Activation of c-jun N-terminal kinase upon influenza A virus (IAV) infection is independent of pathogen-related receptors but dependent on amino acid sequence variations of IAV NS1. (Q34059292)

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English	Activation of c-jun N-terminal kinase upon influenza A virus (IAV) infection is independent of pathogen-related receptors but dependent on amino acid sequence variations of IAV NS1.	scientific article

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scholarly article

1 reference

Europe PubMed Central

30 July 2017

Activation of c-jun N-terminal kinase upon influenza A virus (IAV) infection is independent of pathogen-related receptors but dependent on amino acid sequence variations of IAV NS1. (English)

1 reference

Europe PubMed Central

30 July 2017

influenza A virus

object named as

influenza A virus

1 reference

based on heuristic

inferred from title

object named as

Ahmed Mostafa

4

0 references

object named as

Stephan Ludwig

9

0 references

Stephan Pleschka

7

object named as

Stephan Pleschka

1 reference

Europe PubMed Central

30 July 2017

Wolfgang Nacken

1

object named as

Wolfgang Nacken

1 reference

Europe PubMed Central

30 July 2017

author name string

Darisuren Anhlan

2

1 reference

Europe PubMed Central

30 July 2017

Eike R Hrincius

3

1 reference

Europe PubMed Central

30 July 2017

Thorsten Wolff

5

1 reference

Europe PubMed Central

30 July 2017

Anne Sadewasser

6

1 reference

Europe PubMed Central

30 July 2017

Christina Ehrhardt

8

1 reference

Europe PubMed Central

30 July 2017

language of work or name

0 references

publication date

28 May 2014

1 reference

Europe PubMed Central

30 July 2017

Journal of Virology

1 reference

Europe PubMed Central

30 July 2017

88

1 reference

Europe PubMed Central

30 July 2017

8843-8852

1 reference

Europe PubMed Central

30 July 2017

16

1 reference

Europe PubMed Central

30 July 2017

Improved dual promotor-driven reverse genetics system for influenza viruses.

1 reference

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1 reference

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1 reference

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1 reference

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1 reference

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1 reference

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1 reference

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1 reference

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1 reference

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1 reference

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1 reference

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1 reference

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1 reference

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1 reference

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Epstein-Barr virus-encoded latent membrane protein 1 activates the JNK pathway through its extreme C terminus via a mechanism involving TRADD and TRAF2

1 reference

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Influenza virus NS1 protein interacts with the cellular 30 kDa subunit of CPSF and inhibits 3'end formation of cellular pre-mRNAs

1 reference

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1 reference

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Identifiers

10.1128/JVI.00424-14

1 reference

Europe PubMed Central

30 July 2017

1 reference

Europe PubMed Central

30 July 2017

1 reference

Europe PubMed Central

30 July 2017

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