Caspase-1 modulates incisional sensitization and inflammation (Q34154905)

31 July 2017

title

Caspase-1 modulates incisional sensitization and inflammation (English)

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31 July 2017

De-Yong Liang

1

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31 July 2017

XiangQi Li

2

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31 July 2017

Wen-Wu Li

3

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31 July 2017

Dennis Fiorino

4

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31 July 2017

Yanli Qiao

5

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31 July 2017

Peyman Sahbaie

6

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31 July 2017

David C Yeomans

7

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31 July 2017

J David Clark

8

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31 July 2017

language of work or name

English

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publication date

1 October 2010

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31 July 2017

number of pages

12

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inferred from page(s)

published in

Anesthesiology

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31 July 2017

volume

113

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31 July 2017

issue

4

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31 July 2017

page(s)

945-956

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Inflammasomes: too big to miss

31 July 2017

cites work

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Chronic morphine administration enhances nociceptive sensitivity and local cytokine production after incision

5 July 2018

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5 July 2018

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5 July 2018

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5 July 2018

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5 July 2018

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5 July 2018

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5 July 2018

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Characterization of a rat model of incisional pain.

5 July 2018

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5 July 2018

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5 July 2018

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5 July 2018

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5 July 2018

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5 July 2018

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5 July 2018

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5 July 2018

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Local and systemic release of cytokines, nerve growth factor, prostaglandin E2, and substance P in incisional wounds and serum following cesarean delivery

28 October 2018

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28 October 2018

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28 October 2018

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Blockade of the complement C5a receptor reduces incisional allodynia, edema, and cytokine expression

28 October 2018

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Upregulation of prostaglandin E2 and interleukins in the central nervous system and peripheral tissue during and after surgery in humans

28 October 2018

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28 October 2018

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28 October 2018

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Spinal NF-kB activation induces COX-2 upregulation and contributes to inflammatory pain hypersensitivity

28 October 2018

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28 October 2018

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28 October 2018

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28 October 2018

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28 October 2018

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28 October 2018

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28 October 2018

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12 December 2020

(S)-1-((S)-2-{[1-(4-amino-3-chloro-phenyl)-methanoyl]-amino}-3,3-dimethyl-butanoyl)-pyrrolidine-2-carboxylic acid ((2R,3S)-2-ethoxy-5-oxo-tetrahydro-furan-3-yl)-amide (VX-765), an orally available selective interleukin (IL)-converting enzyme/caspase

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The activity of caspase-1 is increased in lesional psoriatic epidermis

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Neutrophil Migration Induced by IL-1β Depends upon LTB4 Released by Macrophages and upon TNF-α and IL-1β Released by Mast Cells

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Interleukin-1 signaling is required for induction and maintenance of postoperative incisional pain: genetic and pharmacological studies in mice

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Interleukin-1beta induces substance P release from primary afferent neurons through the cyclooxygenase-2 system

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Cytokine involvement in dynorphin-induced allodynia

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Inhibition of caspase-1-like activity by Ac-Tyr-Val-Ala-Asp-chloromethyl ketone induces long-lasting neuroprotection in cerebral ischemia through apoptosis reduction and decrease of proinflammatory cytokines

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Peri-sciatic proinflammatory cytokines, reactive oxygen species, and complement induce mirror-image neuropathic pain in rats

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Processing of precursor interleukin 1 beta and inflammatory disease

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Opiates suppress carrageenan-induced edema and hyperthermia at doses that inhibit hyperalgesia

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A Synthetic Inhibitor of Interleukin-lβ Converting Enzyme Prevents Endotoxin-Induced Interleukin-lβ ProductionIn VitroandIn Vivo

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Formation of active IL-1 beta from pro-IL-1 beta catalyzed by stratum corneum chymotryptic enzyme in vitro

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LPS challenge in D-galactosamine-sensitized mice accounts for caspase-dependent fulminant hepatitis, not for septic shock

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10.1097/ALN.0B013E3181EE2F17

Identifiers

DOI

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31 July 2017

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31 July 2017

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