Protein kinase C delta mediated cytotoxicity of 6-Hydroxydopamine via sustained extracellular signal-regulated kinase 1/2 activation in PC12 cells (Q39084131)

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scientific article published on 6 December 2013

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English	Protein kinase C delta mediated cytotoxicity of 6-Hydroxydopamine via sustained extracellular signal-regulated kinase 1/2 activation in PC12 cells	scientific article published on 6 December 2013

Statements

scholarly article

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Europe PubMed Central

PubMed publication ID

8 September 2017

Protein kinase C delta mediated cytotoxicity of 6-Hydroxydopamine via sustained extracellular signal-regulated kinase 1/2 activation in PC12 cells (English)

1 reference

Europe PubMed Central

PubMed publication ID

8 September 2017

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author name string

Ying Fan

1

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Europe PubMed Central

PubMed publication ID

8 September 2017

Jing Li

2

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Europe PubMed Central

PubMed publication ID

8 September 2017

Yan-Qiao Zhang

3

1 reference

Europe PubMed Central

PubMed publication ID

8 September 2017

Li-Hong Jiang

4

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Europe PubMed Central

PubMed publication ID

8 September 2017

Yi-Na Zhang

5

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Europe PubMed Central

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8 September 2017

Chao-Qi Yan

6

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Europe PubMed Central

PubMed publication ID

8 September 2017

publication date

6 December 2013

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Europe PubMed Central

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8 September 2017

Neurological Research

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8 September 2017

36

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Europe PubMed Central

PubMed publication ID

8 September 2017

1

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PubMed publication ID

8 September 2017

53-64

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PubMed publication ID

8 September 2017

Parkinson's disease. First of two parts

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Incidence of Parkinson disease and parkinsonism in three elderly populations of central Spain.

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Mitogen-activated protein (MAP) kinase pathways: regulation and physiological functions

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6-hydroxy-dopamine induced degeneration of central monoamine neurons

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MEK1 protein kinase inhibition protects against damage resulting from focal cerebral ischemia

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Differential activation of mitogen-activated protein kinase pathways after traumatic brain injury in the rat hippocampus

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Sustained extracellular signal-regulated kinase activation by 6-hydroxydopamine: implications for Parkinson's disease

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Role of Proteolytic Activation of Protein Kinase Cδ in Oxidative Stress-Induced Apoptosis

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Induced expression of early response genes/oxidative injury in rat pheochromocytoma (PC12) cell line by 6-hydroxydopamine: implication for Parkinson's disease

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Dopamine uptake inhibitor-induced rotation in 6-hydroxydopamine-lesioned rats involves both D1 and D2 receptors but is modulated through 5-hydroxytryptamine and noradrenaline receptors

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6-OHDA generated ROS induces DNA damage and p53- and PUMA-dependent cell death

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Molecular pathways involved in the neurotoxicity of 6-OHDA, dopamine and MPTP: contribution to the apoptotic theory in Parkinson's disease

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Neurotoxin-induced cell death in neuronal PC12 cells is mediated by induction of apoptosis

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Meaningful relationships: the regulation of the Ras/Raf/MEK/ERK pathway by protein interactions

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Dopaminergic neurons protected from degeneration by GDNF gene therapy

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Opposing effects of ERK and JNK-p38 MAP kinases on apoptosis

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Dual potential of extracellular signal-regulated kinase for the control of cell survival

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Cytoplasmic aggregates of phosphorylated extracellular signal-regulated protein kinases in Lewy body diseases

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Persistent activation of ERK contributes to glutamate-induced oxidative toxicity in a neuronal cell line and primary cortical neuron cultures

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Growth differentiation factor-15 prevents low potassium-induced cell death of cerebellar granule neurons by differential regulation of Akt and ERK pathways

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ERK activation and nuclear translocation in amyloid-beta peptide- and iron-stressed neuronal cell cultures

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6-Hydroxydopamine induces mitochondrial ERK activation.

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Increased M-calpain expression in the mesencephalon of patients with Parkinson's disease but not in other neurodegenerative disorders involving the mesencephalon: a role in nerve cell death?

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Membrane proximal ERK signaling is required for M-calpain activation downstream of epidermal growth factor receptor signaling.

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RETRACTED: 6-OHDA-induced apoptosis and mitochondrial dysfunction are mediated by early modulation of intracellular signals and interaction of Nrf2 and NF-κB factors

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Protein kinase C isoforms as therapeutic targets in nervous system disease states

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Tissue-specific developmental regulation of protein kinase C isoforms.

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Manganese toxicity is associated with mitochondrial dysfunction and DNA fragmentation in rat primary striatal neurons

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Protein kinase Cdelta is a key downstream mediator of manganese-induced apoptosis in dopaminergic neuronal cells.

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Dieldrin induces apoptosis by promoting caspase-3-dependent proteolytic cleavage of protein kinase Cdelta in dopaminergic cells: relevance to oxidative stress and dopaminergic degeneration

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21 January 2018

Protein kinase C delta (PKC delta): activation mechanisms and functions

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Rottlerin protected dopaminergic cell line from cytotoxicity of 6-hydroxydopamine by inhibiting PKCdelta phosphorylation

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Involvement of protein kinase C delta (PKCdelta) in phorbol ester-induced apoptosis in LNCaP prostate cancer cells. Lack of proteolytic cleavage of PKCdelta

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Rottlerin: an inappropriate and ineffective inhibitor of PKCdelta

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Rottlerin, a novel protein kinase inhibitor

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Elongation factor-2 kinase: effective inhibition by the novel protein kinase inhibitor rottlerin and relative insensitivity towards staurosporine.

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Tyrosine phosphorylation regulates the proteolytic activation of protein kinase Cdelta in dopaminergic neuronal cells

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Suppression of caspase-3-dependent proteolytic activation of protein kinase C delta by small interfering RNA prevents MPP+-induced dopaminergic degeneration

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Positive feedback of protein kinase C proteolytic activation during apoptosis

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Caspase‐3 dependent proteolytic activation of protein kinase Cδ mediates and regulates 1‐methyl‐4‐phenylpyridinium (MPP+)‐induced apoptotic cell death in dopaminergic cells: relevance to oxidative stress in dopaminergic degeneration

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Regulation of cell apoptosis by protein kinase c delta

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6-hydroxydopamine-induced apoptosis is mediated via extracellular auto-oxidation and caspase 3-dependent activation of protein kinase Cdelta

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Identifiers

10.1179/1743132813Y.0000000267

1 reference

Europe PubMed Central

PubMed publication ID

8 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

8 September 2017

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