Autoimmunity as a consequence of retrovirus-mediated expression of C-FLIP in lymphocytes (Q33885931)

Dataset for "Continued use of retracted papers: Temporal trends in citations and (lack of) awareness of retractions shown in citation contexts in biomedicine"

29 July 2017

2 references

Continued use of retracted papers: Temporal trends in citations and (lack of) awareness of retractions shown in citation contexts in biomedicine

title

Autoimmunity as a consequence of retrovirus-mediated expression of C-FLIP in lymphocytes (English)

1 reference

29 July 2017

L Van Parijs

1

1 reference

29 July 2017

Y Refaeli

series ordinal

2

1 reference

29 July 2017

A K Abbas

series ordinal

3

1 reference

29 July 2017

D Baltimore

series ordinal

4

1 reference

29 July 2017

language of work or name

English

0 references

publication date

1 December 1999

1 reference

29 July 2017

1 reference

29 July 2017

11

1 reference

29 July 2017

763-770

1 reference

29 July 2017

6

1 reference

Uncoupling IL-2 signals that regulate T cell proliferation, survival, and Fas-mediated activation-induced cell death

29 July 2017

1 reference

12 January 2024

https://opencitations.net/index/api/v1/citations/10.1016/S1074-7613(00)80103-X

CD95 (Fas)-dependent elimination of self-reactive B cells upon interaction with CD4+ T cells.

1 reference

9 February 2024

https://opencitations.net/index/api/v1/references/10.1016/S1074-7613(00)80150-8

Dominant interfering Fas gene mutations impair apoptosis in a human autoimmune lymphoproliferative syndrome

1 reference

9 February 2024

https://opencitations.net/index/api/v1/references/10.1016/S1074-7613(00)80150-8

The role of Fas in autoimmune diabetes

1 reference

9 February 2024

https://opencitations.net/index/api/v1/references/10.1016/S1074-7613(00)80150-8

FADD, a novel death domain-containing protein, interacts with the death domain of Fas and initiates apoptosis

1 reference

9 February 2024

https://opencitations.net/index/api/v1/references/10.1016/S1074-7613(00)80150-8

Apoptosis by death factor

1 reference

9 February 2024

https://opencitations.net/index/api/v1/references/10.1016/S1074-7613(00)80150-8

Production of high-titer helper-free retroviruses by transient transfection

1 reference

9 February 2024

https://opencitations.net/index/api/v1/references/10.1016/S1074-7613(00)80150-8

A dominant interfering mutant of FADD/MORT1 enhances deletion of autoreactive thymocytes and inhibits proliferation of mature T lymphocytes

1 reference

9 February 2024

https://opencitations.net/index/api/v1/references/10.1016/S1074-7613(00)80150-8

Impaired Negative Selection of T Cells in Hodgkin's Disease Antigen CD30–Deficient Mice

1 reference

9 February 2024

https://opencitations.net/index/api/v1/references/10.1016/S1074-7613(00)80150-8

Requirement of Fas for the development of autoimmune diabetes in nonobese diabetic mice

1 reference

9 February 2024

https://opencitations.net/index/api/v1/references/10.1016/S1074-7613(00)80150-8

HIV-1 Nef protein protects infected primary cells against killing by cytotoxic T lymphocytes.

1 reference

9 February 2024

https://opencitations.net/index/api/v1/references/10.1016/S1074-7613(00)80150-8

Protection against Fas-dependent Th1-mediated apoptosis by antigen receptor engagement in B cells.

1 reference

9 February 2024

https://opencitations.net/index/api/v1/references/10.1016/S1074-7613(00)80150-8

FLICE, a novel FADD-homologous ICE/CED-3-like protease, is recruited to the CD95 (Fas/APO-1) death--inducing signaling complex

1 reference

9 February 2024

https://opencitations.net/index/api/v1/references/10.1016/S1074-7613(00)80150-8

Autocrine T-cell suicide mediated by APO-1/(Fas/CD95)

1 reference

9 February 2024

https://opencitations.net/index/api/v1/references/10.1016/S1074-7613(00)80150-8

Homeostasis and self-tolerance in the immune system: turning lymphocytes off.

1 reference

9 February 2024

https://opencitations.net/index/api/v1/references/10.1016/S1074-7613(00)80150-8

Involvement of MACH, a novel MORT1/FADD-interacting protease, in Fas/APO-1- and TNF receptor-induced cell death

1 reference

9 February 2024

https://opencitations.net/index/api/v1/references/10.1016/S1074-7613(00)80150-8

Complementation of V(D)J recombination deficiency in RAG-1(-/-) B cells reveals a requirement for novel elements in the N-terminus of RAG-1.

1 reference

9 February 2024

https://opencitations.net/index/api/v1/references/10.1016/S1074-7613(00)80150-8

Targeted mutation in the Fas gene causes hyperplasia in peripheral lymphoid organs and liver

1 reference

9 February 2024

https://opencitations.net/index/api/v1/references/10.1016/S1074-7613(00)80150-8

The Vβ-specific superantigen staphylococcal enterotoxin B: Stimulation of mature T cells and clonal deletion in neonatal mice

1 reference

9 February 2024

https://opencitations.net/index/api/v1/references/10.1016/S1074-7613(00)80150-8

Lpr and gld: single gene models of systemic autoimmunity and lymphoproliferative disease

1 reference

9 February 2024

https://opencitations.net/index/api/v1/references/10.1016/S1074-7613(00)80150-8

Cell-autonomous Fas (CD95)/Fas-ligand interaction mediates activation-induced apoptosis in T-cell hybridomas.

1 reference

9 February 2024

https://opencitations.net/index/api/v1/references/10.1016/S1074-7613(00)80150-8

A natural immunological adjuvant enhances T cell clonal expansion through a CD28-dependent, interleukin (IL)-2-independent mechanism

1 reference

9 February 2024

https://opencitations.net/index/api/v1/references/10.1016/S1074-7613(00)80150-8

Mutations in Fas associated with human lymphoproliferative syndrome and autoimmunity

1 reference

9 February 2024

https://opencitations.net/index/api/v1/references/10.1016/S1074-7613(00)80150-8

Fas-mediated apoptosis and activation-induced T-cell proliferation are defective in mice lacking FADD/Mort1

1 reference

9 February 2024

https://opencitations.net/index/api/v1/references/10.1016/S1074-7613(00)80150-8

Fas and Fas ligand: lpr and gld mutations

1 reference

9 February 2024

https://opencitations.net/index/api/v1/references/10.1016/S1074-7613(00)80150-8

T cell tolerance by clonal elimination in the thymus

1 reference

9 February 2024

https://opencitations.net/index/api/v1/references/10.1016/S1074-7613(00)80150-8

A role for FADD in T cell activation and development

1 reference

9 February 2024

https://opencitations.net/index/api/v1/references/10.1016/S1074-7613(00)80150-8

A novel protein that interacts with the death domain of Fas/APO1 contains a sequence motif related to the death domain

1 reference

9 February 2024

https://opencitations.net/index/api/v1/references/10.1016/S1074-7613(00)80150-8

Viral FLICE-inhibitory proteins (FLIPs) prevent apoptosis induced by death receptors

1 reference

9 February 2024

https://opencitations.net/index/api/v1/references/10.1016/S1074-7613(00)80150-8

The fight of viruses against apoptosis

1 reference

9 February 2024

https://opencitations.net/index/api/v1/references/10.1016/S1074-7613(00)80150-8

Biochemical mechanisms of IL-2-regulated Fas-mediated T cell apoptosis.

1 reference

9 February 2024

https://opencitations.net/index/api/v1/references/10.1016/S1074-7613(00)80150-8

How do cytotoxic lymphocytes kill their targets?

1 reference

9 February 2024

https://opencitations.net/index/api/v1/references/10.1016/S1074-7613(00)80150-8

A negative regulatory function of B7 revealed in B7-1 transgenic mice

1 reference

9 February 2024

https://opencitations.net/index/api/v1/references/10.1016/S1074-7613(00)80150-8

Fas(CD95)/FasL interactions required for programmed cell death after T-cell activation.

1 reference

9 February 2024

https://opencitations.net/index/api/v1/references/10.1016/S1074-7613(00)80150-8

Placing death under control

1 reference

9 February 2024

https://opencitations.net/index/api/v1/references/10.1016/S1074-7613(00)80150-8

Inhibition of death receptor signals by cellular FLIP

1 reference

9 February 2024

https://opencitations.net/index/api/v1/references/10.1016/S1074-7613(00)80150-8

Identifiers

DOI

10.1016/S1074-7613(00)80150-8

1 reference

29 July 2017

1 reference