB antagonistically control cytokine-regulated mesenchymal-epidermal interaction in skin

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

c-Jun regulates cell cycle progression and apoptosis by distinct mechanisms

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

p21 is a universal inhibitor of cyclin kinases

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

WAF1, a potential mediator of p53 tumor suppression

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

v-Jun overrides the mitogen dependence of S-phase entry by deregulating retinoblastoma protein phosphorylation and E2F-pocket protein interactions as a consequence of enhanced cyclin E-cdk2 catalytic activity.

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Dominant negative c-jun inhibits activation of the cyclin D1 and cyclin E kinase complexes

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Deregulated expression of human c-jun transforms primary rat embryo cells in cooperation with an activated c-Ha-ras gene and transforms rat-1a cells as a single gene

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

JunB is essential for mammalian placentation

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Cell cycle-dependent variations in c-Jun and JunB phosphorylation: a role in the control of cyclin D1 expression.

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Chronic Myeloid Leukemia with Increased Granulocyte Progenitors in Mice Lacking JunB Expression in the Myeloid Lineage

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

JunB can substitute for Jun in mouse development and cell proliferation

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

JunD protects cells from p53-dependent senescence and apoptosis.

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Tumor surveillance via the ARF-p53 pathway.

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Menin interacts with the AP1 transcription factor JunD and represses JunD-activated transcription

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Mouse JunD negatively regulates fibroblast growth and antagonizes transformation by ras.

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Avian sarcoma virus 17 carries the jun oncogene

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Complete nucleotide sequence of a human c-onc gene: deduced amino acid sequence of the human c-fos protein

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Stepwise transformation of rat embryo fibroblasts: c-Jun, JunB, or JunD can cooperate with Ras for focus formation, but a c-Jun-containing heterodimer is required for immortalization

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Cellular transformation and malignancy induced by ras require c-jun

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Oncogenic transformation by ras and fos is mediated by c-Jun N-terminal phosphorylation

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

JAC, a direct target of oncogenic transcription factor Jun, is involved in cell transformation and tumorigenesis

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Heparin-binding epidermal growth factor-like growth factor, a v-Jun target gene, induces oncogenic transformation

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

c-fos protein can induce cellular transformation: a novel mechanism of activation of a cellular oncogene.

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Transformation by ras modifies AP1 composition and activity

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Role of DNA 5-methylcytosine transferase in cell transformation by fos

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Reactivation of proliferin gene expression is associated with increased angiogenesis in a cell culture model of fibrosarcoma tumor progression

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Autocrine growth and anchorage independence: two complementing Jun-controlled genetic programs of cellular transformation.

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Deregulated c-fos expression interferes with normal bone development in transgenic mice

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

c-fos is required for malignant progression of skin tumors.

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Transgenic mice demonstrate AP-1 (activator protein-1) transactivation is required for tumor promotion

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

AP-1 in mouse development and tumorigenesis

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Pin1 is overexpressed in breast cancer and cooperates with Ras signaling in increasing the transcriptional activity of c-Jun towards cyclin D1.

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Continuous c-fos expression precedes programmed cell death in vivo

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Cascade induction of c-fos, c-myc, and heat shock 70K transcripts during regression of the rat ventral prostate gland

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Altered gene expression in neurons during programmed cell death: identification of c-jun as necessary for neuronal apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

A c-Jun dominant negative mutant protects sympathetic neurons against programmed cell death

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Mitogen-activated protein kinase cascades as regulators of stress responses.

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Induction of apoptosis by the transcription factor c-Jun

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Induction of apoptosis by c-Fos protein

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Withdrawal of survival factors results in activation of the JNK pathway in neuronal cells leading to Fas ligand induction and cell death

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Opposing effects of ERK and JNK-p38 MAP kinases on apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Dominant-negative c-Jun promotes neuronal survival by reducing BIM expression and inhibiting mitochondrial cytochrome c release

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

The absence of c-fos prevents light-induced apoptotic cell death of photoreceptors in retinal degeneration in vivo

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Absence of excitotoxicity-induced apoptosis in the hippocampus of mice lacking the Jnk3 gene

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Induction of gadd45beta by NF-kappaB downregulates pro-apoptotic JNK signalling.

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Inhibition of JNK activation through NF-kappaB target genes

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Dynamic regulation of c-Jun N-terminal kinase activity in mouse brain by environmental stimuli

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Peripheral axotomy induces long-term c-Jun amino-terminal kinase-1 activation and activator protein-1 binding activity by c-Jun and junD in adult rat dorsal root ganglia In vivo.

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Lasting N-terminal phosphorylation of c-Jun and activation of c-Jun N-terminal kinases after neuronal injury.

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Requirement of JNK for stress-induced activation of the cytochrome c-mediated death pathway

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

c-Jun-dependent CD95-L expression is a rate-limiting step in the induction of apoptosis by alkylating agents

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Requirement for ceramide-initiated SAPK/JNK signalling in stress-induced apoptosis.

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Cooperation between STAT3 and c-jun suppresses Fas transcription.

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Wild-type human p53 and a temperature-sensitive mutant induce Fas/APO-1 expression

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

p53-mediated protective responses to UV irradiation.

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

p21-induced cycle arrest in G1 protects cells from apoptosis induced by UV-irradiation or RNA polymerase II blockage.

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

c-Jun is essential for normal mouse development and hepatogenesis

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

The Jnk1 and Jnk2 protein kinases are required for regional specific apoptosis during early brain development.

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Dissection of TNF receptor 1 effector functions: JNK activation is not linked to apoptosis while NF-kappaB activation prevents cell death

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Activation of SAPK/JNK by TNF receptor 1 through a noncytotoxic TRAF2-dependent pathway

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Stress-signalling kinase Sek1 protects thymocytes from apoptosis mediated by CD95 and CD3

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

Bcl-3 expression promotes cell survival following interleukin-4 deprivation and is controlled by AP1 and AP1-like transcription factors

1 reference

https://api.crossref.org/works/10.1038%2FNCB0502-E131

7 January 2021

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=11988758

Identifiers

2 references

30 April 2017

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

1271919

Dimensions Publication ID

1026096121

0 references

OpenCitations bibliographic resource ID

1271919

1 reference