A Distinct Pathway Remodels Mitochondrial Cristae and Mobilizes Cytochrome c during Apoptosis (Q29620536)

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30 April 2017

A Distinct Pathway Remodels Mitochondrial Cristae and Mobilizes Cytochrome c during Apoptosis (English)

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6 February 2025

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Korsmeyer SJ

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30 April 2017

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Luca Scorrano

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6 February 2025

author name string

Mona Ashiya

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6 February 2025

Karolyn Buttle

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6 February 2025

Solly Weiler

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6 February 2025

Scott A. Oakes

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Carmen A. Mannella

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publication date

January 2002

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30 April 2017

language of work or name

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30 April 2017

number of pages

13

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http://www.cell.com/article/S1534580701001162/pdf

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work available at URL

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6 February 2025

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6 February 2025

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30 April 2017

volume

2

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30 April 2017

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1

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30 April 2017

page(s)

55-67

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Mitochondrial transport of cations: channels, exchangers, and permeability transition.

30 April 2017

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Bax-induced Cytochrome C Release from Mitochondria Is Independent of the Permeability Transition Pore but Highly Dependent on Mg2+ Ions

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The Role of Dynamin-Related Protein 1, a Mediator of Mitochondrial Fission, in Apoptosis

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The coordinate release of cytochrome c during apoptosis is rapid, complete and kinetically invariant

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Rate-limiting step preceding cytochrome c release in cells primed for Fas-mediated apoptosis revealed by analysis of cellular mosaicism of respiratory changes

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Imaging the Permeability Pore Transition in Single Mitochondria

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The pro-apoptotic proteins, Bid and Bax, cause a limited permeabilization of the mitochondrial outer membrane that is enhanced by cytosol

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The mitochondrial permeability transition in cell death: a common mechanism in necrosis, apoptosis and autophagy

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Cytochrome c and dATP-Dependent Formation of Apaf-1/Caspase-9 Complex Initiates an Apoptotic Protease Cascade

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Bid, a Bcl2 Interacting Protein, Mediates Cytochrome c Release from Mitochondria in Response to Activation of Cell Surface Death Receptors

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Mitochondrial Proliferation and Paradoxical Membrane Depolarization during Terminal Differentiation and Apoptosis in a Human Colon Carcinoma Cell Line

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Organelle division: Self-assembling GTPase caught in the middle

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A reversible component of mitochondrial respiratory dysfunction in apoptosis can be rescued by exogenous cytochrome c.

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Transient and long-lasting openings of the mitochondrial permeability transition pore can be monitored directly in intact cells by changes in mitochondrial calcein fluorescence

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