Cardiac Stim1 Silencing Impairs Adaptive Hypertrophy and Promotes Heart Failure Through Inactivation of mTORC2/Akt Signaling (Q36791996)

16 August 2017

Cardiac Stim1 Silencing Impairs Adaptive Hypertrophy and Promotes Heart Failure Through Inactivation of mTORC2/Akt Signaling (English)

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16 August 2017

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6

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31 May 2020

Catherine Pavoine

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31 May 2020

Jean-Sébastien Hulot

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31 May 2020

author name string

Ludovic Bénard

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31 May 2020

Jae Gyun Oh

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31 May 2020

Marine Cacheux

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31 May 2020

Ahyoung Lee

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31 May 2020

Mathieu Nonnenmacher

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31 May 2020

Erik Kohlbrenner

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31 May 2020

Changwon Kho

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31 May 2020

Roger J Hajjar

10

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31 May 2020

language of work or name

English

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publication date

2 March 2016

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16 August 2017

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31 May 2020

volume

133

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31 May 2020

issue

15

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31 May 2020

page(s)

1458-71; discussion 1471

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describes a project that uses

31 May 2020

ImageJ

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11 June 2022

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inferred from PubMed Central ID database lookup

cites work

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Critical role for stromal interaction molecule 1 in cardiac hypertrophy

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TRPC channels as effectors of cardiac hypertrophy

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The calcium store sensor, STIM1, reciprocally controls Orai and CaV1.2 channels.

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mTOR attenuates the inflammatory response in cardiomyocytes and prevents cardiac dysfunction in pathological hypertrophy

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5 September 2018

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5 September 2018

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5 September 2018

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5 September 2018

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5 September 2018

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5 September 2018

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5 September 2018

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5 September 2018

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Recombinant adeno-associated virus purification using novel methods improves infectious titer and yield

5 September 2018

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12 December 2020

Cardiac-specific overexpression of E40K active Akt prevents pressure overload-induced heart failure in mice by increasing angiogenesis and reducing apoptosis

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12 December 2020

Essential role of STIM1 in the development of cardiomyocyte hypertrophy

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12 December 2020

10.1161/CIRCULATIONAHA.115.020678

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