SARS coronavirus spike protein-induced innate immune response occurs via activation of the NF-kappaB pathway in human monocyte macrophages in vitro (Q37234254)

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scientific article published on 29 January 2009

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English	SARS coronavirus spike protein-induced innate immune response occurs via activation of the NF-kappaB pathway in human monocyte macrophages in vitro	scientific article published on 29 January 2009

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scholarly article

1 reference

Europe PubMed Central

19 August 2017

SARS coronavirus spike protein-induced innate immune response occurs via activation of the NF-kappaB pathway in human monocyte macrophages in vitro (English)

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Europe PubMed Central

19 August 2017

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based on heuristic

inferred from title

spike glycoprotein [SARS-Cov]

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based on heuristic

inferred from title

author name string

Susan F Dosch

1

1 reference

Europe PubMed Central

19 August 2017

Supriya D Mahajan

2

1 reference

Europe PubMed Central

19 August 2017

Arlene R Collins

3

1 reference

Europe PubMed Central

19 August 2017

language of work or name

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inferred from title

publication date

29 January 2009

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Europe PubMed Central

19 August 2017

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Europe PubMed Central

19 August 2017

142

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Europe PubMed Central

19 August 2017

1-2

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Europe PubMed Central

19 August 2017

19-27

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Europe PubMed Central

19 August 2017

Modulation of cystic fibrosis lung disease by variants in interleukin-8.

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Proteolytic cleavage of VP1-2 is required for release of herpes simplex virus 1 DNA into the nucleus

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Interferon-mediated immunopathological events are associated with atypical innate and adaptive immune responses in patients with severe acute respiratory syndrome

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Cleavage of spike protein of SARS coronavirus by protease factor Xa is associated with viral infectivity

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SARS coronavirus and innate immunity

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The Immunobiology of SARS*.

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Rapid proteolysis of I kappa B-alpha is necessary for activation of transcription factor NF-kappa B

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In vitro detection of apoptosis in monocytes/macrophages infected with human coronavirus

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Amino acid substitutions within the leucine zipper domain of the murine coronavirus spike protein cause defects in oligomerization and the ability to induce cell-to-cell fusion

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The coronavirus spike protein induces endoplasmic reticulum stress and upregulation of intracellular chemokine mRNA concentrations

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Up-regulation of IL-6 and TNF-alpha induced by SARS-coronavirus spike protein in murine macrophages via NF-kappaB pathway

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NF-kappaB regulation in the immune system

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Expression profile of immune response genes in patients with Severe Acute Respiratory Syndrome

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[Expression of immune cells and their roles in the involved tissues of SARS patients]

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10.1016/J.VIRUSRES.2009.01.005

1 reference

Europe PubMed Central

19 August 2017

1 reference

Europe PubMed Central

19 August 2017

1 reference

Europe PubMed Central

19 August 2017

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