TGF-beta1-induced plasminogen activator inhibitor-1 expression in vascular smooth muscle cells requires pp60(c-src)/EGFR(Y845) and Rho/ROCK signaling (Q30440157)

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English	TGF-beta1-induced plasminogen activator inhibitor-1 expression in vascular smooth muscle cells requires pp60(c-src)/EGFR(Y845) and Rho/ROCK signaling	scientific article

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scholarly article

1 reference

Europe PubMed Central

15 August 2017

TGF-beta1-induced plasminogen activator inhibitor-1 expression in vascular smooth muscle cells requires pp60(c-src)/EGFR(Y845) and Rho/ROCK signaling (English)

1 reference

Europe PubMed Central

15 August 2017

1 reference

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author name string

Rohan Samarakoon

1

1 reference

Europe PubMed Central

15 August 2017

Stephen P Higgins

2

1 reference

Europe PubMed Central

15 August 2017

Craig E Higgins

3

1 reference

Europe PubMed Central

15 August 2017

Paul J Higgins

4

1 reference

Europe PubMed Central

15 August 2017

publication date

3 January 2008

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Europe PubMed Central

15 August 2017

Journal of Molecular and Cellular Cardiology

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Europe PubMed Central

15 August 2017

44

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Europe PubMed Central

15 August 2017

3

1 reference

Europe PubMed Central

15 August 2017

527-538

1 reference

Europe PubMed Central

15 August 2017

Regulation of extracellular matrix remodeling following transforming growth factor-beta1/epidermal growth factor-stimulated epithelial-mesenchymal transition in human premalignant keratinocytes

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2394714

20 June 2018

Negative regulation of TGF-beta receptor/Smad signal transduction.

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Transforming growth factor-beta2 and connective tissue growth factor in proliferative vitreoretinal diseases: possible involvement of hyalocytes and therapeutic potential of Rho kinase inhibitor

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The role of transforming growth factor-beta in atherosclerosis

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Plasminogen activator inhibitor-1 in chronic kidney disease: evidence and mechanisms of action

1 reference

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PAI-1 and the metabolic syndrome: links, causes, and consequences

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Regulation of Rho proteins by phosphorylation in the cardiovascular system

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PAI-1 transcriptional regulation during the G0 --> G1 transition in human epidermal keratinocytes

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Rho kinases in cardiovascular physiology and pathophysiology

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TGF-beta 1-induced PAI-1 expression is E box/USF-dependent and requires EGFR signaling

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RhoA modulates Smad signaling during transforming growth factor-beta-induced smooth muscle differentiation

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Decreased perivascular fibrosis but not cardiac hypertrophy in ROCK1+/- haploinsufficient mice

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Identification of intracellular pathways through which TGF-beta1 upregulates PAI-1 expression in endothelial cells

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Smad4 dependency defines two classes of transforming growth factor {beta} (TGF-{beta}) target genes and distinguishes TGF-{beta}-induced epithelial-mesenchymal transition from its antiproliferative and migratory responses

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Non-Smad TGF-beta signals

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MAPK/AP-1-dependent regulation of PAI-1 gene expression by TGF-beta in rat mesangial cells

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Rho-kinase is an important therapeutic target in cardiovascular medicine

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Rho-kinase mediates hyperglycemia-induced plasminogen activator inhibitor-1 expression in vascular endothelial cells

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Pharmacological inhibition and genetic deficiency of plasminogen activator inhibitor-1 attenuates angiotensin II/salt-induced aortic remodeling.

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20 June 2018

Upstream stimulatory factor regulates E box-dependent PAI-1 transcription in human epidermal keratinocytes

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Molecular regulation of vascular smooth muscle cell differentiation in development and disease

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New insights into TGF-beta-Smad signalling

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TGF-beta-induced RhoA and p160ROCK activation is involved in the inhibition of Cdc25A with resultant cell-cycle arrest

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Smad-dependent and Smad-independent pathways in TGF-beta family signalling

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Intramural plasminogen activator inhibitor type-1 and coronary atherosclerosis

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TGF-beta signaling in renal disease

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Age-dependent spontaneous coronary arterial thrombosis in transgenic mice that express a stable form of human plasminogen activator inhibitor-1.

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TGF-beta1-induced PAI-1 gene expression requires MEK activity and cell-to-substrate adhesion.

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Transforming growth factor-beta signal transduction in epithelial cells.

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Genetic programs of epithelial cell plasticity directed by transforming growth factor-beta

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Synergistic cooperation of TFE3 and smad proteins in TGF-beta-induced transcription of the plasminogen activator inhibitor-1 gene

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Direct binding of Smad3 and Smad4 to critical TGF beta-inducible elements in the promoter of human plasminogen activator inhibitor-type 1 gene

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Role of plasminogen activator inhibitor in the reciprocal regulation of bovine aortic endothelial and smooth muscle cell migration by TGF-beta 1.

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Bleomycin-induced pulmonary fibrosis in transgenic mice that either lack or overexpress the murine plasminogen activator inhibitor-1 gene

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Transforming growth factor beta 1 induces neointima formation through plasminogen activator inhibitor-1-dependent pathways

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Rho/Rho-kinase pathway contributes to C-reactive protein-induced plasminogen activator inhibitor-1 expression in endothelial cells

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26 November 2018

Critical role of Rho-kinase and MEK/ERK pathways for angiotensin II-induced plasminogen activator inhibitor type-1 gene expression

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PAI-1 and TGF-beta: unmasking the real driver of TGF-beta-induced vascular pathology

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10.1016/J.YJMCC.2007.12.006

1 reference

Europe PubMed Central

15 August 2017

1 reference

Europe PubMed Central

15 August 2017

1 reference

Europe PubMed Central

15 August 2017

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