Vascular protection by angiotensin receptor antagonism involves differential VEGF expression in both hemispheres after experimental stroke (Q28740866)

5

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author name string

Weihua Guan

1

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Payaningal R Somanath

2

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Anna Kozak

3

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Anna Goc

4

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Adviye Ergul

6

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Maribeth H Johnson

7

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Sahar Soliman

9

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Susan C Fagan

Creative Commons Attribution 4.0 International

10

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language of work or name

English

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publication date

2011

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published in

PLOS One

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volume

6

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issue

9

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page(s)

e24551

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copyright license

start time

1 September 2011

1 reference

April 2022 Public Data File from Crossref

copyright status

copyrighted

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cites work

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Ischemic Postconditioning as a Novel Avenue to Protect against Brain Injury after Stroke

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Candesartan augments ischemia-induced proangiogenic state and results in sustained improvement after stroke.

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VEGF enhances angiogenesis and promotes blood-brain barrier leakage in the ischemic brain

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Candesartan inhibits carotid intimal thickening and ameliorates insulin resistance in balloon-injured diabetic rats

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Induction of vascular endothelial growth factor receptor-3 mRNA in glial cells following focal cerebral ischemia in rats

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Enhanced expression of vascular endothelial growth factor receptor-3 in the subventricular zone of stroke-lesioned rats

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p38 MAP kinase--a molecular switch between VEGF-induced angiogenesis and vascular hyperpermeability.

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Vascular permeability induced by VEGF family members in vivo: role of endogenous PAF and NO synthesis

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VEGF-A and -C but not -B mediate increased vascular permeability in preserved lung grafts

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inferred from PubMed ID database lookup

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based on heuristic

Identifiers

DOI

10.1371/JOURNAL.PONE.0024551

1 reference

2043971

ADS bibcode

2011PLoSO...624551G

0 references

2043971

1 reference

1 reference

1 reference