The death receptor antagonist FAIM promotes neurite outgrowth by a mechanism that depends on ERK and NF-kapp B signaling (Q28571207)

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4 January 2020

title

The death receptor antagonist FAIM promotes neurite outgrowth by a mechanism that depends on ERK and NF-kapp B signaling (English)

2 references

31 January 2017

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4 January 2020

1 reference

1

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4 January 2020

Miguel F Segura

3

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4 January 2020

Humberto Gutierrez

4

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4 January 2020

Raffaella Gozzelino

6

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4 January 2020

Daniel Sanchis

7

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4 January 2020

Jose R. Bayascas

8

1 reference

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4 January 2020

Carme Gallego

9

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4 January 2020

Alun Davies

11

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4 January 2020

Joan X. Comella

12

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4 January 2020

author name string

Xavier Dolcet

2

2 references

31 January 2017

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4 January 2020

Maria-Teresa Diaz-Meco

5

2 references

31 January 2017

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4 January 2020

Jorge Moscat

10

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4 January 2020

language of work or name

English

0 references

publication date

1 November 2004

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4 January 2020

number of pages

14

1 reference

inferred from page(s)

published in

Journal of Cell Biology

2 references

31 January 2017

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volume

167

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4 January 2020

issue

3

2 references

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page(s)

479-492

1 reference

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Fas/tumor necrosis factor receptor death signaling is required for axotomy-induced death of motoneurons in vivo

4 January 2020

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A novel gene coding for a Fas apoptosis inhibitory molecule (FAIM) isolated from inducibly Fas-resistant B lymphocytes

19 March 2017

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Fas/Apo [apoptosis]-1 and associated proteins in the differentiating cerebral cortex: induction of caspase-dependent cell death and activation of NF-kappaB

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Specificity of receptor tyrosine kinase signaling: transient versus sustained extracellular signal-regulated kinase activation

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Embryonic lethality and liver degeneration in mice lacking the RelA component of NF-kappa B

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Molecular and biochemical characterization of the human trk proto-oncogene

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19 March 2017

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Activation of MAP kinase kinase is necessary and sufficient for PC12 differentiation and for transformation of NIH 3T3 cells

7 April 2017

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The CD95(APO-1/Fas) DISC and beyond

7 April 2017

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The neurotrophin receptor p75(NTR): novel functions and implications for diseases of the nervous system

29 September 2017

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Receptor-specific regulation of B-cell susceptibility to Fas-mediated apoptosis and a novel Fas apoptosis inhibitory molecule

29 September 2017

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Neurotrophins: neurotrophic modulation of neurite growth

29 September 2017

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Programmed cell death of embryonic motoneurons triggered through the Fas death receptor

29 September 2017

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TNF-alpha and IFN-gamma render microglia sensitive to Fas ligand-induced apoptosis by induction of Fas expression and down-regulation of Bcl-2 and Bcl-xL.

29 September 2017

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Competitive signaling between TrkA and p75 nerve growth factor receptors determines cell survival.

29 September 2017

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Identification of lysine residues required for signal-induced ubiquitination and degradation of I kappa B-alpha in vivo

29 September 2017

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Mitogen-activated protein kinase activation is insufficient for growth factor receptor-mediated PC12 cell differentiation

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Fas/CD95/APO-1 can function as a death receptor for neuronal cells in vitro and in vivo and is upregulated following cerebral hypoxic-ischemic injury to the developing rat brain

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3 June 2018

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p75-mediated NF-kappaB activation enhances the survival response of developing sensory neurons to nerve growth factor.

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Motoneuron death triggered by a specific pathway downstream of Fas. potentiation by ALS-linked SOD1 mutations.

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An alternatively spliced long form of Fas apoptosis inhibitory molecule (FAIM) with tissue-specific expression in the brain.

21 January 2018

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Oligodendroglial apoptosis occurs along degenerating axons and is associated with FAS and p75 expression following spinal cord injury in the rat

21 January 2018

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NF-kappa B signaling promotes both cell survival and neurite process formation in nerve growth factor-stimulated PC12 cells.

21 January 2018

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Fas ligand and Fas are expressed constitutively in human astrocytes and the expression increases with IL-1, IL-6, TNF-alpha, or IFN-gamma.

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Fas engagement induces neurite growth through ERK activation and p35 upregulation

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p75-deficient trigeminal sensory neurons have an altered response to NGF but not to other neurotrophins

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Characterization and isolation of proteolytically modified nerve growth factor

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Identifiers

DOI

10.1083/JCB.200403093

2 references

31 January 2017

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:15520226%20AND%20SRC:MED&resulttype=core&format=json

4 January 2020

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:15520226%20AND%20SRC:MED&resulttype=core&format=json

4 January 2020

2 references

31 January 2017