Mitochondria and ischemia/reperfusion injury (Q36225172)

12 August 2017

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title

Mitochondria and ischemia/reperfusion injury (English)

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12 August 2017

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Henry M Honda

1

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12 August 2017

Paavo Korge

series ordinal

2

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12 August 2017

James N Weiss

series ordinal

3

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12 August 2017

language of work or name

English

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publication date

1 June 2005

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Annals of the New York Academy of Sciences

12 August 2017

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12 August 2017

1047

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12 August 2017

248-258

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Preconditioning with ischemia: a delay of lethal cell injury in ischemic myocardium

12 August 2017

1 reference

https://api.crossref.org/works/10.1196%2FANNALS.1341.022

Consequences of brief ischemia: stunning, preconditioning, and their clinical implications: part 2.

21 January 2018

1 reference

https://api.crossref.org/works/10.1196%2FANNALS.1341.022

Glycogen synthase kinase-3beta mediates convergence of protection signaling to inhibit the mitochondrial permeability transition pore

21 January 2018

1 reference

https://api.crossref.org/works/10.1196%2FANNALS.1341.022

Inhibition of myocardial injury by ischemic postconditioning during reperfusion: comparison with ischemic preconditioning

21 January 2018

1 reference

https://api.crossref.org/works/10.1196%2FANNALS.1341.022

The role of mitochondrial K(ATP) channels in cardioprotection

21 January 2018

1 reference

https://api.crossref.org/works/10.1196%2FANNALS.1341.022

Mass spectrometric identification of proteins released from mitochondria undergoing permeability transition

21 January 2018

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https://api.crossref.org/works/10.1196%2FANNALS.1341.022

Role of the mitochondrial permeability transition in myocardial disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1196%2FANNALS.1341.022

Mitochondria: releasing power for life and unleashing the machineries of death

21 January 2018

1 reference

https://api.crossref.org/works/10.1196%2FANNALS.1341.022

Evidence for the presence of a reversible Ca2+-dependent pore activated by oxidative stress in heart mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1196%2FANNALS.1341.022

Mitochondrial non-specific pores remain closed during cardiac ischaemia, but open upon reperfusion

21 January 2018

1 reference

https://api.crossref.org/works/10.1196%2FANNALS.1341.022

Effects of fatty acids in isolated mitochondria: implications for ischemic injury and cardioprotection

21 January 2018

1 reference

https://api.crossref.org/works/10.1196%2FANNALS.1341.022

A distinct pathway remodels mitochondrial cristae and mobilizes cytochrome c during apoptosis

21 January 2018

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https://api.crossref.org/works/10.1196%2FANNALS.1341.022

Cytochrome c release from mitochondria proceeds by a two-step process

21 January 2018

1 reference

https://api.crossref.org/works/10.1196%2FANNALS.1341.022

Bid, Bax, and lipids cooperate to form supramolecular openings in the outer mitochondrial membrane

21 January 2018

1 reference

https://api.crossref.org/works/10.1196%2FANNALS.1341.022

Disruption of the outer mitochondrial membrane as a result of large amplitude swelling: the impact of irreversible permeability transition.

21 January 2018

1 reference

https://api.crossref.org/works/10.1196%2FANNALS.1341.022

Protection of cardiac mitochondria by diazoxide and protein kinase C: implications for ischemic preconditioning

21 January 2018

1 reference

https://api.crossref.org/works/10.1196%2FANNALS.1341.022

Characteristics and superoxide-induced activation of reconstituted myocardial mitochondrial ATP-sensitive potassium channels

21 January 2018

1 reference

https://api.crossref.org/works/10.1196%2FANNALS.1341.022

Multiprotein complex containing succinate dehydrogenase confers mitochondrial ATP-sensitive K+ channel activity

21 January 2018

1 reference

https://api.crossref.org/works/10.1196%2FANNALS.1341.022

Mitochondrial ATP-dependent potassium channels. Viable candidate effectors of ischemic preconditioning.

21 January 2018

1 reference

https://api.crossref.org/works/10.1196%2FANNALS.1341.022

Mechanisms by which opening the mitochondrial ATP- sensitive K(+) channel protects the ischemic heart

21 January 2018

1 reference

https://api.crossref.org/works/10.1196%2FANNALS.1341.022

High protonic potential actuates a mechanism of production of reactive oxygen species in mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1196%2FANNALS.1341.022

Mitochondria are excitable organelles capable of generating and conveying electrical and calcium signals.

21 January 2018

1 reference

https://api.crossref.org/works/10.1196%2FANNALS.1341.022

Reactive oxygen species (ROS)-induced ROS release: a new phenomenon accompanying induction of the mitochondrial permeability transition in cardiac myocytes

21 January 2018

1 reference

https://api.crossref.org/works/10.1196%2FANNALS.1341.022

Synchronized whole cell oscillations in mitochondrial metabolism triggered by a local release of reactive oxygen species in cardiac myocytes

21 January 2018

1 reference

https://api.crossref.org/works/10.1196%2FANNALS.1341.022

Propagation of the apoptotic signal by mitochondrial waves

21 January 2018

1 reference

https://api.crossref.org/works/10.1196%2FANNALS.1341.022

21 January 2018

Identifiers

DOI

10.1196/ANNALS.1341.022

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release_7sytc7ysebbw3kjcye6ee254iu

12 August 2017

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Fatcat

https://api.fatcat.wiki/v0/release/7sytc7ysebbw3kjcye6ee254iu

mapped directly with Wikidata item

24 November 2022

based on heuristic

1 reference